2,3,7,8-Tetrachlorodibenzo-p-dioxin-mediated production of reactive oxygen species is an essential step in the mechanism of action to accelerate human keratinocyte differentiation.
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Role of AhR/ARNT system in skin homeostasisComplementary roles of specific cysteines in keratin 14 toward the assembly, organization, and dynamics of intermediate filaments in skin keratinocytesThe emerging role of Nrf2 in dermatotoxicologyAssociation of dioxin and other persistent organic pollutants (POPs) with diabetes: epidemiological evidence and new mechanisms of beta cell dysfunctionAntioxidant soybean tar Glyteer rescues T-helper-mediated downregulation of filaggrin expression via aryl hydrocarbon receptorAn aryl hydrocarbon receptor induces VEGF expression through ATF4 under glucose deprivation in HepG2A role for disulfide bonding in keratin intermediate filament organization and dynamics in skin keratinocytes.Proteomic analysis of human keratinocyte response to 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) exposure.Effects of in utero exposure of C57BL/6J mice to 2,3,7,8-tetrachlorodibenzo-p-dioxin on epidermal permeability barrier development and functionA metabolomic study of rats with doxorubicin-induced cardiomyopathy and Shengmai injection treatment.Disruption of Ah Receptor Signaling during Mouse Development Leads to Abnormal Cardiac Structure and Function in the Adult.Loss of multidrug resistance-associated protein 1 potentiates chronic doxorubicin-induced cardiac dysfunction in mice.Elevated glutathione is not sufficient to protect against doxorubicin-induced nuclear damage in heart in multidrug resistance-associated protein 1 (Mrp1/Abcc1) null miceAh Receptor Signaling Controls the Expression of Cardiac Development and Homeostasis Genes.Ah Receptor Activation by Dioxin Disrupts Activin, BMP, and WNT Signals During the Early Differentiation of Mouse Embryonic Stem Cells and Inhibits Cardiomyocyte FunctionsEffects of Developmental Activation of the Aryl Hydrocarbon Receptor by 2,3,7,8-Tetrachlorodibenzo-p-dioxin on Long-term Self-renewal of Murine Hematopoietic Stem Cells.Mitochondrial-targeted aryl hydrocarbon receptor and the impact of 2,3,7,8-tetrachlorodibenzo-p-dioxin on cellular respiration and the mitochondrial proteome.Role of EGF receptor ligands in TCDD-induced EGFR down-regulation and cellular proliferationAntioxidant Artemisia princeps Extract Enhances the Expression of Filaggrin and Loricrin via the AHR/OVOL1 Pathway.Contribute to the selection of the American College of Toxicology.Pharmacogenomics of chemically distinct classes of Keap1-Nrf2 activators identify common and unique gene, protein, and pathway responses in vivo.Two faces of the coin: Minireview for dissecting the role of reactive oxygen species in stem cell potency and lineage commitment.
P2860
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P2860
2,3,7,8-Tetrachlorodibenzo-p-dioxin-mediated production of reactive oxygen species is an essential step in the mechanism of action to accelerate human keratinocyte differentiation.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
2,3,7,8-Tetrachlorodibenzo-p-d ...... keratinocyte differentiation.
@ast
2,3,7,8-Tetrachlorodibenzo-p-d ...... keratinocyte differentiation.
@en
type
label
2,3,7,8-Tetrachlorodibenzo-p-d ...... keratinocyte differentiation.
@ast
2,3,7,8-Tetrachlorodibenzo-p-d ...... keratinocyte differentiation.
@en
prefLabel
2,3,7,8-Tetrachlorodibenzo-p-d ...... keratinocyte differentiation.
@ast
2,3,7,8-Tetrachlorodibenzo-p-d ...... keratinocyte differentiation.
@en
P2093
P2860
P356
P1476
2,3,7,8-Tetrachlorodibenzo-p-d ...... keratinocyte differentiation.
@en
P2093
Carrie Hayes Sutter
Elizabeth Kensicki
Lawrence H Kennedy
Quynh T Tran
Robert P Mohney
Sandra Leon Carrion
Sridevi Bodreddigari
Thomas R Sutter
P2860
P304
P356
10.1093/TOXSCI/KFS325
P577
2012-11-14T00:00:00Z