Modeling HIV-1 Latency in Primary T Cells Using a Replication-Competent Virus.
about
Benzotriazoles Reactivate Latent HIV-1 through Inactivation of STAT5 SUMOylation.HIV Integration Site Analysis of Cellular Models of HIV Latency with a Probe-Enriched Next-Generation Sequencing Assay.Ex Vivo Bioactivity and HIV-1 Latency Reversal by Ingenol Dibenzoate and Panobinostat in Resting CD4(+) T Cells from Aviremic Patients.Transcriptomic Analysis Implicates the p53 Signaling Pathway in the Establishment of HIV-1 Latency in Central Memory CD4 T Cells in an In Vitro Model.Kill: boosting HIV-specific immune responses.Protein expression from unintegrated HIV-1 DNA introduces bias in primary in vitro post-integration latency modelsMycobacterium tuberculosis reactivates latent HIV-1 in T cells in vitro.T-cell responses targeting HIV Nef uniquely correlate with infected cell frequencies after long-term antiretroviral therapy.Latent HIV reservoirs exhibit inherent resistance to elimination by CD8+ T cells.
P2860
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P2860
Modeling HIV-1 Latency in Primary T Cells Using a Replication-Competent Virus.
description
2015 nî lūn-bûn
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2015年の論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年论文
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2015年论文
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name
Modeling HIV-1 Latency in Primary T Cells Using a Replication-Competent Virus.
@ast
Modeling HIV-1 Latency in Primary T Cells Using a Replication-Competent Virus.
@en
type
label
Modeling HIV-1 Latency in Primary T Cells Using a Replication-Competent Virus.
@ast
Modeling HIV-1 Latency in Primary T Cells Using a Replication-Competent Virus.
@en
prefLabel
Modeling HIV-1 Latency in Primary T Cells Using a Replication-Competent Virus.
@ast
Modeling HIV-1 Latency in Primary T Cells Using a Replication-Competent Virus.
@en
P2093
P2860
P50
P356
P1476
Modeling HIV-1 Latency in Primary T Cells Using a Replication-Competent Virus
@en
P2093
Adam M Spivak
Camille L Novis
Eva Malatinkova
Laura J Martins
Linos Vanderkerckhove
Pawel Bonczkowski
Vicente Planelles
Wim Trypsteen
P2860
P304
P356
10.1089/AID.2015.0106
P577
2015-07-14T00:00:00Z