Initiation of latent DNA replication in the Epstein-Barr virus genome can occur at sites other than the genetically defined origin
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Plasticity of DNA replication initiation in Epstein-Barr virus episomesA role for MRE11, NBS1, and recombination junctions in replication and stable maintenance of EBV episomes.Kaposi's sarcoma-associated herpesvirus latency-associated nuclear antigen 1 mediates episome persistence through cis-acting terminal repeat (TR) sequence and specifically binds TR DNA.The replicator of the Epstein-Barr virus latent cycle origin of DNA replication, oriP, is composed of multiple functional elements.Protein-DNA binding and CpG methylation at nucleotide resolution of latency-associated promoters Qp, Cp, and LMP1p of Epstein-Barr virusEstablishment of latent Epstein-Barr virus infection and stable episomal maintenance in murine B-cell lines.Transformation abrogates an early G1-phase arrest point required for specification of the Chinese hamster DHFR replication origin.The dyad symmetry element of Epstein-Barr virus is a dominant but dispensable replication origin.Episomal vectors for gene expression in mammalian cells.Human DNA replication initiation factors, ORC and MCM, associate with oriP of Epstein-Barr virus.Detection of Epstein-Barr virus genomes in peripheral blood B cells from solid-organ transplant recipients by fluorescence in situ hybridization.Strong minor groove base conservation in sequence logos implies DNA distortion or base flipping during replication and transcription initiation.Human origin recognition complex binds to the region of the latent origin of DNA replication of Epstein-Barr virus.Identification of a herpesvirus Saimiri cis-acting DNA fragment that permits stable replication of episomes in transformed T cellsLatent and lytic Epstein-Barr virus replication strategies.Origins of bidirectional replication of Epstein-Barr virus: models for understanding mammalian origins of DNA synthesis.Open chromatin structures regulate the efficiencies of pre-RC formation and replication initiation in Epstein-Barr virusThe plasmid replicon of Epstein-Barr virus: mechanistic insights into efficient, licensed, extrachromosomal replication in human cells.Identifying a property of origins of DNA synthesis required to support plasmids stably in human cells.Epstein-Barr Nuclear Antigen 1 modulates replication of oriP-plasmids by impeding replication and transcription fork migration through the family of repeats.Involvement of SSRP1 in latent replication of Kaposi's sarcoma-associated herpesvirusThe origin recognition complex in human diseases.The long-lasting love affair between the budding yeast Saccharomyces cerevisiae and the Epstein-Barr virus.DNA of a circular minichromosome linearized by restriction enzymes or other reagents is resistant to further cleavage: an influence of chromatin topology on the accessibility of DNA.Evidence that protein binding specifies sites of DNA demethylation.Genetic evidence that EBNA-1 is needed for efficient, stable latent infection by Epstein-Barr virusThe replisome pausing factor Timeless is required for episomal maintenance of latent Epstein-Barr virus.CD21-mediated entry and stable infection by Epstein-Barr virus in canine and rat cells.Activation of TRAF5 and TRAF6 signal cascades negatively regulates the latent replication origin of Epstein-Barr virus through p38 mitogen-activated protein kinase.Initiation of DNA replication within oriP is dispensable for stable replication of the latent Epstein-Barr virus chromosome after infection of established cell lines.An autonomous replicating element within the KSHV genome.Establishment of an oriP/EBNA1-based episomal vector transcribing human genomic beta-globin in cultured murine fibroblasts.Chromatin profiling of Epstein-Barr virus latency control regionThe latent origin of replication of Epstein-Barr virus directs viral genomes to active regions of the nucleusEssential elements of a licensed, mammalian plasmid origin of DNA synthesis.Role of the EBNA-1 protein in pausing of replication forks in the Epstein-Barr virus genome.Bacterial artificial chromosomes establish replication timing and sub-nuclear compartment de novo as extra-chromosomal vectors.EBNA1: Oncogenic Activity, Immune Evasion and Biochemical Functions Provide Targets for Novel Therapeutic Strategies against Epstein-Barr Virus- Associated Cancers.The ColE1 unidirectional origin acts as a polar replication fork pausing site.Transcript availability dictates the balance between strand-asynchronous and strand-coupled mitochondrial DNA replication
P2860
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P2860
Initiation of latent DNA replication in the Epstein-Barr virus genome can occur at sites other than the genetically defined origin
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
1995年论文
@zh
1995年论文
@zh-cn
name
Initiation of latent DNA repli ...... the genetically defined origin
@ast
Initiation of latent DNA repli ...... the genetically defined origin
@en
type
label
Initiation of latent DNA repli ...... the genetically defined origin
@ast
Initiation of latent DNA repli ...... the genetically defined origin
@en
prefLabel
Initiation of latent DNA repli ...... the genetically defined origin
@ast
Initiation of latent DNA repli ...... the genetically defined origin
@en
P2860
P356
P1476
Initiation of latent DNA repli ...... the genetically defined origin
@en
P2093
C L Schildkraut
R D Little
P2860
P304
P356
10.1128/MCB.15.5.2893
P407
P577
1995-05-01T00:00:00Z