Increased glial glutamate transporter EAAT2 expression reduces epileptogenic processes following pilocarpine-induced status epilepticus.
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Current approaches to enhance glutamate transporter function and expressionSmall-molecule activator of glutamate transporter EAAT2 translation provides neuroprotectionAdenosine kinase, glutamine synthetase and EAAT2 as gene therapy targets for temporal lobe epilepsy.Genetic deletion of the neuronal glutamate transporter, EAAC1, results in decreased neuronal death after pilocarpine-induced status epilepticus.Glutamate transporter EAAT2: a new target for the treatment of neurodegenerative diseases.Restored glial glutamate transporter EAAT2 function as a potential therapeutic approach for Alzheimer's diseaseThe neuroprotective mechanism of ampicillin in a mouse model of transient forebrain ischemia.Translational enhancers of EAAT2: therapeutic implications for neurodegenerative disease.Glutamate transporter EAAT2: regulation, function, and potential as a therapeutic target for neurological and psychiatric disease.Glutamatergic Mechanisms Associated with Seizures and Epilepsy.Brain endothelial cells induce astrocytic expression of the glutamate transporter GLT-1 by a Notch-dependent mechanism.Mechanisms of Excessive Extracellular Glutamate Accumulation in Temporal Lobe Epilepsy.Aquaporins in Brain Edema and Neuropathological Conditions.Increased expression of glutamate transporter GLT-1 in peritumoral tissue associated with prolonged survival and decreases in tumor growth in a rat model of experimental malignant glioma.Astroglia as a cellular target for neuroprotection and treatment of neuro-psychiatric disorders.Atorvastatin Prevents Glutamate Uptake Reduction Induced by Quinolinic Acid Via MAPKs Signaling.Regulation of astrocyte glutamate transporter-1 (GLT1) and aquaporin-4 (AQP4) expression in a model of epilepsy.Pharmacologic inhibition of Hsp90 to prevent GLT-1 degradation as an effective therapy for epilepsy.Astrocytic JWA deletion exacerbates dopaminergic neurodegeneration by decreasing glutamate transporters in mice.Clustering of spontaneous recurrent seizures separated by long seizure-free periods: An extended video-EEG monitoring study of a pilocarpine mouse model.Astroglial role in the pathophysiology of status epilepticus: an overview.Inhibition of p38 MAPK Signaling Regulates the Expression of EAAT2 in the Brains of Epileptic Rats
P2860
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P2860
Increased glial glutamate transporter EAAT2 expression reduces epileptogenic processes following pilocarpine-induced status epilepticus.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Increased glial glutamate tran ...... ne-induced status epilepticus.
@ast
Increased glial glutamate tran ...... ne-induced status epilepticus.
@en
type
label
Increased glial glutamate tran ...... ne-induced status epilepticus.
@ast
Increased glial glutamate tran ...... ne-induced status epilepticus.
@en
prefLabel
Increased glial glutamate tran ...... ne-induced status epilepticus.
@ast
Increased glial glutamate tran ...... ne-induced status epilepticus.
@en
P2093
P2860
P1476
Increased glial glutamate tran ...... ne-induced status epilepticus.
@en
P2093
Chien-Liang Glenn Lin
Delanie Schulte
Kou Takahashi
Nathan Stouffer
Qiongman Kong
Yuchen Lin
P2860
P304
P356
10.1016/J.NBD.2012.03.032
P577
2012-04-06T00:00:00Z