Diabetic nephropathy: the role of inflammation in fibroblast activation and kidney fibrosis. - Wikidata - awgldk - TriplyDB

Diabetic nephropathy: the role of inflammation in fibroblast activation and kidney fibrosis.

Diabetic nephropathy: the role of inflammation in fibroblast activation and kidney fibrosis.

http://www.wikidata.org/entity/Q36589609

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The role of progranulin in diabetes and kidney disease N-acetyl-seryl-aspartyl-lysyl-proline: a valuable endogenous anti-fibrotic peptide for combating kidney fibrosis in diabetes Impact of heparanase on renal fibrosis Positioning of Tacrolimus for the Treatment of Diabetic Nephropathy Based on Computational Network Analysis Piceatannol Attenuates Renal Fibrosis Induced by Unilateral Ureteral Obstruction via Downregulation of Histone Deacetylase 4/5 or p38-MAPK Signaling Human fibrocytes express multiple antigens associated with autoimmune endocrine diseases.Effect of pentoxifylline on renal function and urinary albumin excretion in patients with diabetic kidney disease: the PREDIAN trial.Curtailing endothelial TGF-β signaling is sufficient to reduce endothelial-mesenchymal transition and fibrosis in CKD The anti-inflammatory and antifibrotic effects of Coreopsis tinctoria Nutt on high-glucose-fat diet and streptozotocin-induced diabetic renal damage in rats.Quantitative non-invasive cell characterisation and discrimination based on multispectral autofluorescence features Heparanase: A Potential New Factor Involved in the Renal Epithelial Mesenchymal Transition (EMT) Induced by Ischemia/Reperfusion (I/R) Injury.Association of Haemostatic and Inflammatory Biomarkers with Nephropathy in Type 1 Diabetes Mellitus Mini-review: diabetic renal complications, a potential stinky remedy.M2 macrophage polarization modulates epithelial-mesenchymal transition in cisplatin-induced tubulointerstitial fibrosis Protective role of propofol on the kidney during early unilateral ureteral obstruction through inhibition of epithelial-mesenchymal transition.The Protective Effect of Beraprost Sodium on Diabetic Nephropathy by Inhibiting Inflammation and p38 MAPK Signaling Pathway in High-Fat Diet/Streptozotocin-Induced Diabetic Rats.Identification of genes and signaling pathways associated with diabetic neuropathy using a weighted correlation network analysis: A consort study.Alpinia oxyphylla Miq. extract changes miRNA expression profiles in db-/db- mouse kidney Diabetic nephropathy: Treatment with phosphodiesterase type 5 inhibitors.Role of Toll-like receptors in diabetic nephropathy.The biological significance of angiotensin-converting enzyme inhibition to combat kidney fibrosis.(+)-Catechin ameliorates diabetic nephropathy by trapping methylglyoxal in type 2 diabetic mice.Synergistic induction of CCL2/MCP-1 expression driven by oncostatin M and IL-1β in human proximal tubular cells depends on STAT3 and p65 NFκB/RelA Oral Administration of N-Acetyl-seryl-aspartyl-lysyl-proline Ameliorates Kidney Disease in Both Type 1 and Type 2 Diabetic Mice via a Therapeutic Regimen.Mori cortex prevents kidney damage through inhibiting expression of inflammatory factors in the glomerulus in streptozocin-induced diabetic rats.Plasma and Urinary Type IV Collagen Levels for Early Detection of Nephropathy in Type 2 Diabetes Mellitus Patients.Effect of Antifibrotic MicroRNAs Crosstalk on the Action of N-acetyl-seryl-aspartyl-lysyl-proline in Diabetes-related Kidney Fibrosis Effect of Puerarin on Expression of ICAM-1 and TNF-α in Kidneys of Diabetic Rats.The role of the immune system in the pathogenesis of diabetic complications Macrophage trafficking as key mediator of adenine-induced kidney injury.Tempol effect on epithelial-mesenchymal transition induced by hyperglycemia.N-acetyl-seryl-aspartyl-lysyl-proline inhibits diabetes-associated kidney fibrosis and endothelial-mesenchymal transition.Vascular Cell Adhesion Molecule 1, Intercellular Adhesion Molecule 1, and Cluster of Differentiation 146 Levels in Patients with Type 2 Diabetes with Complications.Association of Urinary Biomarkers of Inflammation, Injury, and Fibrosis with Renal Function Decline: The ACCORD Trial.1,25(OH)2D3 treatment attenuates high glucose‑induced peritoneal epithelial to mesenchymal transition in mice.Renoprotective Effects of the Dipeptidyl Peptidase-4 Inhibitor Sitagliptin: A Review in Type 2 Diabetes.NF-κβ: A Potential Target in the Management of Vascular Complications of Diabetes.Eucalyptol ameliorates Snail1/β-catenin-dependent diabetic disjunction of renal tubular epithelial cells and tubulointerstitial fibrosis.CERA Attenuates Kidney Fibrogenesis in the db/db Mouse by Influencing the Renal Myofibroblast Generation.Monoamine oxidase-dependent endoplasmic reticulum-mitochondria dysfunction and mast cell degranulation lead to adverse cardiac remodeling in diabetes.

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Diabetic nephropathy: the role of inflammation in fibroblast activation and kidney fibrosis.

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Frontiers in Endocrinology

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Daisuke Koya

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Gangadhar Taduri

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Keizo Kanasaki

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P2860

TGF-beta signaling by Smad proteins

Inhibition of fibrocyte differentiation by serum amyloid P

Epithelial-mesenchymal transition and its implications for fibrosis

Cellular and molecular mechanisms of fibrosis

Galectin-3 expression and secretion links macrophages to the promotion of renal fibrosis

The basics of epithelial-mesenchymal transition

Role of intensive glucose control in development of renal end points in type 2 diabetes mellitus: systematic review and meta-analysis intensive glucose control in type 2 diabetes

Epithelial-mesenchymal transitions in tumour progression

The effect of intensive treatment of diabetes on the development and progression of long-term complications in insulin-dependent diabetes mellitus. The Diabetes Control and Complications Trial Research Group

Transforming growth factor beta in tissue fibrosis

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10.3389/FENDO.2013.00007

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