All-trans retinoic acid and rapamycin normalize Hutchinson Gilford progeria fibroblast phenotype.
about
Molecular insights into the premature aging disease progeriaPotential therapeutic effects of the MTOR inhibitors for preventing ageing and progeria-related disordersTemsirolimus Partially Rescues the Hutchinson-Gilford Progeria Cellular PhenotypeIdentification of Salvia haenkei as gerosuppressant agent by using an integrated senescence-screening assayBarrier-to-autointegration factor (BAF) involvement in prelamin A-related chromatin organization changes.A High Throughput Phenotypic Screening reveals compounds that counteract premature osteogenic differentiation of HGPS iPS-derived mesenchymal stem cells.Effects of bioactive compounds on senescence and components of senescence associated secretory phenotypes in vitro.The nuclear lamina in health and disease.Hutchinson-Gilford Progeria Syndrome: A premature aging disease caused by LMNA gene mutations.The telomeric protein AKTIP interacts with A- and B-type lamins and is involved in regulation of cellular senescence.Vitamin D receptor signaling improves Hutchinson-Gilford progeria syndrome cellular phenotypes.Aged induced pluripotent stem cell (iPSCs) as a new cellular model for studying premature aging.Metformin alleviates ageing cellular phenotypes in Hutchinson-Gilford progeria syndrome dermal fibroblasts.Emerging candidate treatment strategies for Hutchinson-Gilford progeria syndrome.A Cell-Intrinsic Interferon-like Response Links Replication Stress to Cellular Aging Caused by Progerin.Autophagic Removal of Farnesylated Carboxy-Terminal Lamin Peptides.Causes and consequences of genomic instability in laminopathies: replication stress and interferon response.An overview of treatment strategies for Hutchinson-Gilford Progeria syndrome.Progerin phosphorylation in interphase is lower and less mechanosensitive than lamin-A,C in iPS-derived mesenchymal stem cells.Pathological modelling of pigmentation disorders associated with Hutchinson-Gilford Progeria Syndrome (HGPS) revealed an impaired melanogenesis pathway in iPS-derived melanocytes.
P2860
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P2860
All-trans retinoic acid and rapamycin normalize Hutchinson Gilford progeria fibroblast phenotype.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
All-trans retinoic acid and ra ...... progeria fibroblast phenotype.
@ast
All-trans retinoic acid and ra ...... progeria fibroblast phenotype.
@en
type
label
All-trans retinoic acid and ra ...... progeria fibroblast phenotype.
@ast
All-trans retinoic acid and ra ...... progeria fibroblast phenotype.
@en
prefLabel
All-trans retinoic acid and ra ...... progeria fibroblast phenotype.
@ast
All-trans retinoic acid and ra ...... progeria fibroblast phenotype.
@en
P2860
P50
P356
P1433
P1476
All-trans retinoic acid and ra ...... progeria fibroblast phenotype
@en
P2093
Maria Rosaria D'Apice
Michela Murdocca
P2860
P304
29914-29928
P356
10.18632/ONCOTARGET.4939
P407
P50
P577
2015-10-01T00:00:00Z