mVps34 deletion in podocytes causes glomerulosclerosis by disrupting intracellular vesicle trafficking. - Wikidata - awgldk - TriplyDB

mVps34 deletion in podocytes causes glomerulosclerosis by disrupting intracellular vesicle trafficking.

mVps34 deletion in podocytes causes glomerulosclerosis by disrupting intracellular vesicle trafficking.

http://www.wikidata.org/entity/Q36574743

about

Podocytes Autophagy: A Novel Therapeutic Target for Diabetic Nephropathy Podocyte endocytosis in the regulation of the glomerular filtration barrier The function of endocytosis in podocytes Regulation of autophagy by TGF-β: emerging role in kidney fibrosis Podocyte-associated talin1 is critical for glomerular filtration barrier maintenance.Podocytes degrade endocytosed albumin primarily in lysosomes Phosphorylation of ribosomal protein S6 mediates compensatory renal hypertrophy Class III PI 3-kinase is the main source of PtdIns3P substrate and membrane recruitment signal for PIKfyve constitutive function in podocyte endomembrane homeostasis Albumin-associated free fatty acids induce macropinocytosis in podocytes.Expression of ODC Antizyme Inhibitor 2 (AZIN2) in Human Secretory Cells and Tissues Blocking rpS6 Phosphorylation Exacerbates Tsc1 Deletion-Induced Kidney Growth Lysosome dysfunction in the pathogenesis of kidney diseases.Mechanisms and biological functions of autophagy in diseased and ageing kidneys.Causes and pathogenesis of focal segmental glomerulosclerosis.Autophagy in renal tubular injury and repair.Autophagy and kidney inflammation.Distinct Requirements for Vacuolar Protein Sorting 34 Downstream Effector Phosphatidylinositol 3-Phosphate 5-Kinase in Podocytes Versus Proximal Tubular Cells.Cathepsin D in Podocytes Is Important in the Pathogenesis of Proteinuria and CKD.Phosphatidylinositol 3-kinase signaling determines kidney size.aPKCλ maintains the integrity of the glomerular slit diaphragm through trafficking of nephrin to the cell surface.Distinct functions of Crumbs regulating slit diaphragms and endocytosis in Drosophila nephrocytes.Autophagy in diabetic kidney disease: regulation, pathological role and therapeutic potential.APOL1-Mediated Cell Injury Involves Disruption of Conserved Trafficking Processes.Involvement of Caveolin-1 in CD83 Internalization in Mouse Dendritic Cells.Class III phosphoinositide 3-kinase/VPS34 and dynamin are critical for apical endocytic recycling.Deficiency in class III PI3-kinase confers postnatal lethality with IBD-like features in zebrafish.

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P2860

mVps34 deletion in podocytes causes glomerulosclerosis by disrupting intracellular vesicle trafficking.

http://www.wikidata.org/entity/Q36574743

description

2013 nî lūn-bûn

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

@zh-hk

2013年論文

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2013年論文

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2013年论文

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2013年论文

@zh

2013年论文

@zh-cn

name

mVps34 deletion in podocytes c ...... acellular vesicle trafficking.

@ast

mVps34 deletion in podocytes c ...... acellular vesicle trafficking.

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type

label

mVps34 deletion in podocytes c ...... acellular vesicle trafficking.

@ast

mVps34 deletion in podocytes c ...... acellular vesicle trafficking.

@en

prefLabel

mVps34 deletion in podocytes c ...... acellular vesicle trafficking.

@ast

mVps34 deletion in podocytes c ...... acellular vesicle trafficking.

@en

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Journal of the American Society of Nephrology

P1476

mVps34 deletion in podocytes c ...... acellular vesicle trafficking.

@en

P2093

Agnes B Fogo

http://www.w3.org/2001/XMLSchema#string

Jian-Kang Chen

http://www.w3.org/2001/XMLSchema#string

Jianchun Chen

http://www.w3.org/2001/XMLSchema#string

Mystie X Chen

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Raymond C Harris

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P2860

Two Beclin 1-binding proteins, Atg14L and Rubicon, reciprocally regulate autophagy at different stages

A human phosphatidylinositol 3-kinase complex related to the yeast Vps34p-Vps15p protein sorting system

Distinct regulation of autophagic activity by Atg14L and Rubicon associated with Beclin 1-phosphatidylinositol-3-kinase complex

Dissection of autophagosome formation using Apg5-deficient mouse embryonic stem cells

Phosphatidylinositol 3-kinase encoded by yeast VPS34 gene essential for protein sorting.

Autophagy: process and function

The evolution of phosphatidylinositol 3-kinases as regulators of growth and metabolism

Autophagy as a regulated pathway of cellular degradation

Beclin 1 forms two distinct phosphatidylinositol 3-kinase complexes with mammalian Atg14 and UVRAG

High-efficiency deleter mice show that FLPe is an alternative to Cre-loxP

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3559479

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