Postexercise skeletal muscle glucose transport is normal in kininogen-deficient rats.
about
Sustained postexercise increases in AS160 Thr642 and Ser588 phosphorylation in skeletal muscle without sustained increases in kinase phosphorylationMechanisms for greater insulin-stimulated glucose uptake in normal and insulin-resistant skeletal muscle after acute exercise.Quantitative Biology of Exercise-Induced Signal Transduction Pathways.Role of AMP-Activated Protein Kinase for Regulating Post-exercise Insulin Sensitivity.Bradykinin, insulin, and glycemia responses to exercise performed above and below lactate threshold in individuals with type 2 diabetes.Serum Is Not Necessary for Prior Pharmacological Activation of AMPK to Increase Insulin Sensitivity of Mouse Skeletal Muscle.
P2860
Postexercise skeletal muscle glucose transport is normal in kininogen-deficient rats.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Postexercise skeletal muscle glucose transport is normal in kininogen-deficient rats.
@ast
Postexercise skeletal muscle glucose transport is normal in kininogen-deficient rats.
@en
type
label
Postexercise skeletal muscle glucose transport is normal in kininogen-deficient rats.
@ast
Postexercise skeletal muscle glucose transport is normal in kininogen-deficient rats.
@en
prefLabel
Postexercise skeletal muscle glucose transport is normal in kininogen-deficient rats.
@ast
Postexercise skeletal muscle glucose transport is normal in kininogen-deficient rats.
@en
P2860
P1476
Postexercise skeletal muscle glucose transport is normal in kininogen-deficient rats
@en
P2093
George G Schweitzer
P2860
P304
P356
10.1249/MSS.0B013E31820A7F65
P577
2011-07-01T00:00:00Z