Frequent genomic imbalances suggest commonly altered tumour genes in human hepatocarcinogenesis - Wikidata - awgldk - TriplyDB

Frequent genomic imbalances suggest commonly altered tumour genes in human hepatocarcinogenesis

Frequent genomic imbalances suggest commonly altered tumour genes in human hepatocarcinogenesis

http://www.wikidata.org/entity/Q36623649

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Identification of frequent cytogenetic aberrations in hepatocellular carcinoma using gene-expression microarray data MicroRNA expression in hepatitis C virus-related malignancies: A brief review Recurrent amplification of RTEL1 and ABCA13 and its synergistic effect associated with clinicopathological data of gastric adenocarcinoma.Molecular Pathogenesis of Hepatitis-B-virus-associated Hepatocellular Carcinoma.Tissue microarray for high-throughput analysis of gene expression profiles in hepatocellular carcinoma Detection of chromosomal imbalances in hepatocellular carcinoma.Genetic mechanisms of hepatocarcinogenesis.Genomic losses at 5q13.2 and 8p23.1 in dysplastic hepatocytes are common events in hepatitis B virus-related hepatocellular carcinoma Generation and analysis of genetically defined liver carcinomas derived from bipotential liver progenitors Generation of a mouse model for studying the role of upregulated RTEL1 activity in tumorigenesis Chronic hepatitis B in hepatocarcinogenesis Target genes discovery through copy number alteration analysis in human hepatocellular carcinoma.Chromosome 1q21 amplification and oncogenes in hepatocellular carcinoma.Early diagnosis of liver cancer: an appraisal of international recommendations and future perspectives.Somatic LMCD1 mutations promoted cell migration and tumor metastasis in hepatocellular carcinoma.Block of proliferation 1 (BOP1) plays an oncogenic role in hepatocellular carcinoma by promoting epithelial-to-mesenchymal transition.Transcriptional regulation of serine/threonine kinase-15 (STK15) expression by hypoxia and HIF-1.Ex vivo analysis of antineoplastic agents in precision-cut tissue slices of human origin: effects of cyclooxygenase-2 inhibition in hepatocellular carcinoma.Oncogenetic tree modeling of human hepatocarcinogenesis.Analysis of genetic damage and gene polymorphism in hepatocellular carcinoma (HCC) patients in a South Indian population.Evaluation of loss of heterozygosity before and after interferon therapy in patients with hepatitis C virus infection who developed hepatocellular carcinoma during follow up.Molecular karyotyping of human hepatocellular carcinoma using single-nucleotide polymorphism arrays.

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P2860

Frequent genomic imbalances suggest commonly altered tumour genes in human hepatocarcinogenesis

http://www.wikidata.org/entity/Q36623649

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2001 nî lūn-bûn

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2001年の論文

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2001年論文

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2001年論文

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2001年論文

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2001年論文

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2001年論文

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2001年论文

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2001年论文

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2001年论文

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name

Frequent genomic imbalances su ...... in human hepatocarcinogenesis

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Frequent genomic imbalances su ...... in human hepatocarcinogenesis

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type

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Frequent genomic imbalances su ...... in human hepatocarcinogenesis

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Frequent genomic imbalances su ...... in human hepatocarcinogenesis

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prefLabel

Frequent genomic imbalances su ...... in human hepatocarcinogenesis

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Frequent genomic imbalances su ...... in human hepatocarcinogenesis

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British Journal of Cancer

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Frequent genomic imbalances su ...... in human hepatocarcinogenesis

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P2093

Caselmann WH

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Decker HJ

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Dienes HP

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Geissler F

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P2860

E-cadherin is a tumour/invasion suppressor gene mutated in human lobular breast cancers

Interaction of the Ski oncoprotein with Smad3 regulates TGF-beta signaling

A novel steroid thyroid hormone receptor-related gene inappropriately expressed in human hepatocellular carcinoma

A new retinoic acid receptor identified from a hepatocellular carcinoma

Identification of the breast cancer susceptibility gene BRCA2

Activation of ras oncogene in aflatoxin-induced rat liver carcinogenesis.

Allele loss on chromosome 16 associated with progression of human hepatocellular carcinoma.

Somatic mutations of the beta-catenin gene are frequent in mouse and human hepatocellular carcinomas

Identification by representational difference analysis of a homozygous deletion in pancreatic carcinoma that lies within the BRCA2 region.

Loss of heterozygosity suggests tumor suppressor gene responsible for primary hepatocellular carcinoma.

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