Humoral response to herpes simplex virus is complement-dependent. - Wikidata - awgldk - TriplyDB

Humoral response to herpes simplex virus is complement-dependent.

Humoral response to herpes simplex virus is complement-dependent.

http://www.wikidata.org/entity/Q36552488

about

Complement activation is required for induction of a protective antibody response against West Nile virus infection West Nile virus nonstructural protein NS1 inhibits complement activation by binding the regulatory protein factor H Protective immune responses against West Nile virus are primed by distinct complement activation pathways Evasion of innate and adaptive immunity by flaviviruses Complement: a key system for immune surveillance and homeostasis Uncoupling CD21 and CD19 of the B-cell coreceptor Surviving mousepox infection requires the complement system Complement as an endogenous adjuvant for dendritic cell-mediated induction of retrovirus-specific CTLs.Interleukin-12 (IL-12) and IL-18 are important in innate defense against genital herpes simplex virus type 2 infection in mice but are not required for the development of acquired gamma interferon-mediated protective immunity.Herpes simplex virus type 1 latency-associated transcript expression protects trigeminal ganglion neurons from apoptosis.Antibody against extracellular vaccinia virus (EV) protects mice through complement and Fc receptors.Prophylactic and therapeutic modulation of innate and adaptive immunity against mucosal infection of herpes simplex virus Complement and viral pathogenesis Herpes simplex virus vectors elicit durable immune responses in the presence of preexisting host immunity.The Vaccinia virus complement control protein modulates adaptive immune responses during infection Complement-activating IgM enhances the humoral but not the T cell immune response in mice.Effects of innate immunity on herpes simplex virus and its ability to kill tumor cells.Local delivery of CpG oligodeoxynucleotides induces rapid changes in the genital mucosa and inhibits replication, but not entry, of herpes simplex virus type 2.Complement-Opsonized HIV-1 Overcomes Restriction in Dendritic Cells.The complement system in regulation of adaptive immunity.A protective role for innate immunity in systemic lupus erythematosus.Complement-dependent transport of antigen into B cell follicles.Protective T cell-independent antiviral antibody responses are dependent on complement.Specific IgM and Regulation of Antibody Responses.T cell-independent and T cell-dependent immunoglobulin G responses to polyomavirus infection are impaired in complement receptor 2-deficient mice.Prophylactic vaccine strategies and the potential of therapeutic vaccines against herpes simplex virus Complement receptors CD21 and CD35 in humoral immunity.Tumor genotype determines susceptibility to oncolytic herpes simplex virus mutants: strategies for clinical application.Vaccinia virus extracellular enveloped virion neutralization in vitro and protection in vivo depend on complement Blocking immune evasion as a novel approach for prevention and treatment of herpes simplex virus infection Complement component 3 deficiency prolongs MHC-II disparate skin allograft survival by increasing the CD4(+) CD25(+) regulatory T cells population.Kinetic analysis of the interactions between vaccinia virus complement control protein and human complement proteins C3b and C4b.Immune responses to herpesviral vectors.Immunization with HSV-1 glycoprotein C prevents immune evasion from complement and enhances the efficacy of an HSV-1 glycoprotein D subunit vaccine Heavily isotype-dependent protective activities of human antibodies against vaccinia virus extracellular virion antigen B5.Complement and humoral immunity.Herpes simplex virus as a tool to define the role of complement in the immune response to peripheral infection.The complement system: a gateway to gene-environment interactions in schizophrenia pathogenesis.The Neonatal Fc Receptor and Complement Fixation Facilitate Prophylactic Vaccine-Mediated Humoral Protection against Viral Infection in the Ocular Mucosa.Let's Tie the Knot: Marriage of Complement and Adaptive Immunity in Pathogen Evasion, for Better or Worse.

