Torsades de pointes following acute myocardial infarction: evidence for a deadly link with a common genetic variant. - Wikidata - awgldk - TriplyDB

Torsades de pointes following acute myocardial infarction: evidence for a deadly link with a common genetic variant.

Torsades de pointes following acute myocardial infarction: evidence for a deadly link with a common genetic variant.

http://www.wikidata.org/entity/Q36593290

about

Molecular and genetic basis of sudden cardiac death Epidemiology and genetics of ventricular fibrillation during acute myocardial infarction Monogenec Arrhythmic Syndromes: From Molecular and Genetic Aspects to Bedside.Methadone, QTc interval prolongation and torsade de pointes: Case reports offer the best understanding of this problem.A Clinical Perspective on Sudden Cardiac Death.Genetics of sudden cardiac death caused by ventricular arrhythmias.Cardiac arrhythmias in acute coronary syndromes: position paper from the joint EHRA, ACCA, and EAPCI task force.Control of cardiac repolarization by phosphoinositide 3-kinase signaling to ion channels.The evolution of cardiology: changes, future challenges and opportunities.Medico-legal perspectives on sudden cardiac death in young athletes.Nav 1.5 mutations linked to dilated cardiomyopathy phenotypes: Is the gating pore current the missing link?

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P2860

Torsades de pointes following acute myocardial infarction: evidence for a deadly link with a common genetic variant.

http://www.wikidata.org/entity/Q36593290

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2012 nî lūn-bûn

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Torsades de pointes following ...... with a common genetic variant.

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Alejandra Guerchicoff

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P2860

Rac GTPase signaling through the PP5 protein phosphatase

Long-QT syndrome: from genetics to management

Genome-wide association study identifies a susceptibility locus at 21q21 for ventricular fibrillation in acute myocardial infarction

Rapid signaling at the plasma membrane by a nuclear receptor for thyroid hormone

Linkage and mutation analysis in two Taiwanese families with long QT syndrome.

Increased risk of arrhythmic events in long-QT syndrome with mutations in the pore region of the human ether-a-go-go-related gene potassium channel.

Active cascade screening in primary inherited arrhythmia syndromes: does it lead to prophylactic treatment?

A common single nucleotide polymorphism can exacerbate long-QT type 2 syndrome leading to sudden infant death.

The response of the QT interval to the brief tachycardia provoked by standing: a bedside test for diagnosing long QT syndrome.

Family history as a risk factor for primary cardiac arrest.

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