Expression of HIV-Tat protein is associated with learning and memory deficits in the mouse.
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HIV-associated neurocognitive disorder--pathogenesis and prospects for treatmentExposure to HIV-1 Tat in brain impairs sensorimotor gating and activates microglia in limbic and extralimbic brain regions of male mice.Anxiety-like behavior of mice produced by conditional central expression of the HIV-1 regulatory protein, Tat.HIV-1 Tat-induced changes in synaptically-driven network activity adapt during prolonged exposure.Conditional Tat protein brain expression in the GT-tg bigenic mouse induces cerebral fractional anisotropy abnormalities.STAT3 and its phosphorylation are involved in HIV-1 Tat-induced transactivation of glial fibrillary acidic proteinTime-Dependent, HIV-Tat-Induced Perturbation of Human Neurons In Vitro: Towards a Model for the Molecular Pathology of HIV-Associated Neurocognitive DisordersProgesterone protects normative anxiety-like responding among ovariectomized female mice that conditionally express the HIV-1 regulatory protein, Tat, in the CNS.Interactive HIV-1 Tat and morphine-induced synaptodendritic injury is triggered through focal disruptions in Na⁺ influx, mitochondrial instability, and Ca²⁺ overload.Anti-tat Hutat2:Fc mediated protection against tat-induced neurotoxicity and HIV-1 replication in human monocyte-derived macrophagesAssociations between brain microstructures, metabolites, and cognitive deficits during chronic HIV-1 infection of humanized miceHIV-1 Tat activates a RhoA signaling pathway to reduce NMDA-evoked calcium responses in hippocampal neurons via an actin-dependent mechanismTat-induced histopathological alterations mediate hippocampus-associated behavioural impairments in rats.Effects of chronic HIV-1 Tat exposure in the CNS: heightened vulnerability of males versus females to changes in cell numbers, synaptic integrity, and behaviorSensitization of enteric neurons to morphine by HIV-1 Tat protein.Molecular mechanism of HIV-1 Tat interacting with human dopamine transporter.Intra-ventral tegmental area HIV-1 Tat1-86 attenuates nicotine-mediated locomotor sensitization and alters mesocorticolimbic ERK and CREB signaling in ratsOligodendrocytes Are Targets of HIV-1 Tat: NMDA and AMPA Receptor-Mediated Effects on Survival and Development.HIV-1 subtypes B and C Tat differentially impact synaptic plasticity expression and implicates HIV-associated neurocognitive disorders.The HIV-1 transgenic rat model of neuroHIV.Synaptic dysfunction in the hippocampus accompanies learning and memory deficits in human immunodeficiency virus type-1 Tat transgenic mice.Conditional Tat protein expression in the GT-tg bigenic mouse brain induces gray matter density reductions.Methamphetamine Exposure Combined with HIV-1 Disease or gp120 Expression: Comparison of Learning and Executive Functions in Humans and MiceCannabinoids Occlude the HIV-1 Tat-Induced Decrease in GABAergic Neurotransmission in Prefrontal Cortex Slices.Targeting the glutamatergic system for the treatment of HIV-associated neurocognitive disordersRole of Histidine 547 of Human Dopamine Transporter in Molecular Interaction with HIV-1 Tat and Dopamine UptakeCentral administration of angiotensin IV rapidly enhances novel object recognition among miceEffects of conditional central expression of HIV-1 tat protein to potentiate cocaine-mediated psychostimulation and reward among male mice.NMDARs Adapt to Neurotoxic HIV Protein Tat Downstream of a GluN2A-Ubiquitin Ligase Signaling Pathway.HIV-1 Tat and Cocaine Impair Survival of Cultured Primary Neuronal Cells via a Mitochondrial Pathway.Synaptodendritic injury with HIV-Tat protein: What is the therapeutic target?HIV-1 transgenic rats display an increase in [(3)H]dopamine uptake in the prefrontal cortex and striatum.Animal models for depression associated with HIV-1 infection.Chronic low-level expression of HIV-1 Tat promotes a neurodegenerative phenotype with aging.Effects of HIV and Methamphetamine on Brain and Behavior: Evidence from Human Studies and Animal Models.HIV-1 Vpr disrupts mitochondria axonal transport and accelerates neuronal aging.HIV-associated synaptic degeneration.Effects of sex and HIV serostatus on spatial navigational learning and memory among cocaine users.Endocannabinoids exert CB1 receptor-mediated neuroprotective effects in models of neuronal damage induced by HIV-1 Tat protein.A GluN2B-Selective NMDAR Antagonist Reverses Synapse Loss and Cognitive Impairment Produced by the HIV-1 Protein Tat.
P2860
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P2860
Expression of HIV-Tat protein is associated with learning and memory deficits in the mouse.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Expression of HIV-Tat protein ...... memory deficits in the mouse.
@ast
Expression of HIV-Tat protein ...... memory deficits in the mouse.
@en
type
label
Expression of HIV-Tat protein ...... memory deficits in the mouse.
@ast
Expression of HIV-Tat protein ...... memory deficits in the mouse.
@en
prefLabel
Expression of HIV-Tat protein ...... memory deficits in the mouse.
@ast
Expression of HIV-Tat protein ...... memory deficits in the mouse.
@en
P2093
P2860
P1476
Expression of HIV-Tat protein ...... memory deficits in the mouse.
@en
P2093
Amanda N Carey
Elizabeth I Sypek
Harminder D Singh
Jay P McLaughlin
Marc J Kaufman
P2860
P356
10.1016/J.BBR.2011.12.019
P407
P577
2011-12-20T00:00:00Z