Glucocorticoid-stimulated CCAAT/enhancer-binding protein alpha expression is required for steroid-induced G1 cell cycle arrest of minimal-deviation rat hepatoma cells.
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The CCAAT enhancer-binding protein alpha (C/EBPalpha) requires a SWI/SNF complex for proliferation arrestCCAAT/enhancer binding protein alpha uses distinct domains to prolong pituitary cells in the growth 1 and DNA synthesis phases of the cell cycleCell cycle and hormonal control of nuclear-cytoplasmic localization of the serum- and glucocorticoid-inducible protein kinase, Sgk, in mammary tumor cells. A novel convergence point of anti-proliferative and proliferative cell signaling pathwaysThe C/EBP family of transcription factors in the liver and other organs.Distinct glucocorticoid receptor transcriptional regulatory surfaces mediate the cytotoxic and cytostatic effects of glucocorticoidsC/EBP transcription factors in the lung epithelium.CCAAT/enhancer binding protein alpha regulates p21 protein and hepatocyte proliferation in newborn mice.CCAAT enhancer-binding protein alpha is a molecular target of 1,25-dihydroxyvitamin D3 in MCF-7 breast cancer cells.Dexamethasone treatment of calves latently infected with bovine herpesvirus 1 leads to activation of the bICP0 early promoter, in part by the cellular transcription factor C/EBP-alphaTandem repeat of C/EBP binding sites mediates PPARgamma2 gene transcription in glucocorticoid-induced adipocyte differentiation.Glucocorticoid-Induced Leucine Zipper in Central Nervous System Health and Disease.Among translational effectors, p70S6k is uniquely sensitive to inhibition by glucocorticoids.CCAAT/enhancer-binding protein-delta (C/EBP-delta) is induced in growth-arrested mouse mammary epithelial cells.Signaling pathways through which insulin regulates CCAAT/enhancer binding protein alpha (C/EBPalpha) phosphorylation and gene expression in 3T3-L1 adipocytes. Correlation with GLUT4 gene expression.Factors affecting adipose tissue development in chickens: A review.C/EBP regulates hepatic transcription of 11beta -hydroxysteroid dehydrogenase type 1. A novel mechanism for cross-talk between the C/EBP and glucocorticoid signaling pathways.Human chorionic gonadotropin-alpha gene is transcriptionally activated by epidermal growth factor through cAMP response element in trophoblast cells.
P2860
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P2860
Glucocorticoid-stimulated CCAAT/enhancer-binding protein alpha expression is required for steroid-induced G1 cell cycle arrest of minimal-deviation rat hepatoma cells.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
1996年论文
@zh
1996年论文
@zh-cn
name
Glucocorticoid-stimulated CCAA ...... -deviation rat hepatoma cells.
@ast
Glucocorticoid-stimulated CCAA ...... -deviation rat hepatoma cells.
@en
type
label
Glucocorticoid-stimulated CCAA ...... -deviation rat hepatoma cells.
@ast
Glucocorticoid-stimulated CCAA ...... -deviation rat hepatoma cells.
@en
prefLabel
Glucocorticoid-stimulated CCAA ...... -deviation rat hepatoma cells.
@ast
Glucocorticoid-stimulated CCAA ...... -deviation rat hepatoma cells.
@en
P2093
P2860
P356
P1476
Glucocorticoid-stimulated CCAA ...... -deviation rat hepatoma cells.
@en
P2093
A C Maiyar
C C Ridder
G L Firestone
P2860
P304
P356
10.1128/MCB.16.10.5288
P407
P577
1996-10-01T00:00:00Z