Development of genetically engineered mice lacking all three nitric oxide synthases. - Wikidata - awgldk - TriplyDB

Development of genetically engineered mice lacking all three nitric oxide synthases.

Development of genetically engineered mice lacking all three nitric oxide synthases.

http://www.wikidata.org/entity/Q36618944

about

Oxidative stress and epigenetic regulation in ageing and age-related diseases Reactive Oxygen-Related Diseases: Therapeutic Targets and Emerging Clinical Indications Subcellular and cellular locations of nitric oxide synthase isoforms as determinants of health and disease Nitric oxide synthase is not essential for Drosophila development.Nitric oxide exerts protective effects against bleomycin-induced pulmonary fibrosis in mice.Nitric oxide receptor soluble guanylyl cyclase undergoes splicing regulation in differentiating human embryonic cells.Nitric oxide coordinates metabolism, growth, and development via the nuclear receptor E75.Nitric oxide-cyclic GMP signaling in stem cell differentiation Contributions of nitric oxide synthases, dietary nitrite/nitrate, and other sources to the formation of NO signaling products.Cardiovascular and pharmacological implications of haem-deficient NO-unresponsive soluble guanylate cyclase knock-in mice.Dietary nitrite restores NO homeostasis and is cardioprotective in endothelial nitric oxide synthase-deficient mice.Nitric oxide, C-type natriuretic peptide and cGMP as regulators of endochondral ossification.Association of serum NO( x ) level with clustering of metabolic syndrome components in middle-aged and elderly general populations in Japan.Nitric oxide signaling in brain function, dysfunction, and dementia.Mechanotransduction in embryonic vascular development.Role of nitric oxide in the maintenance of pluripotency and regulation of the hypoxia response in stem cells.Animal models of coronary heart disease.Tipping off endothelial tubes: nitric oxide drives tip cells.Arginine deficiency causes runting in the suckling period by selectively activating the stress kinase GCN2.Severe dyslipidaemia, atherosclerosis, and sudden cardiac death in mice lacking all NO synthases fed a high-fat diet.Diabetic eNOS knockout mice develop distinct macro- and microvascular complications.Decreased Bronchial Eosinophilic Inflammation and Mucus Hypersecretion in Asthmatic Mice Lacking All Nitric Oxide Synthase Isoforms.

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P2860

Development of genetically engineered mice lacking all three nitric oxide synthases.

http://www.wikidata.org/entity/Q36618944

description

2006 nî lūn-bûn

@nan

2006年の論文

@ja

2006年論文

@yue

2006年論文

@zh-hant

2006年論文

@zh-hk

2006年論文

@zh-mo

2006年論文

@zh-tw

2006年论文

@wuu

2006年论文

@zh

2006年论文

@zh-cn

name

Development of genetically engineered mice lacking all three nitric oxide synthases.

@ast

Development of genetically engineered mice lacking all three nitric oxide synthases.

@en

type

label

Development of genetically engineered mice lacking all three nitric oxide synthases.

@ast

Development of genetically engineered mice lacking all three nitric oxide synthases.

@en

prefLabel

Development of genetically engineered mice lacking all three nitric oxide synthases.

@ast

Development of genetically engineered mice lacking all three nitric oxide synthases.

@en

P1433

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P1433

Journal of Pharmacological Sciences

P1476

Development of genetically engineered mice lacking all three nitric oxide synthases.

@en

P2093

Hiroaki Shimokawa

http://www.w3.org/2001/XMLSchema#string

Masato Tsutsui

http://www.w3.org/2001/XMLSchema#string

Nobuyuki Yanagihara

http://www.w3.org/2001/XMLSchema#string

Tsuyoshi Morishita

http://www.w3.org/2001/XMLSchema#string

Yasuhide Nakashima

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P304

147-154

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P31

scholarly article

P356

10.1254/JPHS.CPJ06015X

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2

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102

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P577

2006-10-07T00:00:00Z

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P698

17031076

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