Iron accumulation in Alzheimer disease is a source of redox-generated free radicals. - Wikidata - awgldk - TriplyDB

Iron accumulation in Alzheimer disease is a source of redox-generated free radicals.

Iron accumulation in Alzheimer disease is a source of redox-generated free radicals.

http://www.wikidata.org/entity/Q36577461

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Retinal iron homeostasis in health and disease Iron and neurodegeneration: from cellular homeostasis to disease Oxidative Stress Signaling in Alzheimers Disease Towards a unifying, systems biology understanding of large-scale cellular death and destruction caused by poorly liganded iron: Parkinson's, Huntington's, Alzheimer's, prions, bactericides, chemical toxicology and others as examples Iron-export ferroxidase activity of β-amyloid precursor protein is inhibited by zinc in Alzheimer's disease Histological co-localization of iron in Abeta plaques of PS/APP transgenic mice Pathways towards and away from Alzheimer's disease Assessment of Alzheimer's disease case-control associations using family-based methods Metals and amyloid-beta in Alzheimer's disease Down-regulation of aminolevulinate synthase, the rate-limiting enzyme for heme biosynthesis in Alzheimer's disease Fenton chemistry and oxidative stress mediate the toxicity of the beta-amyloid peptide in a Drosophila model of Alzheimer's disease Alzheimer Disease and Oxidative Stress Linking multiple pathogenic pathways in Alzheimer's disease Metals and Neuronal Metal Binding Proteins Implicated in Alzheimer's Disease Environmental pollutants as risk factors for neurodegenerative disorders: Alzheimer and Parkinson diseases The Janus face of the heme oxygenase/biliverdin reductase system in Alzheimer disease: it's time for reconciliation Common defects of mitochondria and iron in neurodegeneration and diabetes (MIND): a paradigm worth exploring Iron chelation and multiple sclerosis Elevated risk of type 2 diabetes for development of Alzheimer disease: a key role for oxidative stress in brain Bridging the gap between statistical and biological epistasis in Alzheimer's disease Iron homeostasis and toxicity in retinal degeneration Evidence for the Influence of the Iron Regulatory MHC Class I Molecule HFE on Tumor Progression in Experimental Models and Clinical Populations Iron behaving badly: inappropriate iron chelation as a major contributor to the aetiology of vascular and other progressive inflammatory and degenerative diseases Mechanisms of oxidative damage in multiple sclerosis and neurodegenerative diseases: therapeutic modulation via fumaric acid esters Iron deficiency alters expression of genes implicated in Alzheimer disease pathogenesis Cellular and biochemical actions of melatonin which protect against free radicals: role in neurodegenerative disorders Aceruloplasminemia With Psychomotor Excitement and Neurological Sign Was Improved by Minocycline (Case Report)Nanoscale biogenic iron oxides and neurodegenerative disease.In vivo quantitative susceptibility mapping (QSM) in Alzheimer's disease The presence and role of iron in mild traumatic brain injury: an imaging perspective.Ex vivo T2 relaxation: associations with age-related neuropathology and cognition Membrane-inserted conformation of transmembrane domain 4 of divalent-metal transporter.Identification of iron responsive genes by screening cDNA libraries from suppression subtractive hybridization with antisense probes from three iron conditions.The relationship between iron dyshomeostasis and amyloidogenesis in Alzheimer's disease: Two sides of the same coin.Expression and localization of mitochondrial ferritin mRNA in Alzheimer's disease cerebral cortex.Molecular mechanisms for Alzheimer's disease: implications for neuroimaging and therapeutics.Iron accumulation in deep cortical layers accounts for MRI signal abnormalities in ALS: correlating 7 tesla MRI and pathology.Homocysteine potentiates copper- and amyloid beta peptide-mediated toxicity in primary neuronal cultures: possible risk factors in the Alzheimer's-type neurodegenerative pathways.Clues to neuro-degeneration in Niemann-Pick type C disease from global gene expression profiling Genes involved in the balance between neuronal survival and death during inflammation

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P2860

Iron accumulation in Alzheimer disease is a source of redox-generated free radicals.

http://www.wikidata.org/entity/Q36577461

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1997 nî lūn-bûn

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1997年論文

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1997年論文

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1997年論文

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1997年論文

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1997年论文

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1997年论文

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1997年论文

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Iron accumulation in Alzheimer disease is a source of redox-generated free radicals.

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Iron accumulation in Alzheimer disease is a source of redox-generated free radicals.

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Iron accumulation in Alzheimer disease is a source of redox-generated free radicals.

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Iron accumulation in Alzheimer disease is a source of redox-generated free radicals.

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Iron accumulation in Alzheimer disease is a source of redox-generated free radicals.

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PNAS.94.18.9866

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Proceedings of the National Academy of Sciences of the United States of America

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Iron accumulation in Alzheimer disease is a source of redox-generated free radicals.

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Harris PL

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Sayre LM

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P2860

Selective accumulation of aluminum and iron in the neurofibrillary tangles of Alzheimer's disease: a laser microprobe (LAMMA) study.

Immunohistochemical evidence of oxidative [corrected] stress in Alzheimer's disease.

Lactotransferrin immunocytochemistry in Alzheimer and normal human brain.

Heme oxygenase-1 is associated with the neurofibrillary pathology of Alzheimer's disease.

Excess brain protein oxidation and enzyme dysfunction in normal aging and in Alzheimer disease.

Radical AGEing in Alzheimer's disease.

Regional distribution of iron and iron-regulatory proteins in the brain in aging and Alzheimer's disease.

Apolipoprotein E allele-specific antioxidant activity and effects on cytotoxicity by oxidative insults and beta-amyloid peptides.

Association of heparan sulfate proteoglycan with the neurofibrillary tangles of Alzheimer's disease.

Chemical modification of glucosamine-6-phosphate synthase by diethyl pyrocarbonate: evidence of histidine requirement for enzymatic activity.

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Alzheimer's disease

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23283

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