Bile salt-induced intermolecular disulfide bond formation activates Vibrio cholerae virulence. - Wikidata - awgldk - TriplyDB

Bile salt-induced intermolecular disulfide bond formation activates Vibrio cholerae virulence.

Bile salt-induced intermolecular disulfide bond formation activates Vibrio cholerae virulence.

http://www.wikidata.org/entity/Q36598118

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Biochemical basis for activation of virulence genes by bile salts in Vibrio parahaemolyticus.Bile salt receptor complex activates a pathogenic type III secretion system A Genome-Wide Screen Reveals that the Vibrio cholerae Phosphoenolpyruvate Phosphotransferase System Modulates Virulence Gene Expression Members of the human gut microbiota involved in recovery from Vibrio cholerae infection.Efficient responses to host and bacterial signals during Vibrio cholerae colonization Bile acids and bicarbonate inversely regulate intracellular cyclic di-GMP in Vibrio cholerae.Host intestinal signal-promoted biofilm dispersal induces Vibrio cholerae colonization.Vibrio cholerae represses polysaccharide synthesis to promote motility in mucosa.Dual Zinc Transporter Systems in Vibrio cholerae Promote Competitive Advantages over Gut Microbiome.Functional RelBE-Family Toxin-Antitoxin Pairs Affect Biofilm Maturation and Intestine Colonization in Vibrio cholerae.Expression and Quorum Sensing Regulation of Type III Secretion System Genes of Vibrio harveyi during Infection of Gnotobiotic Brine Shrimp.Biofilm Formation and Detachment in Gram-Negative Pathogens Is Modulated by Select Bile Acids Intestinal GPS: bile and bicarbonate control cyclic di-GMP to provide Vibrio cholerae spatial cues within the small intestine.Vibrio cholerae leuO Transcription Is Positively Regulated by ToxR and Contributes to Bile Resistance OxyR-activated expression of Dps is important for Vibrio cholerae oxidative stress resistance and pathogenesis.Differential Thiol-Based Switches Jump-Start Vibrio cholerae Pathogenesis Formation of an Intramolecular Periplasmic Disulfide Bond in TcpP Protects TcpP and TcpH from Degradation in Vibrio cholerae.Staying Alive: Vibrio cholerae's Cycle of Environmental Survival, Transmission, and Dissemination Small-molecule inhibitors of toxT expression in Vibrio cholerae.Transcriptional Profile during Deoxycholate-Induced Sporulation in a Clostridium perfringens Isolate Causing Foodborne Illness.Transcriptomic and phenotypic analysis of paralogous spx gene function in Bacillus anthracis Sterne.Enhanced interaction of Vibrio cholerae virulence regulators TcpP and ToxR under oxygen-limiting conditions Metabolic crosstalk between host and pathogen: sensing, adapting and competing.Characterization of V. cholerae T3SS-dependent cytotoxicity in cultured intestinal epithelial cells.Survival of the Fittest: How Bacterial Pathogens Utilize Bile To Enhance Infection.Calcium Enhances Bile Salt-Dependent Virulence Activation in Vibrio cholerae.Redox pathway sensing bile salts activates virulence gene expression in Vibrio cholerae.RNA-mediated signal perception in pathogenic bacteria.Electrostatic interactions between the CTX phage minor coat protein and the bacterial host receptor TolA drive the pathogenic conversion of Vibrio cholerae.Thiol-based switch mechanism of virulence regulator AphB modulates oxidative stress response in Vibrio cholerae.Relation between virulence of Vibrio anguillarum strains and response to the host factors mucin, bile salts and cholesterol.Bicarbonate increases binding affinity of Vibrio cholerae ToxT to virulence gene promoters.Vibrio parahaemolyticus VtrA is a membrane-bound regulator and is activated via oligomerization.Identification of the Regulon of AphB and Its Essential Roles in LuxR and Exotoxin Asp Expression in the Pathogen Vibrio alginolyticus.Structural and Functional Characterization of the FadR Regulatory Protein from Vibrio alginolyticus.The outer membrane protein TolC of Vibrio cholerae serves as a second cell-surface receptor for the VP3 phage.The bile salt sodium taurocholate induces Campylobacter jejuni outer membrane vesicle production and increases OMV-associated proteolytic activity.The Global Acetylome of the Human Pathogen Vibrio cholerae V52 Reveals Lysine Acetylation of Major Transcriptional Regulators.Transmembrane redox control and proteolysis of PdeC, a novel type of c-di-GMP phosphodiesterase.Hypermutation-induced in vivo oxidative stress resistance enhances Vibrio cholerae host adaptation

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P2860

Bile salt-induced intermolecular disulfide bond formation activates Vibrio cholerae virulence.

http://www.wikidata.org/entity/Q36598118

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Bile salt-induced intermolecul ...... tes Vibrio cholerae virulence.

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Bile salt-induced intermolecul ...... tes Vibrio cholerae virulence.

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Bile salt-induced intermolecul ...... tes Vibrio cholerae virulence.

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Bile salt-induced intermolecul ...... tes Vibrio cholerae virulence.

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Bile salt-induced intermolecul ...... tes Vibrio cholerae virulence.

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Proceedings of the National Academy of Sciences of the United States of America

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Bile salt-induced intermolecul ...... tes Vibrio cholerae virulence.

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Andrew M Stern

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Biao Kan

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Chambers Hughes

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Hui Wang

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Jun Zhu

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Menghua Yang

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William Fenical

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Zengtao Zhong

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Zhi Liu

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P2860

A general method of in vitro preparation and specific mutagenesis of DNA fragments: study of protein and DNA interactions

Protein thiol modifications visualized in vivo

Structure of Vibrio cholerae ToxT reveals a mechanism for fatty acid regulation of virulence genes

Tight regulation, modulation, and high-level expression by vectors containing the arabinose PBAD promoter

Virulence regulator AphB enhances toxR transcription in Vibrio cholerae.

Construction and characterization of amplifiable multicopy DNA cloning vehicles derived from the P15A cryptic miniplasmid

A bacterial two-hybrid system based on a reconstituted signal transduction pathway

Regulatory small RNAs circumvent the conventional quorum sensing pathway in pandemic Vibrio cholerae.

Genetic evidence for direct sensing of phenolic compounds by the VirA protein of Agrobacterium tumefaciens.

Bile acid-induced virulence gene expression of Vibrio parahaemolyticus reveals a novel therapeutic potential for bile acid sequestrants.

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2348-2353

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110

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Vibrio cholerae

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3568309

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