P2860

Q27469834-7BAE893A-06B3-4DAE-B0D4-2DECEF12F075 Q27477691-792E2F4E-B68D-4AF2-B933-C2EA1F3962A2 Q27482651-4DC45464-66AC-4E18-8BA7-870A3C04FBC7 Q28204656-C32E2935-8EFB-4050-914E-45D4B92441F3 Q29615484-DE1F43F8-B840-4900-8189-A0892B26DDA9 Q30489801-6F9BA2E3-DC5A-403B-88A2-E1BA06E52031 Q33396210-6817C96E-A34C-4422-88D9-4F42F2CD237E Q33818860-8C566534-08F0-4AF0-8237-45CE180220C1 Q33843902-F64E98F0-EFCA-490B-83C9-FC72410B4CCE Q33884066-679B4764-C4D0-4BDC-A870-099A020A8FE0 Q33936717-42F92840-4C90-46C8-A838-C640706AA99F Q34105712-63EF65F9-38C8-49BE-ADDF-A01935501F98 Q34162421-EDD25778-7765-4FBF-8995-36E86FB2B665 Q34335296-5C635C9D-0BCD-4B6B-927F-5446CF43FDA6 Q34742926-87211130-F683-4F34-86BE-3C780FB4FF34 Q35046710-B1394629-54F2-4C2D-B9C3-0E59D6802AFA Q35131235-B9571FFB-FB0C-4A10-A247-A8366E8DF1F6 Q35182216-F458005C-18DE-4ACF-8959-7BB14195E009 Q35677445-41EFDA78-88E3-4101-83E6-EC34FDBE8288 Q35904390-99DDFF29-8982-47E8-A259-9666FD709691 Q35906102-66A8112A-7969-4083-8F91-5F95DEA73431 Q36337689-AF2C864E-0626-4B07-9208-859909A63E94 Q36375462-7551F876-6461-4996-A5E3-3652C35BC87B Q36413052-EC880054-E1DF-4975-93F4-6932B2504428 Q36467689-F1A89CF5-B9A0-47E2-93E2-FF65D141A04A Q36879345-AFB44EE9-FAEE-4477-9D85-96011BACBE5D Q36938627-492DC2F4-060F-49A5-B2C8-A982FE4AF8EF Q36967306-1A7D998D-D538-42A2-969C-478266167281 Q37051617-62F2154D-BC1B-445F-B2CA-6D762910B8BF Q37060288-071C5E99-C91B-4110-977D-428F73A42D6B Q37264666-5A978002-5F98-4EE9-BA00-104B295A0732 Q37348541-AF97EF37-E724-4972-9027-872652530D32 Q37392882-B6083EE4-9143-4653-90CE-B02E13D3E868 Q37439603-70FBD259-16D6-4500-9CBA-1313C81D0FF3 Q37452067-A61B33B9-16DD-4054-A246-77C0A3D66E84 Q37456601-4D601272-C7C0-49C2-A197-AA4D51A58261 Q37456642-AE47665C-0838-466E-979E-2B37D6330E23 Q38650723-4B379EB7-33B3-43AA-ADDC-1C30EA326708 Q38650844-F482AE4A-2917-4315-9727-17084E985281 Q38757564-B2152E77-1EFF-4184-96E8-E5C0D3AC5EB2

P2860

Humoral response to herpes simplex virus is complement-dependent.

http://www.wikidata.org/entity/Q36552488

description

1999 nî lūn-bûn

@nan

1999年の論文

@ja

1999年論文

@yue

1999年論文

@zh-hant

1999年論文

@zh-hk

1999年論文

@zh-mo

1999年論文

@zh-tw

1999年论文

@wuu

1999年论文

@zh

1999年论文

@zh-cn

name

Humoral response to herpes simplex virus is complement-dependent.

@ast

Humoral response to herpes simplex virus is complement-dependent.

@en

type

label

Humoral response to herpes simplex virus is complement-dependent.

@ast

Humoral response to herpes simplex virus is complement-dependent.

@en

prefLabel

Humoral response to herpes simplex virus is complement-dependent.

@ast

Humoral response to herpes simplex virus is complement-dependent.

