Dysfunction of inflammation-resolving pathways is associated with exaggerated postoperative cognitive decline in a rat model of the metabolic syndrome.
about
Effect of Ulinastatin in the Treatment of Postperative Cognitive Dysfunction: Review of Current LiteraturePostoperative Cognitive Dysfunction: Minding the Gaps in Our Knowledge of a Common Postoperative Complication in the ElderlyMicroglia mediate postoperative hippocampal inflammation and cognitive decline in mice.Vagus nerve through α7 nAChR modulates lung infection and inflammation: models, cells, and signalsExaggerated Acute Lung Injury and Impaired Antibacterial Defenses During Staphylococcus aureus Infection in Rats with the Metabolic Syndrome.Inverse Relationship between Serum Lipoxin A4 Level and the Risk of Metabolic Syndrome in a Middle-Aged Chinese PopulationObesity and post-operative cognitive dysfunction: a systematic review and meta-analysis.Relation between acute and long-term cognitive decline after surgery: Influence of metabolic syndrome.The α7-nicotinic receptor is upregulated in immune cells from HIV-seropositive women: consequences to the cholinergic anti-inflammatory responseInflammatory bowel disease as a disorder of an imbalance between pro- and anti-inflammatory molecules and deficiency of resolution bioactive lipids.Rapamycin increases grip strength and attenuates age-related decline in maximal running distance in old low capacity runner rats.The neuroinflammatory response of postoperative cognitive declineHigh inborn aerobic capacity does not protect the heart following myocardial infarction.High-mobility group box 1 protein initiates postoperative cognitive decline by engaging bone marrow-derived macrophagesThe Effect of Propofol Versus Isoflurane Anesthesia on Human Cerebrospinal Fluid Markers of Alzheimer's Disease: Results of a Randomized Trial.Prior infection exacerbates postoperative cognitive dysfunction in aged rats.Combination of aspirin with essential fatty acids is superior to aspirin alone to prevent or ameliorate sepsis or ARDS.Rats bred for low and high running capacity display alterations in peripheral tissues and nerves relevant to neuropathy and pain.Perioperative management of the obese surgical patient.Exercise Prevents Enhanced Postoperative Neuroinflammation and Cognitive Decline and Rectifies the Gut Microbiome in a Rat Model of Metabolic Syndrome.Screening of Potential Genes and Transcription Factors of Postoperative Cognitive Dysfunction via Bioinformatics Methods.Neurocognitive Function after Cardiac Surgery: From Phenotypes to Mechanisms.Detection of impaired cognitive function in rat with hepatosteatosis model and improving effect of GLP-1 analogs (exenatide) on cognitive function in hepatosteatosis
P2860
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P2860
Dysfunction of inflammation-resolving pathways is associated with exaggerated postoperative cognitive decline in a rat model of the metabolic syndrome.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Dysfunction of inflammation-re ...... del of the metabolic syndrome.
@ast
Dysfunction of inflammation-re ...... del of the metabolic syndrome.
@en
type
label
Dysfunction of inflammation-re ...... del of the metabolic syndrome.
@ast
Dysfunction of inflammation-re ...... del of the metabolic syndrome.
@en
prefLabel
Dysfunction of inflammation-re ...... del of the metabolic syndrome.
@ast
Dysfunction of inflammation-re ...... del of the metabolic syndrome.
@en
P2093
P2860
P1433
P1476
Dysfunction of inflammation-re ...... odel of the metabolic syndrome
@en
P2093
Ajay Chawla
Lauren G Koch
Mervyn Maze
Steven L Britton
Xiaomei Feng
P2860
P2888
P304
P356
10.2119/MOLMED.2012.00351
P407
P577
2013-02-08T00:00:00Z
P6179
1068965589