Association Between Helicobacter pylori cagA, babA2 Virulence Factors and Gastric Mucosal Interleukin-33 mRNA Expression and Clinical Outcomes in Dyspeptic Patients.
about
Oxidative Stress Resulting From Helicobacter pylori Infection Contributes to Gastric Carcinogenesis.Helicobacter pylori Activates HMGB1 Expression and Recruits RAGE into Lipid Rafts to Promote Inflammation in Gastric Epithelial Cells.Low Rate of babA2 Genotype among Iranian Helicobacter pylori Clinical Isolates.Low Rate of babA2 Genotype among Iranian Helicobacter pylori Clinical Isolates.Helicobacter pylori-induced IL-33 modulates mast cell responses, benefits bacterial growth, and contributes to gastritis.
P2860
Association Between Helicobacter pylori cagA, babA2 Virulence Factors and Gastric Mucosal Interleukin-33 mRNA Expression and Clinical Outcomes in Dyspeptic Patients.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
Association Between Helicobact ...... utcomes in Dyspeptic Patients.
@ast
Association Between Helicobact ...... utcomes in Dyspeptic Patients.
@en
type
label
Association Between Helicobact ...... utcomes in Dyspeptic Patients.
@ast
Association Between Helicobact ...... utcomes in Dyspeptic Patients.
@en
prefLabel
Association Between Helicobact ...... utcomes in Dyspeptic Patients.
@ast
Association Between Helicobact ...... utcomes in Dyspeptic Patients.
@en
P2093
P2860
P1476
Association Between Helicobact ...... utcomes in Dyspeptic Patients.
@en
P2093
Hedayatollah Shirzad
Heshmat Shahi
Loghman Salimzadeh
Rasol Bahreini
Somayeh Reiisi
P2860
P304
P577
2015-01-01T00:00:00Z