@en

P1433

Q36552488-B4E109BE-85C8-4C2A-8A5A-03E4E7995FEA

P1476

Q36552488-DB19397C-9C83-402F-85C9-4F697FD491C6

P2093

Q36552488-10428D77-E532-4392-B8DD-337EEC481CA0

Q36552488-2CCF4F4D-E5D9-4781-86AB-FD3EA15EFB79

Q36552488-4A9A6FA0-BB55-496B-BF95-620C24EA5CD6

Q36552488-AF152929-15F7-4346-835D-8CAB2E613684

Q36552488-C5746139-DE2D-4C54-A62C-480ADECCFBDE

Q36552488-C79F8000-98A7-4AF4-88FA-81DCBE14242F

Q36552488-E9B50E32-99D1-4AC2-94EE-E7B1EE15805D

P2860

Q36552488-04D69822-C192-4E8F-9249-EDC5080EEB63

Q36552488-06DBC6B8-14E4-4F2E-80AB-B289287A8C86

Q36552488-0720E344-B7F4-4183-9F79-3D874739BBEF

Q36552488-15F52FA1-D27A-4E75-8B67-4EA79F643225

Q36552488-16BB6C04-A157-4DE4-9BF6-CB86923D7E83

Q36552488-18E234E0-5765-4269-A803-93177ADED435

Q36552488-1C911193-4BBC-469D-AB42-1A06548FBD66

Q36552488-24826B08-6E80-409E-888E-DEE3D37BEBA1

Q36552488-26EAE47C-9519-46C6-8AE7-13F9717E0253

Q36552488-2C1B493B-FDFF-4230-9DBE-33FD053CB6E0

P304

Q36552488-AE15A20F-2597-4511-BEFD-70DE5AFA4C57

P31

Q36552488-D212EFBC-58C8-4C71-87F3-B591E71B4DF9

P356

Q36552488-B7584947-6C93-4AA9-9D61-83F97497BC6D

P407

Q36552488-D33437A2-D652-4704-BAE7-3AF2EBCC7F8C

P433

Q36552488-343B907D-F9F7-4EF9-8362-115884D7B740

P478

Q36552488-327B0C84-82EC-45B7-BCF2-B9CFB5DE5E86

P50

Q36552488-D00DF9FC-D5EC-43F0-BD68-689D38E011D8

P577

Q36552488-C298374D-B479-4936-99A5-7C4970520B0A

P5875

Q36552488-6B2A0718-018E-4581-B617-0867762C14D2

P698

Q36552488-E9FAE8C4-7605-415B-954C-BF676D4B4897

P932

Q36552488-F995062B-74CA-4597-AF08-C0EDE28D7D2E

P356

PNAS.96.22.12708

P1433

Proceedings of the National Academy of Sciences of the United States of America

P1476

Humoral response to herpes simplex virus is complement-dependent.

@en

P2093

D M Knipe

http://www.w3.org/2001/XMLSchema#string

E Alicot

http://www.w3.org/2001/XMLSchema#string

M B Fischer

http://www.w3.org/2001/XMLSchema#string

M C Carroll

http://www.w3.org/2001/XMLSchema#string

M Ma

http://www.w3.org/2001/XMLSchema#string

X J Da Costa

http://www.w3.org/2001/XMLSchema#string

X Zhou

http://www.w3.org/2001/XMLSchema#string

P2860

CD19: lowering the threshold for antigen receptor stimulation of B lymphocytes

Intersection of the complement and immune systems: a signal transduction complex of the B lymphocyte-containing complement receptor type 2 and CD19

Herpes simplex virus glycoproteins gC-1 and gC-2 bind to the third component of complement and provide protection against complement-mediated neutralization of viral infectivity

Local opsonization by secreted macrophage complement components. Role of receptors for complement in uptake of zymosan

The CD19/CD21 signal transducing complex of human B lymphocytes includes the target of antiproliferative antibody-1 and Leu-13 molecules

C3d of complement as a molecular adjuvant: bridging innate and acquired immunity

Molecular organization and function of the complement system

Germinal centers

Studies of group B streptococcal infection in mice deficient in complement component C3 or C4 demonstrate an essential role for complement in both innate and acquired immunity.

Herpes simplex virus type 1 glycoprotein gC mediates immune evasion in vivo

P304

12708-12712

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1073/PNAS.96.22.12708

http://www.w3.org/2001/XMLSchema#string

P407

P433

22

http://www.w3.org/2001/XMLSchema#string

P478

96

http://www.w3.org/2001/XMLSchema#string

P50

Mark A. Brockman

P577

1999-10-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

12764640

http://www.w3.org/2001/XMLSchema#string

P698

10535987

http://www.w3.org/2001/XMLSchema#string

P932

23060

http://www.w3.org/2001/XMLSchema#string