Mycobacteria in Crohn's disease: a persistent hypothesis. - Wikidata - awgldk - TriplyDB

Mycobacteria in Crohn's disease: a persistent hypothesis.

Mycobacteria in Crohn's disease: a persistent hypothesis.

http://www.wikidata.org/entity/Q36609219

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Mycobacteria in Crohn’s disease: how innate immune deficiency may result in chronic inflammation Microbial fuel cells and microbial ecology: applications in ruminant health and production research Identification of IL18RAP/IL18R1 and IL12B as leprosy risk genes demonstrates shared pathogenesis between inflammation and infectious diseases A Missense LRRK2 Variant Is a Risk Factor for Excessive Inflammatory Responses in Leprosy.Metagenomic Characterization of Microbial Communities In Situ Within the Deeper Layers of the Ileum in Crohn's Disease.Leprosy as a genetic disease.Genetics of infectious diseases: hidden etiologies and common pathways.Crohn's disease and the mycobacterioses: a quarter century later. Causation or simple association?Evolution, revolution and heresy in the genetics of infectious disease susceptibility.Genetics of leprosy reactions: an overview.Bacterial Intestinal Superinfections in Inflammatory Bowel Diseases Beyond Clostridum difficile.

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P2860

Mycobacteria in Crohn's disease: a persistent hypothesis.

http://www.wikidata.org/entity/Q36609219

description

2006 nî lūn-bûn

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2006年の論文

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2006年論文

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2006年論文

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2006年论文

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2006年论文

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name

Mycobacteria in Crohn's disease: a persistent hypothesis.

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Mycobacteria in Crohn's disease: a persistent hypothesis.

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Mycobacteria in Crohn's disease: a persistent hypothesis.

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Mycobacteria in Crohn's disease: a persistent hypothesis.

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Mycobacteria in Crohn's disease: a persistent hypothesis.

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Mycobacteria in Crohn's disease: a persistent hypothesis.

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Inflammatory Bowel Diseases

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Mycobacteria in Crohn's disease: a persistent hypothesis.

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Erwin Schurr

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Marcel A Behr

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P2860

A frameshift mutation in NOD2 associated with susceptibility to Crohn's disease

Nod2 is a general sensor of peptidoglycan through muramyl dipeptide (MDP) detection

Reduced Paneth cell alpha-defensins in ileal Crohn's disease

NOD2 and toll-like receptors are nonredundant recognition systems of Mycobacterium tuberculosis

Flagellin-deficient Legionella mutants evade caspase-1- and Naip5-mediated macrophage immunity

Association of NOD2 leucine-rich repeat variants with susceptibility to Crohn's disease

Host recognition of bacterial muramyl dipeptide mediated through NOD2. Implications for Crohn's disease

Toll-like receptor triggering of a vitamin D-mediated human antimicrobial response

Protagonist. Mycobacterium avium subspecies paratuberculosis is a cause of Crohn's disease

Nod2-dependent regulation of innate and adaptive immunity in the intestinal tract

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1000-1004

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Crohn's disease