Inhibition of protein synthesis in L cells infected with vesicular stomatitis virus.
about
Differential inhibition of host protein synthesis in L cells infected with RNA - temperature-sensitive mutants of vesicular stomatitis virusInhibition of pseudorabies virus replication by vesicular stomatitis virus. II Activity of defective interfering particlesInhibition of pseudorabies virus replication by vesicular stomicles virus I. Activity of infectious and inactivated B particles.Use of UV irradiation to identify the genetic information of vesicular stomatitis virus responsible for shutting off cellular RNA synthesis.Transcription of vesicular stomatitis virus is required to shut off cellular RNA synthesis.Role of temperature-sensitive mutants in persistent infections initiated with vesicular stomatitis virus.Homotypic and heterotypic exclusion of vesicular stomatitis virus replication by high levels of recombinant polymerase protein LComparative inhibition of cellular transcription by vesicular stomatitis virus serotypes New Jersey and Indiana: role of each viral leader RNAInhibition of cellular DNA synthesis by vesicular stomatitis virus.Amino acid residues within conserved domain VI of the vesicular stomatitis virus large polymerase protein essential for mRNA cap methyltransferase activityTemperature-sensitive mutants of vesicular stomatitis virus are conditionally defective particles that interfere with and are rescued by wild-type virus.Selective blockage of initiation of host protein synthesis in RNA-virus-infected cells.Vesicular stomatitis virus infection induces a nuclear DNA-binding factor specific for the interferon-stimulated response element.Effect of viral infection on host protein synthesis and mRNA association with the cytoplasmic cytoskeletal structure.Somatostatin discriminates between the intracellular pathways of secretory and membrane proteins.Alterations in the protein synthetic apparatus of Friend erythroleukemia cells infected with vesicular stomatitis virus or herpes simplex virus.Analysis of uridine incorporation in chicken embryo cells infected by vesicular stomatitis virus and its temperature-sensitive mutants: uridine transportScanning electron microscopic studies of virus-infected cells. I. Cytopathic effects and maturation of vesicular stomatitis virus in L2 cells.Nongenetic complementation of group V temperature-sensitive mutants of vesicular stomatitis virus by UV-irradiated virus.Reovirus: effect of noninfective viral components on cellular deoxyribonucleic acid synthesisIn vitro synthesis of proteins by membrane-bound polyribosomes from vesicular stomatitis virus-infected HeLa cellsEncephalomyocarditis virus infection of mouse plasmacytoma cells. I. Inhibition of cellular protein synthesis.Effect of vesicular stomatitis virus infection on the histocompatibility antigen of L cellsMutant of vesicular stomatitis virus which allows deoxyribonucleic acid synthesis and division in cells synthesizing viral ribonucleic acidTransient inhibition of polyoma virus synthesis by Sendai virus (parainfluenza I). I. Demonstration and nature of the inhibition by inactivated virus.Transient inhibition of polyoma virus synthesis by sendai virus (parainfluenza I). II. Mechanism of the interference by inactivated virusRNA synthesis by vesicular stomatitis virus and a small plaque mutant: effects of cycloheximide.Effect of vesicular stomatitis virus matrix protein on host-directed translation in vivoRapid inhibition of processing and assembly of small nuclear ribonucleoproteins after infection with vesicular stomatitis virus.Translational control of vesicular stomatitis virus protein synthesis: isolation of an mRNA-sequestering particleEffect of intracellular vesicular stomatitis virus mRNA concentration on the inhibition of host cell protein synthesisTwo transcription products of the vesicular stomatitis virus genome may control L-cell protein synthesis.Regulation of protein synthesis in vesicular stomatitis virus-infected mouse L-929 cells by decreased protein synthesis initiation factor 2 activityPreferential translation of vesicular stomatitis virus mRNAs is conferred by transcription from the viral genome.Translation of individual host mRNA's in MPC-11 cells is differentially suppressed after infection by vesicular stomatitis virus.Nucleotide sequence and host La protein interactions of rabies virus leader RNA.New mRNAs are preferentially translated during vesicular stomatitis virus infection.
P2860
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P2860
Inhibition of protein synthesis in L cells infected with vesicular stomatitis virus.
description
1972 nî lūn-bûn
@nan
1972年の論文
@ja
1972年論文
@yue
1972年論文
@zh-hant
1972年論文
@zh-hk
1972年論文
@zh-mo
1972年論文
@zh-tw
1972年论文
@wuu
1972年论文
@zh
1972年论文
@zh-cn
name
Inhibition of protein synthesis in L cells infected with vesicular stomatitis virus.
@ast
Inhibition of protein synthesis in L cells infected with vesicular stomatitis virus.
@en
type
label
Inhibition of protein synthesis in L cells infected with vesicular stomatitis virus.
@ast
Inhibition of protein synthesis in L cells infected with vesicular stomatitis virus.
@en
prefLabel
Inhibition of protein synthesis in L cells infected with vesicular stomatitis virus.
@ast
Inhibition of protein synthesis in L cells infected with vesicular stomatitis virus.
@en
P2860
P1433
P1476
Inhibition of protein synthesis in L cells infected with vesicular stomatitis virus.
@en
P2093
P2860
P407
P577
1972-01-01T00:00:00Z