Peri-infarct blood-brain barrier dysfunction facilitates induction of spreading depolarization associated with epileptiform discharges.
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Monitoring stroke progression: in vivo imaging of cortical perfusion, blood-brain barrier permeability and cellular damage in the rat photothrombosis model.Blood-brain barrier imaging in human neuropathologies.Spreading depression requires microglia and is decreased by their M2a polarization from environmental enrichment.Albumin induces excitatory synaptogenesis through astrocytic TGF-β/ALK5 signaling in a model of acquired epilepsy following blood-brain barrier dysfunctionNeuronal uptake of serum albumin is associated with neuron damage during the development of epilepsyInflammatory pathways of seizure disorders.Should losartan be administered following brain injury?Anatomy and physiology of the blood-brain barrier.Practical Applications of in Vivo and ex Vivo MRI in Toxicologic Pathology Using a Novel High-performance Compact MRI System.Recording, analysis, and interpretation of spreading depolarizations in neurointensive care: Review and recommendations of the COSBID research group.Spreading Depolarizations: A Therapeutic Target Against Delayed Cerebral Ischemia After Subarachnoid Hemorrhage.A potential role for glia-derived extracellular matrix remodeling in postinjury epilepsy.Epileptiform activity and spreading depolarization in the blood-brain barrier-disrupted peri-infarct hippocampus are associated with impaired GABAergic inhibition and synaptic plasticity.TGFβ signaling is associated with changes in inflammatory gene expression and perineuronal net degradation around inhibitory neurons following various neurological insults.Effects of anti-epileptic drugs on spreading depolarization-induced epileptiform activity in mouse hippocampal slices.Neuroinflammation, Mast Cells, and Glia: Dangerous Liaisons.Alteration in rectification of potassium channels in perinatal hypoxia ischemia brain damage.Blood-brain barrier dysfunction can contribute to pharmacoresistance of seizures.Targeting Adenosine Monophosphate-Activated Protein Kinase by Metformin Adjusts Post-Ischemic Hyperemia and Extracellular Neuronal Discharge in Transient Global Cerebral Ischemia.Inhibition of 2-AG hydrolysis differentially regulates blood brain barrier permeability after injury.
P2860
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P2860
Peri-infarct blood-brain barrier dysfunction facilitates induction of spreading depolarization associated with epileptiform discharges.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Peri-infarct blood-brain barri ...... with epileptiform discharges.
@ast
Peri-infarct blood-brain barri ...... with epileptiform discharges.
@en
type
label
Peri-infarct blood-brain barri ...... with epileptiform discharges.
@ast
Peri-infarct blood-brain barri ...... with epileptiform discharges.
@en
prefLabel
Peri-infarct blood-brain barri ...... with epileptiform discharges.
@ast
Peri-infarct blood-brain barri ...... with epileptiform discharges.
@en
P2093
P2860
P1476
Peri-infarct blood-brain barri ...... d with epileptiform discharges
@en
P2093
A Friedman
A Maslarova
E G Lapilover
K Lippmann
U Heinemann
P2860
P304
P356
10.1016/J.NBD.2012.06.024
P577
2012-07-07T00:00:00Z