A systems biology approach reveals that tissue tropism to West Nile virus is regulated by antiviral genes and innate immune cellular processes - Wikidata - awgldk - TriplyDB

A systems biology approach reveals that tissue tropism to West Nile virus is regulated by antiviral genes and innate immune cellular processes

A systems biology approach reveals that tissue tropism to West Nile virus is regulated by antiviral genes and innate immune cellular processes

http://www.wikidata.org/entity/Q36594860

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Usutu virus: an emerging flavivirus in Europe Zika Virus Disrupts Phospho-TBK1 Localization and Mitosis in Human Neuroepithelial Stem Cells and Radial Glia Role of natural killer and Gamma-delta T cells in West Nile virus infection Systems analysis of West Nile virus infection Experimental infection of rhesus macaques and common marmosets with a European strain of West Nile virus New insights into innate immune restriction of West Nile virus infection Human genome-wide RNAi screen identifies an essential role for inositol pyrophosphates in Type-I interferon response Zika Virus Antagonizes Type I Interferon Responses during Infection of Human Dendritic Cells Multiplexed digital mRNA profiling of the inflammatory response in the West Nile Swiss Webster mouse model Triggering receptor expressed on myeloid cells-1 (TREM-1): a new player in antiviral immunity?Systems immunology reveals markers of susceptibility to West Nile virus infection.Differentiation of neurons restricts Arbovirus replication and increases expression of the alpha isoform of IRF-7.Innate immune sensing of flaviviruses.Deficient IFN signaling by myeloid cells leads to MAVS-dependent virus-induced sepsis.Induction of virus-specific effector immune cell response limits virus replication and severe disease in mice infected with non-lethal West Nile virus Eg101 strain.Identification of host genes leading to West Nile virus encephalitis in mice brain using RNA-seq analysis.Systems biology: A tool for charting the antiviral landscape.The essential, nonredundant roles of RIG-I and MDA5 in detecting and controlling West Nile virus infection.A Systems Approach Reveals MAVS Signaling in Myeloid Cells as Critical for Resistance to Ebola Virus in Murine Models of Infection.IFN-dependent and -independent reduction in West Nile virus infectivity in human dermal fibroblasts.Using a systems-based approach to overcome reductionist strategies in the development of diagnostics.The innate immune playbook for restricting West Nile virus infection.Current developments in understanding of West Nile virus central nervous system disease.Age-related alterations in immune responses to West Nile virus infection.TLR signaling controls lethal encephalitis in WNV-infected brain.TLR8 Couples SOCS-1 and Restrains TLR7-Mediated Antiviral Immunity, Exacerbating West Nile Virus Infection in Mice.Serum sTREM-1 level is quite higher in Crimean Congo Hemorrhagic Fever, a viral infection.The Epimmunity Theory: The Single Cell Defenses against Infectious and Genetic Diseases.Subcapsular sinus macrophages limit dissemination of West Nile virus particles after inoculation but are not essential for the development of West Nile virus-specific T cell responses.Of Mice and Men: Protective and Pathogenic Immune Responses to West Nile virus Infection.Systems biological analyses reveal the hepatitis C virus (HCV)-specific regulation of hematopoietic development.Interferon regulated gene (IRG) expression-signature in a mouse model of chikungunya virus neurovirulence.Distinct Effects of Type I and III Interferons on Enteric Viruses.Splenic macrophages are required for protective innate immunity against West Nile virus.Host Competence: An Organismal Trait to Integrate Immunology and Epidemiology.Recent advances in understanding West Nile virus host immunity and viral pathogenesis.Interleukin-1β Signaling in Dendritic Cells Induces Antiviral Interferon Responses.Zika Virus Trafficking and Interactions in the Human Male Reproductive Tract.Gene expression profiles in neurological tissues during West Nile virus infection: a critical meta-analysis

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A systems biology approach reveals that tissue tropism to West Nile virus is regulated by antiviral genes and innate immune cellular processes

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Gabriele Blahnik

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Hilario J Ramos

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Mehul S Suthar

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A diverse range of gene products are effectors of the type I interferon antiviral response

Innate immunity induced by composition-dependent RIG-I recognition of hepatitis C virus RNA

The IFITM proteins mediate cellular resistance to influenza A H1N1 virus, West Nile virus, and dengue virus

Alpha/beta interferon protects against lethal West Nile virus infection by restricting cellular tropism and enhancing neuronal survival

CD8+ T Cells Require Perforin To Clear West Nile Virus from Infected Neurons

Gamma Interferon Plays a Crucial Early Antiviral Role in Protection against West Nile Virus Infection

PKR and RNase L Contribute to Protection against Lethal West Nile Virus Infection by Controlling Early Viral Spread in the Periphery and Replication in Neurons

Resistance to Alpha/Beta Interferon Is a Determinant of West Nile Virus Replication Fitness and Virulence

Pathogenesis of West Nile Virus Infection: a Balance between Virulence, Innate and Adaptive Immunity, and Viral Evasion

Coordinated Regulation and Widespread Cellular Expression of Interferon-Stimulated Genes (ISG) ISG-49, ISG-54, and ISG-56 in the Central Nervous System after Infection with Distinct Viruses

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