Neurofascin as a target for autoantibodies in peripheral neuropathies. - Wikidata - awgldk - TriplyDB

Neurofascin as a target for autoantibodies in peripheral neuropathies.

Neurofascin as a target for autoantibodies in peripheral neuropathies.

http://www.wikidata.org/entity/Q36524437

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Neuroinflammation in the peripheral nerve: Cause, modulator, or bystander in peripheral neuropathies?Neuro-glial interactions at the nodes of Ranvier: implication in health and diseases Node of Ranvier disruption as a cause of neurological diseases Mapping autoantigen epitopes: molecular insights into autoantibody-associated disorders of the nervous system Neurofascin IgG4 antibodies in CIDP associate with disabling tremor and poor response to IVIg.Postnatal Loss of Neuronal and Glial Neurofascins Differentially Affects Node of Ranvier Maintenance and Myelinated Axon Function.Endogenous antibodies contribute to macrophage-mediated demyelination in a mouse model for CMT1B.Contactin-1 and Neurofascin-155/-186 Are Not Targets of Auto-Antibodies in Multifocal Motor Neuropathy Chronic inflammatory demyelinating polyneuropathy (CIDP): change of serum IgG dimer levels during treatment with intravenous immunoglobulins.Chronic inflammatory demyelinating polyradiculoneuropathy: from pathology to phenotype.Subtype-specific therapy for autoimmune neuropathies?Characterization of IgG4 anti-neurofascin 155 antibody-positive polyneuropathy.Neurofascin-155 IgG4 in chronic inflammatory demyelinating polyneuropathy.Neurofascin-155 as a putative antigen in combined central and peripheral demyelination Maternal neurofascin-specific autoantibodies bind to structures of the fetal nervous system during pregnancy, but have no long term effect on development in the rat.An update in guillain-barré syndrome Specific contactin N-glycans are implicated in neurofascin binding and autoimmune targeting in peripheral neuropathies.Animal models of autoimmune neuropathy.Long-term maintenance of Na+ channels at nodes of Ranvier depends on glial contact mediated by gliomedin and NrCAM.To look for a needle in a haystack: the search for autoantibodies in multiple sclerosis.Chronic inflammatory demyelinating polyradiculoneuropathy and variants: where we are and where we should go.Optimizing IgG therapy in chronic autoimmune neuropathies: a hypothesis driven approach.Autoimmune antigenic targets at the node of Ranvier in demyelinating disorders.Subcellular patterning: axonal domains with specialized structure and function.Fulminant Central Plus Peripheral Nervous System Demyelination without Antibodies to Neurofascin.The Nodes of Ranvier: Molecular Assembly and Maintenance.Autoantibodies to nodal isoforms of neurofascin in chronic inflammatory demyelinating polyneuropathy.Axonal domain disorganization in Caspr1 and Caspr2 mutant myelinated axons affects neuromuscular junction integrity, leading to muscle atrophy.Intravenous immunoglobulin: pharmacological properties and use in polyneuropathies.Autoantibodies in chronic inflammatory neuropathies: diagnostic and therapeutic implications.Inhibitory axons are targeted in hippocampal cell culture by anti-Caspr2 autoantibodies associated with limbic encephalitis.Cellular investigations with human antibodies associated with the anti-IgLON5 syndrome.Diagnostic Utility of Auto Antibodies in Inflammatory Nerve Disorders.Differences in peripheral myelin antigen-specific T cell responses and T memory subsets in atypical versus typical CIDP The paranodal cytoskeleton clusters Na+ channels at nodes of Ranvier.Rituximab in treatment-resistant CIDP with antibodies against paranodal proteins.Antibodies against peripheral nerve antigens in chronic inflammatory demyelinating polyradiculoneuropathy.Disruption of nodal architecture in skin biopsies of patients with demyelinating neuropathies.Neurofascin (NF)155- and NF186-Specific T Cell Response in a Patient Developing a Central Pontocerebellar Demyelination after 10 Years of CIDP.Neurofascin antibodies in autoimmune, genetic, and idiopathic neuropathies.

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P2860

Neurofascin as a target for autoantibodies in peripheral neuropathies.

http://www.wikidata.org/entity/Q36524437

description

2012 nî lūn-bûn

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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2012年论文

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2012年论文

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name

Neurofascin as a target for autoantibodies in peripheral neuropathies.

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Neurofascin as a target for autoantibodies in peripheral neuropathies.

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Neurofascin as a target for autoantibodies in peripheral neuropathies.

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Neurofascin as a target for autoantibodies in peripheral neuropathies.

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Neurofascin as a target for autoantibodies in peripheral neuropathies.

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Neurofascin as a target for autoantibodies in peripheral neuropathies.

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Neurofascin as a target for autoantibodies in peripheral neuropathies.

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Claudia Sommer

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Edgar Meinl

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Harald Prüss

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Hendrik Harms

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Joachim Malotka

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Judy King Man Ng

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Klaus Dornmair

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Lutz Harms

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Masaaki Odaka

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Naoto Kawakami

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P2860

Guillain–Barré Syndrome

Guillain-Barré syndrome

Identification of a novel contactin-associated transmembrane receptor with multiple domains implicated in protein-protein interactions.

High-level and high-throughput recombinant protein production by transient transfection of suspension-growing human 293-EBNA1 cells

Neurofascin is a glial receptor for the paranodin/Caspr-contactin axonal complex at the axoglial junction

Improved green fluorescence

A glial signal consisting of gliomedin and NrCAM clusters axonal Na+ channels during the formation of nodes of Ranvier

Neurofascins are required to establish axonal domains for saltatory conduction

Rapid disruption of axon-glial integrity in response to mild cerebral hypoperfusion

Pathogenesis and treatment of immune-mediated neuropathies.

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Matthew N. Rasband

Christopher Linington

Björn Tackenberg

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2012-10-24T00:00:00Z

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232705948

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