Brainstem mechanisms integrating gut-derived satiety signals and descending forebrain information in the control of meal size.
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Kv1.3 gene-targeted deletion alters longevity and reduces adiposity by increasing locomotion and metabolism in melanocortin-4 receptor-null miceBrain-gut-adipose-tissue communication pathways at a glanceHyperlipidemic diet causes loss of olfactory sensory neurons, reduces olfactory discrimination, and disrupts odor-reversal learning.Immune challenge and satiety-related activation of both distinct and overlapping neuronal populations in the brainstem indicate parallel pathways for viscerosensory signalingLeptin and the systems neuroscience of meal size control.Phenotype of neurons in the nucleus of the solitary tract that express CCK-induced activation of the ERK signaling pathwayA potential role for hypothalamomedullary POMC projections in leptin-induced suppression of food intake.Comparison of C57BL/6 and DBA/2 mice in food motivation and satietyFood demand and meal size in mice with single or combined disruption of melanocortin type 3 and 4 receptors.Modulation of orexigenic and anorexigenic peptides gene expression in the rat DVC and hypothalamus by acute immobilization stress.Ascending projections from the caudal visceral nucleus of the solitary tract to brain regions involved in food intake and energy expenditure.Impact of obesity-related genes in Spanish populationNMDA-type glutamate receptors participate in reduction of food intake following hindbrain melanocortin receptor activation.Melanocortin control of energy balance: evidence from rodent models.High-fat feeding impairs nutrient sensing and gut brain integration in the caudomedial nucleus of the solitary tract in mice.Determination of obesity associated gene variants related to TMEM18 through ultra-deep targeted re-sequencing in a case-control cohort for pediatric obesity.Volume transmission of beta-endorphin via the cerebrospinal fluid; a reviewDivergent leptin signaling in proglucagon neurons of the nucleus of the solitary tract in mice and rats.PVN pathways controlling energy homeostasis.Stress induced obesity: lessons from rodent models of stressNeural systems controlling the drive to eat: mind versus metabolism.Presynaptic melanocortin-4 receptors on vagal afferent fibers modulate the excitability of rat nucleus tractus solitarius neurons.Central and peripheral regulation of food intake and physical activity: pathways and genesParabrachial coding of sapid sucrose: relevance to reward and obesity.Loss of Vagal Sensitivity to Cholecystokinin in Rats Born with Intrauterine Growth Retardation and Consequence on Food IntakePlasticity of vagal brainstem circuits in the control of gastrointestinal function.Neuronal and intracellular signaling pathways mediating GLP-1 energy balance and glycemic effects.Hindbrain neurons as an essential hub in the neuroanatomically distributed control of energy balance.Central leucine sensing in the control of energy homeostasis.The endocrinology of food intake.Vagal neurocircuitry and its influence on gastric motilityNutritional status-dependent endocannabinoid signalling regulates the integration of rat visceral information.Interoceptive modulation of neuroendocrine, emotional, and hypophagic responses to stress.Parabrachial Nucleus Contributions to Glucagon-Like Peptide-1 Receptor Agonist-Induced Hypophagia.Knockdown of Neuropeptide Y in the Dorsomedial Hypothalamus Promotes Hepatic Insulin Sensitivity in Male Rats.Exposure to a high fat diet during the perinatal period alters vagal motoneurone excitability, even in the absence of obesity.Regulation of hindbrain Pyy expression by acute food deprivation, prolonged caloric restriction, and weight loss surgery in miceNesfatin-130-59 Injected Intracerebroventricularly Differentially Affects Food Intake Microstructure in Rats Under Normal Weight and Diet-Induced Obese Conditions.Hindbrain leptin and glucagon-like-peptide-1 receptor signaling interact to suppress food intake in an additive manner.Nesfatin-1(30-59) but not the N- and C-terminal fragments, nesfatin-1(1-29) and nesfatin-1(60-82) injected intracerebroventricularly decreases dark phase food intake by increasing inter-meal intervals in mice.
P2860
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P2860
Brainstem mechanisms integrating gut-derived satiety signals and descending forebrain information in the control of meal size.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
Brainstem mechanisms integrati ...... n in the control of meal size.
@ast
Brainstem mechanisms integrati ...... n in the control of meal size.
@en
type
label
Brainstem mechanisms integrati ...... n in the control of meal size.
@ast
Brainstem mechanisms integrati ...... n in the control of meal size.
@en
prefLabel
Brainstem mechanisms integrati ...... n in the control of meal size.
@ast
Brainstem mechanisms integrati ...... n in the control of meal size.
@en
P2093
P1476
Brainstem mechanisms integrati ...... n in the control of meal size.
@en
P2093
Gregory M Sutton
Huiyuan Zheng
Laurel M Patterson
R Leigh Townsend
P304
P356
10.1016/J.PHYSBEH.2006.08.018
P407
P577
2006-09-25T00:00:00Z