ROS-mediated activation of AKT induces apoptosis via pVHL in prostate cancer cells - Wikidata - awgldk - TriplyDB

ROS-mediated activation of AKT induces apoptosis via pVHL in prostate cancer cells

ROS-mediated activation of AKT induces apoptosis via pVHL in prostate cancer cells

http://www.wikidata.org/entity/Q36625600

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Deguelin induces the apoptosis of lung cancer cells through regulating a ROS driven Akt pathway.Licochalcone A-induced human gastric cancer BGC-823 cells apoptosis by regulating ROS-mediated MAPKs and PI3K/AKT signaling pathways Suppression of MAPK Signaling and Reversal of mTOR-Dependent MDR1-Associated Multidrug Resistance by 21α-Methylmelianodiol in Lung Cancer Cells bFGF Promotes the Migration of Human Dermal Fibroblasts under Diabetic Conditions through Reactive Oxygen Species Production via the PI3K/Akt-Rac1- JNK Pathways.Identification of Novel Epigenetic Markers of Prostate Cancer by NotI-Microarray Analysis Novel derivative of aminobenzenesulfonamide (3c) induces apoptosis in colorectal cancer cells through ROS generation and inhibits cell migration.Mitochondrial metabolites extend lifespan.Estrogen controls the survival of BRCA1-deficient cells via a PI3K-NRF2-regulated pathway.Methylseleninic acid elevates REDD1 and inhibits prostate cancer cell growth despite AKT activation and mTOR dysregulation in hypoxia.A CD13 inhibitor, ubenimex, synergistically enhances the effects of anticancer drugs in hepatocellular carcinoma.Curcumin induced human gastric cancer BGC-823 cells apoptosis by ROS-mediated ASK1-MKK4-JNK stress signaling pathway.Extremely low frequency magnetic fields regulate differentiation of regulatory T cells: Potential role for ROS-mediated inhibition on AKT.Stevioside induced cytotoxicity in colon cancer cells via reactive oxygen species and mitogen-activated protein kinase signaling pathways-mediated apoptosis.Juglanin inhibits lung cancer by regulation of apoptosis, ROS and autophagy induction.Reactive oxygen species extend insect life span using components of the insulin-signaling pathway.CD38 promotes pristane-induced chronic inflammation and increases susceptibility to experimental lupus by an apoptosis-driven and TRPM2-dependent mechanism.Phosphorylation of Smac by Akt promotes the caspase-3 activation during etoposide-induced apoptosis in HeLa cells.MicroRNA-21-5p induces the metastatic phenotype of human cervical carcinoma cells in vitro by targeting the von Hippel-Lindau tumor suppressor.

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P2860

ROS-mediated activation of AKT induces apoptosis via pVHL in prostate cancer cells

http://www.wikidata.org/entity/Q36625600

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2013 nî lūn-bûn

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name

ROS-mediated activation of AKT induces apoptosis via pVHL in prostate cancer cells

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ROS-mediated activation of AKT induces apoptosis via pVHL in prostate cancer cells

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type

label

ROS-mediated activation of AKT induces apoptosis via pVHL in prostate cancer cells

@ast

ROS-mediated activation of AKT induces apoptosis via pVHL in prostate cancer cells

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prefLabel

ROS-mediated activation of AKT induces apoptosis via pVHL in prostate cancer cells

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ROS-mediated activation of AKT induces apoptosis via pVHL in prostate cancer cells

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Molecular and Cellular Biochemistry

P1476

ROS-mediated activation of AKT induces apoptosis via pVHL in prostate cancer cells

@en

P2093

Ayesha S Don-Salu-Hewage

http://www.w3.org/2001/XMLSchema#string

Cimona V Hinton

http://www.w3.org/2001/XMLSchema#string

Danaya A Bethea

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Kia J Jones

http://www.w3.org/2001/XMLSchema#string

Mahandranauth A Chetram

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Valerie A Odero-Marah

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P2860

Hypoxia-inducible factor 1 is a basic-helix-loop-helix-PAS heterodimer regulated by cellular O2 tension

Mechanism of activation of protein kinase B by insulin and IGF-1

The tumor suppressor, PTEN/MMAC1, dephosphorylates the lipid second messenger, phosphatidylinositol 3,4,5-trisphosphate

Switching Akt: from survival signaling to deadly response

Akt phosphorylation of BAD couples survival signals to the cell-intrinsic death machinery

Loss of PTEN permits CXCR4-mediated tumorigenesis through ERK1/2 in prostate cancer cells

Caspase-3 is required for alpha-fodrin cleavage but dispensable for cleavage of other death substrates in apoptosis

Phosphorylation of the transcription factor forkhead family member FKHR by protein kinase B

Mitochondria, oxidants, and aging

Mitochondrial formation of reactive oxygen species

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s11010-012-1549-7

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63-71

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10.1007/S11010-012-1549-7

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1-2

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3578043

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