mVps34 deletion in podocytes causes glomerulosclerosis by disrupting intracellular vesicle trafficking.
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PodocytesAutophagy: A Novel Therapeutic Target for Diabetic NephropathyPodocyte endocytosis in the regulation of the glomerular filtration barrierThe function of endocytosis in podocytesRegulation of autophagy by TGF-β: emerging role in kidney fibrosisPodocyte-associated talin1 is critical for glomerular filtration barrier maintenance.Podocytes degrade endocytosed albumin primarily in lysosomesPhosphorylation of ribosomal protein S6 mediates compensatory renal hypertrophyClass III PI 3-kinase is the main source of PtdIns3P substrate and membrane recruitment signal for PIKfyve constitutive function in podocyte endomembrane homeostasisAlbumin-associated free fatty acids induce macropinocytosis in podocytes.Expression of ODC Antizyme Inhibitor 2 (AZIN2) in Human Secretory Cells and TissuesBlocking rpS6 Phosphorylation Exacerbates Tsc1 Deletion-Induced Kidney GrowthLysosome dysfunction in the pathogenesis of kidney diseases.Mechanisms and biological functions of autophagy in diseased and ageing kidneys.Causes and pathogenesis of focal segmental glomerulosclerosis.Autophagy in renal tubular injury and repair.Autophagy and kidney inflammation.Distinct Requirements for Vacuolar Protein Sorting 34 Downstream Effector Phosphatidylinositol 3-Phosphate 5-Kinase in Podocytes Versus Proximal Tubular Cells.Cathepsin D in Podocytes Is Important in the Pathogenesis of Proteinuria and CKD.Phosphatidylinositol 3-kinase signaling determines kidney size.aPKCλ maintains the integrity of the glomerular slit diaphragm through trafficking of nephrin to the cell surface.Distinct functions of Crumbs regulating slit diaphragms and endocytosis in Drosophila nephrocytes.Autophagy in diabetic kidney disease: regulation, pathological role and therapeutic potential.APOL1-Mediated Cell Injury Involves Disruption of Conserved Trafficking Processes.Involvement of Caveolin-1 in CD83 Internalization in Mouse Dendritic Cells.Class III phosphoinositide 3-kinase/VPS34 and dynamin are critical for apical endocytic recycling.Deficiency in class III PI3-kinase confers postnatal lethality with IBD-like features in zebrafish.
P2860
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P2860
mVps34 deletion in podocytes causes glomerulosclerosis by disrupting intracellular vesicle trafficking.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
mVps34 deletion in podocytes c ...... acellular vesicle trafficking.
@ast
mVps34 deletion in podocytes c ...... acellular vesicle trafficking.
@en
type
label
mVps34 deletion in podocytes c ...... acellular vesicle trafficking.
@ast
mVps34 deletion in podocytes c ...... acellular vesicle trafficking.
@en
prefLabel
mVps34 deletion in podocytes c ...... acellular vesicle trafficking.
@ast
mVps34 deletion in podocytes c ...... acellular vesicle trafficking.
@en
P2093
P2860
P356
P1476
mVps34 deletion in podocytes c ...... acellular vesicle trafficking.
@en
P2093
Agnes B Fogo
Jian-Kang Chen
Jianchun Chen
Mystie X Chen
Raymond C Harris
P2860
P304
P356
10.1681/ASN.2012010101
P577
2013-01-04T00:00:00Z