Carbon nanoparticles induce ceramide- and lipid raft-dependent signalling in lung epithelial cells: a target for a preventive strategy against environmentally-induced lung inflammation - Wikidata - awgldk - TriplyDB

Carbon nanoparticles induce ceramide- and lipid raft-dependent signalling in lung epithelial cells: a target for a preventive strategy against environmentally-induced lung inflammation

Carbon nanoparticles induce ceramide- and lipid raft-dependent signalling in lung epithelial cells: a target for a preventive strategy against environmentally-induced lung inflammation

http://www.wikidata.org/entity/Q36536111

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Carbon black and titanium dioxide nanoparticles induce distinct molecular mechanisms of toxicity Paracelsus in nanotoxicology Intracellular signal modulation by nanomaterials Activation of Proinflammatory Responses in Cells of the Airway Mucosa by Particulate Matter: Oxidant- and Non-Oxidant-Mediated Triggering Mechanisms Polydatin protects the respiratory system from PM2.5 exposure Noninterventional open-label trial investigating the efficacy and safety of ectoine containing nasal spray in comparison with beclomethasone nasal spray in patients with allergic rhinitis.Mechanistic understanding of nanoparticles' interactions with extracellular matrix: the cell and immune system Signalling-dependent adverse health effects of carbon nanoparticles are prevented by the compatible solute mannosylglycerate (firoin) in vitro and in vivo Preventing carbon nanoparticle-induced lung inflammation reduces antigen-specific sensitization and subsequent allergic reactions in a mouse model.No involvement of alveolar macrophages in the initiation of carbon nanoparticle induced acute lung inflammation in mice.Reduction of neutrophilic lung inflammation by inhalation of the compatible solute ectoine: a randomized trial with elderly individuals.The compatible solute ectoine reduces the exacerbating effect of environmental model particles on the immune response of the airways.Potential applications of stress solutes from extremophiles in protein folding diseases and healthcare.Ectoine as a promising protective agent in humans and animals.Treatment of rhinitis sicca anterior with ectoine containing nasal spray.Effect of Particulate Matter Air Pollution on Cardiovascular Oxidative Stress Pathways.Non-Canonical Activation of the Epidermal Growth Factor Receptor by Carbon Nanoparticles.Resveratrol prevents nanoparticles-induced inflammation and oxidative stress via downregulation of PKC-α and NADPH oxidase in lung epithelial A549 cells.

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P2860

Carbon nanoparticles induce ceramide- and lipid raft-dependent signalling in lung epithelial cells: a target for a preventive strategy against environmentally-induced lung inflammation

http://www.wikidata.org/entity/Q36536111

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Carbon nanoparticles induce ce ...... ally-induced lung inflammation

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Carbon nanoparticles induce ce ...... ally-induced lung inflammation

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Carbon nanoparticles induce ce ...... ally-induced lung inflammation

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Carbon nanoparticles induce ce ...... ally-induced lung inflammation

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Carbon nanoparticles induce ce ...... ally-induced lung inflammation

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Particle and Fibre Toxicology

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Carbon nanoparticles induce ce ...... ally-induced lung inflammation

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Christiane Bieschke

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Daniel Stöckmann

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Henrike Peuschel

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Ingo Felsner

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Jean Krutmann

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Klaus Unfried

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Marijan Gotić

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Matthias Kroker

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Sascha Jakob

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Susanne Grether-Beck

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P2860

The stabilization of proteins by osmolytes

Combustion-derived nanoparticles: a review of their toxicology following inhalation exposure

Long-term air pollution exposure and living close to busy roads are associated with COPD in women.

Organic osmolytes as compatible, metabolic and counteracting cytoprotectants in high osmolarity and other stresses

Non-enzymatic triggering of the ceramide signalling cascade by solar UVA radiation

Carbon black and titanium dioxide nanoparticles elicit distinct apoptotic pathways in bronchial epithelial cells.

EGF receptor exposed to oxidative stress acquires abnormal phosphorylation and aberrant activated conformation that impairs canonical dimerization

Lung injury and cancer: Mechanistic insights into ceramide and EGFR signaling under cigarette smoke.

Black carbon as an additional indicator of the adverse health effects of airborne particles compared with PM10 and PM2.5.

Current hypotheses on the mechanisms of toxicity of ultrafine particles.

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10.1186/1743-8977-9-48

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Judith Haendeler

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