Maternal infection leads to abnormal gene regulation and brain atrophy in mouse offspring: implications for genesis of neurodevelopmental disorders - Wikidata - awgldk - TriplyDB

Maternal infection leads to abnormal gene regulation and brain atrophy in mouse offspring: implications for genesis of neurodevelopmental disorders

Maternal infection leads to abnormal gene regulation and brain atrophy in mouse offspring: implications for genesis of neurodevelopmental disorders

http://www.wikidata.org/entity/Q36559730

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The neurodevelopmental hypothesis of schizophrenia, revisited What are the Best Animal Models for Testing Early Intervention in Cerebral Palsy?Impact of prenatal environmental stress on cortical development Neuroinflammation in Autism: Plausible Role of Maternal Inflammation, Dietary Omega 3, and Microbiota Neurodevelopmental animal models of schizophrenia: role in novel drug discovery and development Maternal immune activation causes age- and region-specific changes in brain cytokines in offspring throughout development.Pre-clinical models of neurodevelopmental disorders: focus on the cerebellum Epigenetic modifications of GABAergic interneurons are associated with the schizophrenia-like phenotype induced by prenatal stress in mice.Viral infection, inflammation and schizophrenia Neonatal behavioral changes in rats with gestational exposure to lipopolysaccharide: a prenatal infection model for developmental neuropsychiatric disorders.Effect of maternal lipopolysaccharide administration on the development of dopaminergic receptors and transporter in the rat offspring.The role of tumor necrosis factor receptor superfamily members in mammalian brain development, function and homeostasis The Long and the Short of it: Gene and Environment Interactions During Early Cortical Development and Consequences for Long-Term Neurological Disease.Prenatal stress: role in psychotic and depressive diseases.Contribution of nonprimate animal models in understanding the etiology of schizophrenia.Review of pathological hallmarks of schizophrenia: comparison of genetic models with patients and nongenetic models Imaging phenotypes of major depressive disorder: genetic correlates Animal models of virus-induced neurobehavioral sequelae: recent advances, methodological issues, and future prospects.Prenatal immune challenge is an environmental risk factor for brain and behavior change relevant to schizophrenia: evidence from MRI in a mouse model Maternal immune activation promotes hippocampal kindling epileptogenesis in mice.Genome-Wide Transcriptional Profiling and Structural Magnetic Resonance Imaging in the Maternal Immune Activation Model of Neurodevelopmental Disorders.Regulation of gap junction channels by infectious agents and inflammation in the CNS.Evolution of structural abnormalities in the rat brain following in utero exposure to maternal immune activation: A longitudinal in vivo MRI study.mRNA and protein levels for GABAAalpha4, alpha5, beta1 and GABABR1 receptors are altered in brains from subjects with autism Maternal immune activation and autism spectrum disorder: interleukin-6 signaling as a key mechanistic pathway The timing and specificity of prenatal immune risk factors for autism modeled in the mouse and relevance to schizophrenia.Prenatal maternal immune activation causes epigenetic differences in adolescent mouse brain.Epigenetic signaling in schizophrenia Peptide sharing between influenza A H1N1 hemagglutinin and human axon guidance proteins.Decreased cortical serotonin in neonatal rabbits exposed to endotoxin in utero Late prenatal immune activation in mice leads to behavioral and neurochemical abnormalities relevant to the negative symptoms of schizophrenia.Novel roles for immune molecules in neural development: implications for neurodevelopmental disorders.Schizophrenia susceptibility genes directly implicated in the life cycles of pathogens: cytomegalovirus, influenza, herpes simplex, rubella, and Toxoplasma gondii.Foxc1 dependent mesenchymal signalling drives embryonic cerebellar growth.Gene expression profiling in rodent models for schizophrenia.Alterations of white matter integrity related to the season of birth in schizophrenia: a DTI study.Abnormalities of the corpus callosum in non-psychotic high-risk offspring of schizophrenia patients Effect of paliperidone and risperidone on extracellular glutamate in the prefrontal cortex of rats exposed to prenatal immune activation or MK-801.The viral theory of schizophrenia revisited: abnormal placental gene expression and structural changes with lack of evidence for H1N1 viral presence in placentae of infected mice or brains of exposed offspring.Risperidone administered during asymptomatic period of adolescence prevents the emergence of brain structural pathology and behavioral abnormalities in an animal model of schizophrenia.

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Maternal infection leads to abnormal gene regulation and brain atrophy in mouse offspring: implications for genesis of neurodevelopmental disorders

http://www.wikidata.org/entity/Q36559730

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J.SCHRES.2007.11.018

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Schizophrenia Research

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Christine Winter

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David A Pearce

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Georg Juckel

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Hao Huang

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Kenichi Oishi

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Reinhard Sohr

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S Hossein Fatemi

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Teri J Reutiman

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Timothy D Folsom

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P2860

The teratology of autism

Altered neuregulin 1-erbB4 signaling contributes to NMDA receptor hypofunction in schizophrenia

The SPCH1 region on human 7q31: genomic characterization of the critical interval and localization of translocations associated with speech and language disorder

Role of numb in dendritic spine development with a Cdc42 GEF intersectin and EphB2.

RIG-I-mediated antiviral responses to single-stranded RNA bearing 5'-phosphates

The axonal chemorepellant semaphorin 3A is increased in the cerebellum in schizophrenia and may contribute to its synaptic pathology

Human studies of prepulse inhibition of startle: normal subjects, patient groups, and pharmacological studies

Autistic disorder and viral infections

An extended family with a dominantly inherited speech disorder

The neurodevelopmental impact of prenatal infections at different times of pregnancy: the earlier the worse?

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10.1016/J.SCHRES.2007.11.018

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neurodevelopmental disorders

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