Insulitis and β-Cell Mass in the Natural History of Type 1 Diabetes. - Wikidata - awgldk - TriplyDB

Insulitis and β-Cell Mass in the Natural History of Type 1 Diabetes.

Insulitis and β-Cell Mass in the Natural History of Type 1 Diabetes.

http://www.wikidata.org/entity/Q36608775

about

Early prediction of autoimmune (type 1) diabetes.Insulitis in the pathogenesis of type 1 diabetes Tissue distribution and clonal diversity of the T and B cell repertoire in type 1 diabetes Analysis of self-antigen specificity of islet-infiltrating T cells from human donors with type 1 diabetes.Viral infections in type 1 diabetes mellitus--why the β cells?Narrowing in on the anti-β cell-specific T cells: looking 'where the action is'.Bringing the human pancreas into focus: new paradigms for the understanding of Type 1 diabetes.Aberrant Accumulation of the Diabetes Autoantigen GAD65 in Golgi Membranes in Conditions of ER Stress and Autoimmunity Promoting Immune Regulation in Type 1 Diabetes Using Low-Dose Interleukin-2.Stress-induced adaptive islet cell identity changes Early sympathetic islet neuropathy in autoimmune diabetes: lessons learned and opportunities for investigation.The Role of the Intestinal Microbiome in Type 1 Diabetes Pathogenesis.Increase in Pancreatic Proinsulin and Preservation of β-Cell Mass in Autoantibody-Positive Donors Prior to Type 1 Diabetes Onset.Differentiation of Diabetes by Pathophysiology, Natural History, and Prognosis.Memory T Cells in Type 1 Diabetes: the Devil is in the Detail.An ancestral retroviral protein identified as a therapeutic target in type-1 diabetes.Type I Interferon Is a Catastrophic Feature of the Diabetic Islet Microenvironment Human beta cell mass and function in diabetes: Recent advances in knowledge and technologies to understand disease pathogenesis.Generation of an immortalized mesenchymal stem cell line producing a secreted biosensor protein for glucose monitoring.The Four-Way Stop Sign: Viruses, 12-Lipoxygenase, Islets, and Natural Killer Cells in Type 1 Diabetes Progression T Cell Receptor Profiling in Type 1 Diabetes.Human Type 1 Diabetes Is Characterized by an Early, Marked, Sustained, and Islet-Selective Loss of Sympathetic Nerves.Islet-Derived CD4 T Cells Targeting Proinsulin in Human Autoimmune Diabetes.Isogenic Cellular Systems Model the Impact of Genetic Risk Variants in the Pathogenesis of Type 1 Diabetes.Antithymocyte Globulin Plus G-CSF Combination Therapy Leads to Sustained Immunomodulatory and Metabolic Effects in a Subset of Responders With Established Type 1 Diabetes.β-Cell mass versus function in type 1 diabetes mellitus: truth or dare?A nanobody-based tracer targeting DPP6 for non-invasive imaging of human pancreatic endocrine cells.SRp55 Regulates a Splicing Network that Controls Human Pancreatic Beta Cell Function and Survival.Persistence of Pancreatic Insulin mRNA Expression and Proinsulin Protein in Type 1 Diabetes Pancreata.Re-addressing the 2013 consensus guidelines for the diagnosis of insulitis in human type 1 diabetes: is change necessary?Exercise to preserve β-cell function in recent-onset Type 1 diabetes mellitus (EXTOD) - a randomized controlled pilot trial.Autoreactive T cells in type 1 diabetes.Aetiology of type 1 diabetes: Physiological growth in children affects disease progression.Screening, staging, and naming of presymptomatic type 1 diabetes.Decreased α-cell mass and early structural alterations of the exocrine pancreas in patients with type 1 diabetes: An analysis based on the nPOD repository.Insulitis in Autoantibody-Positive Pancreatic Donor With History of Gestational Diabetes Mellitus.Type 1 Diabetes: A Chronic Anti-Self-Inflammatory Response.Mitochondrial Reactive Oxygen Species and Type 1 Diabetes.A subclass of serum anti-ZnT8 antibodies directed to the surface of live pancreatic β-cells.Loss of intra-islet heparan sulfate is a highly sensitive marker of type 1 diabetes progression in humans.

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Insulitis and β-Cell Mass in the Natural History of Type 1 Diabetes.

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Insulitis and β-Cell Mass in the Natural History of Type 1 Diabetes.

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Insulitis and β-Cell Mass in the Natural History of Type 1 Diabetes.

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Insulitis and β-Cell Mass in the Natural History of Type 1 Diabetes

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Alberto Pugliese

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Ann Fu

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Clive Wasserfall

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Desmond A Schatz

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Mark A Atkinson

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Martha Campbell-Thompson

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P2860

Marked expansion of exocrine and endocrine pancreas with incretin therapy in humans with increased exocrine pancreas dysplasia and the potential for glucagon-producing neuroendocrine tumors

Allele frequency net: a database and online repository for immune gene frequencies in worldwide populations

Type 1 diabetes

The Juvenile Diabetes Research Foundation Network for Pancreatic Organ Donors with Diabetes (nPOD) Program: goals, operational model and emerging findings

Network for Pancreatic Organ Donors with Diabetes (nPOD): developing a tissue biobank for type 1 diabetes

Immunological responses to exogenous insulin

Novel Association Between Immune-Mediated Susceptibility Loci and Persistent Autoantibody Positivity in Type 1 Diabetes.

Fall in C-peptide during first 2 years from diagnosis: evidence of at least two distinct phases from composite Type 1 Diabetes TrialNet data

Mononuclear cell infiltration and its relation to the expression of major histocompatibility complex antigens and adhesion molecules in pancreas biopsy specimens from newly diagnosed insulin-dependent diabetes mellitus patients.

Residual insulin production and pancreatic ß-cell turnover after 50 years of diabetes: Joslin Medalist Study.

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type-1 diabetes

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