Limited maximal exercise capacity in patients with chronic heart failure: partitioning the contributors. - Wikidata - awgldk - TriplyDB

Limited maximal exercise capacity in patients with chronic heart failure: partitioning the contributors.

Limited maximal exercise capacity in patients with chronic heart failure: partitioning the contributors.

http://www.wikidata.org/entity/Q36658793

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Exercise intolerance in heart failure with preserved ejection fraction: more than a heart problem Using exercise to measure and modify cardiac function Determinants of exercise intolerance in elderly heart failure patients with preserved ejection fraction Left Ventricular Assist Device as a Bridge to Recovery for Patients With Advanced Heart Failure Inflammatory cytokine response to exercise in alpha-1-antitrypsin deficient COPD patients 'on' or 'off' augmentation therapy.Cardiovascular phenotype in HFpEF patients with or without diabetes: a RELAX trial ancillary study.Hemodynamic responses to small muscle mass exercise in heart failure patients with reduced ejection fraction.Muscle oxygen supply impairment during exercise in poorly controlled type 1 diabetes Isolated quadriceps training increases maximal exercise capacity in chronic heart failure: the role of skeletal muscle convective and diffusive oxygen transport Role of physical training in heart failure with preserved ejection fraction Characterization of exercise limitations by evaluating individual cardiac output patterns: a prospective cohort study in patients with chronic heart failure Muscle oxygen transport and utilization in heart failure: implications for exercise (in)tolerance.Incremental large and small muscle mass exercise in patients with heart failure: evidence of preserved peripheral haemodynamics and metabolism.Skeletal muscle mitochondrial density, gene expression, and enzyme activities in human heart failure: minimal effects of the disease and resistance training.Differential Responses of Post-Exercise Recovery of Leg Blood Flow and Oxygen Uptake Kinetics in HFpEF versus HFrEF Exercise training in chronic heart failure: improving skeletal muscle O2 transport and utilization.Relationship between aerobic capacity and cardiovascular disease risk factors in Thai men and women with normolipidemia and dyslipidemia Determinants of exercise intolerance in patients with heart failure and reduced or preserved ejection fraction.A differing role of oxidative stress in the regulation of central and peripheral hemodynamics during exercise in heart failure.Tai chi in patients with heart failure with preserved ejection fraction.Relationship of flow-mediated arterial dilation and exercise capacity in older patients with heart failure and preserved ejection fraction.Intact skeletal muscle mitochondrial enzyme activity but diminished exercise capacity in advanced heart failure patients on optimal medical and device therapy.Effects of endurance, circuit, and relaxing training on cardiovascular risk factors in hypertensive elderly patients.The resistive and elastic work of breathing during exercise in patients with chronic heart failure.Heart failure with preserved ejection fraction in the elderly: scope of the problem.Intrinsic skeletal muscle alterations in chronic heart failure patients: a disease-specific myopathy or a result of deconditioning?Skeletal muscle vasodilatation during maximal exercise in health and disease.Skeletal muscle protein metabolism in human heart failure.The Mechanoreflex and Hemodynamic Response to Passive Leg Movement in Heart Failure Muscle sympathetic activity in resting and exercising humans with and without heart failure.Decreases in maximal oxygen uptake following long-duration spaceflight: Role of convective and diffusive O2 transport mechanisms.Peripheral vascular function, oxygen delivery and utilization: the impact of oxidative stress in aging and heart failure with reduced ejection fraction.A novel cardiopulmonary exercise test protocol and criterion to determine maximal oxygen uptake in chronic heart failure.Exercise training in chronic heart failure Pathophysiology of exercise intolerance in chronic diseases: the role of diminished cardiac performance in mitochondrial and heart failure patients Aerobic exercise intensity assessment and prescription in cardiac rehabilitation: a joint position statement of the European Association for Cardiovascular Prevention and Rehabilitation, the American Association of Cardiovascular and Pulmonary Rehab Exercise Intolerance in HFpEF: Diagnosing and Ranking its Causes Using Personalized O2 Pathway Analysis.Exercise-Based Rehabilitation for Heart Failure: Clinical Evidence.A mitochondrial-targeted antioxidant improves myofilament Ca2+ sensitivity during prolonged low frequency force depression at low PO2.Exercise training in Tgαq*44 mice during the progression of chronic heart failure: cardiac vs. peripheral (soleus muscle) impairments to oxidative metabolism.

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P2860

Limited maximal exercise capacity in patients with chronic heart failure: partitioning the contributors.

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Odile Mathieu-Costello

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P2860

Comprehensive local muscle training increases aerobic working capacity and quality of life and decreases neurohormonal activation in patients with chronic heart failure.

Percutaneous needle biopsy of skeletal muscle in physiological and clinical research.

Reduction in hemoglobin-oxygen affinity results in the improvement of exercise capacity in mice with chronic heart failure.

Evidence of O2 supply-dependent VO2 max in the exercise-trained human quadriceps.

Measurement of cardiac output during exercise by open-circuit acetylene uptake.

A histochemical method for the simultaneous demonstration of capillaries and fiber type in skeletal muscle.

A more sensitive and specific radioenzymatic assay for catecholamines.

Relation between central and peripheral hemodynamics during exercise in patients with chronic heart failure. Muscle blood flow is reduced with maintenance of arterial perfusion pressure.

Skeletal muscle oxidative metabolism in sedentary humans: 31P-MRS assessment of O2 supply and demand limitations.

Effects of hyperoxia on maximal leg O2 supply and utilization in men.

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