Wnt/β-catenin signaling is a key downstream mediator of MET signaling in glioblastoma stem cells. - Wikidata - awgldk - TriplyDB

Wnt/β-catenin signaling is a key downstream mediator of MET signaling in glioblastoma stem cells.

Wnt/β-catenin signaling is a key downstream mediator of MET signaling in glioblastoma stem cells.

http://www.wikidata.org/entity/Q36544112

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Targeting the epithelial to mesenchymal transition in glioblastoma: the emerging role of MET signaling Ockham's razor for the MET-driven invasive growth linking idiopathic pulmonary fibrosis and cancer Registered report: Wnt activity defines colon cancer stem cells and is regulated by the microenvironment The evolving landscape of glioblastoma stem cells.NFAT1 is highly expressed in, and regulates the invasion of, glioblastoma multiforme cells Glioblastoma specific antigens, GD2 and CD90, are not involved in cancer stemness.HOTAIR is a therapeutic target in glioblastoma.Resveratrol Impairs Glioma Stem Cells Proliferation and Motility by Modulating the Wnt Signaling Pathway In vivo RNAi screen identifies NLK as a negative regulator of mesenchymal activity in glioblastoma Assessing current therapeutic approaches to decode potential resistance mechanisms in glioblastomas.Ionizing radiation in glioblastoma initiating cells.Atypical Diabetic Foot Ulcer Keratinocyte Protein Signaling Correlates with Impaired Wound Healing.MiR-146b-5p overexpression attenuates stemness and radioresistance of glioma stem cells by targeting HuR/lincRNA-p21/β-catenin pathway.Integrated mRNA and microRNA transcriptome sequencing characterizes sequence variants and mRNA-microRNA regulatory network in nasopharyngeal carcinoma model systems.MicroRNA-320 regulates the radiosensitivity of cervical cancer cells C33AR by targeting β-catenin.Cancer stem cells in glioma: challenges and opportunities.Mechanisms of RAS/β-catenin interactions.New treatment strategies to eradicate cancer stem cells and niches in glioblastoma.Mechanisms regulating glioma invasion.WNT signaling in glioblastoma and therapeutic opportunities.Induction of miR-3648 Upon ER Stress and Its Regulatory Role in Cell Proliferation Nuclear phosphorylated Y142 β-catenin accumulates in astrocytomas and glioblastomas and regulates cell invasion.Sox2: regulation of expression and contribution to brain tumors.LoVo colon cancer cells resistant to oxaliplatin overexpress c-MET and VEGFR-1 and respond to VEGF with dephosphorylation of c-MET.Cool-1-mediated inhibition of c-Cbl modulates multiple critical properties of glioblastomas, including the ability to generate tumors in vivo.Long non-coding RNA in glioma: signaling pathways.Role and Therapeutic Targeting of the HGF/MET Pathway in Glioblastoma.PI3K inhibitor combined with miR-125b inhibitor sensitize TMZ-induced anti-glioma stem cancer effects through inactivation of Wnt/β-catenin signaling pathway.Inhibition of EGFR induces a c-MET-driven stem cell population in glioblastoma.Blocking epithelial-to-mesenchymal transition in glioblastoma with a sextet of repurposed drugs: the EIS regimen.Targeting WNT Signaling for Multifaceted Glioblastoma Therapy.Aberrantly activated Cox-2 and Wnt signaling interact to maintain cancer stem cells in glioblastoma.Sulforaphane suppresses the growth of glioblastoma cells, glioblastoma stem cell-like spheroids, and tumor xenografts through multiple cell signaling pathways.Molecular Determinants of Malignant Brain Cancers: From Intracellular Alterations to Invasion Mediated by Extracellular Vesicles.Targeting different domains of gap junction protein to control malignant glioma.Natural Bioactive Compounds: Alternative Approach to the Treatment of Glioblastoma Multiforme.Changes in chromatin state reveal ARNT2 at a node of a tumorigenic transcription factor signature driving glioblastoma cell aggressiveness.Developmentally regulated signaling pathways in glioma invasion.Decreased miRNA-637 is an unfavorable prognosis marker and promotes glioma cell growth, migration and invasion via direct targeting Akt1.Function of the c-Met receptor tyrosine kinase in carcinogenesis and associated therapeutic opportunities.

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P2860

Wnt/β-catenin signaling is a key downstream mediator of MET signaling in glioblastoma stem cells.

http://www.wikidata.org/entity/Q36544112

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Do-Hyun Nam

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Eun Hee Kim

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Ho Jun Seol

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Hyun Jin Jin

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Jeongwu Lee

http://www.w3.org/2001/XMLSchema#string

Jinguen Rheey

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Kang Ho Kim

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Kyeung Min Joo

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Yeri Lee

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P2860

LDL-receptor-related proteins in Wnt signal transduction

Identification of the hepatocyte growth factor receptor as the c-met proto-oncogene product

Wnt induces LRP6 signalosomes and promotes dishevelled-dependent LRP6 phosphorylation

Cancerous stem cells can arise from pediatric brain tumors

PLAGL2 regulates Wnt signaling to impede differentiation in neural stem cells and gliomas

Comprehensive genomic characterization defines human glioblastoma genes and core pathways

SGX523 is an exquisitely selective, ATP-competitive inhibitor of the MET receptor tyrosine kinase with antitumor activity in vivo

Glioma stem cells promote radioresistance by preferential activation of the DNA damage response

Radiotherapy plus concomitant and adjuvant temozolomide for glioblastoma

Identification of human brain tumour initiating cells

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