TLR2 was overexpressed independent of IL-6 in patients with valvular atrial fibrillation.

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Inflammation and the pathogenesis of atrial fibrillation.Pentraxin 3 and other inflammatory biomarkers related to atrial fibrillation in cardiac surgery.

P2860

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P2860

TLR2 was overexpressed independent of IL-6 in patients with valvular atrial fibrillation.

Item

http://www.wikidata.org/entity/Q36683218

description

2011 nî lūn-bûn

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2011年の論文

@ja

2011年論文

@yue

2011年論文

@zh-hant

2011年論文

@zh-hk

2011年論文

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2011年論文

@zh-tw

2011年论文

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2011年论文

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2011年论文

@zh-cn

name

TLR2 was overexpressed independent of IL-6 in patients with valvular atrial fibrillation.

@ast

TLR2 was overexpressed independent of IL-6 in patients with valvular atrial fibrillation.

@en

type

Item

label

TLR2 was overexpressed independent of IL-6 in patients with valvular atrial fibrillation.

@ast

TLR2 was overexpressed independent of IL-6 in patients with valvular atrial fibrillation.

@en

prefLabel

TLR2 was overexpressed independent of IL-6 in patients with valvular atrial fibrillation.

@ast

TLR2 was overexpressed independent of IL-6 in patients with valvular atrial fibrillation.

@en

P1476

TLR2 was overexpressed independent of IL-6 in patients with valvular atrial fibrillation.

@en

P2093

Fei Cui

http://www.w3.org/2001/XMLSchema#string

Hailong Cao

http://www.w3.org/2001/XMLSchema#string

Jian Wang

http://www.w3.org/2001/XMLSchema#string

Lei Xue

http://www.w3.org/2001/XMLSchema#string

Ting Dai

http://www.w3.org/2001/XMLSchema#string

Yijiang Chen

http://www.w3.org/2001/XMLSchema#string

P2860

Prevalence of diagnosed atrial fibrillation in adults: national implications for rhythm management and stroke prevention: the AnTicoagulation and Risk Factors in Atrial Fibrillation (ATRIA) Study

Expression of TLR2, TLR4, and TLR9 in dermatomyositis and polymyositis.

Heat shock protein 96 is elevated in rheumatoid arthritis and activates macrophages primarily via TLR2 signaling.

Remodeling of ion channel expression in patients with chronic atrial fibrillation and mitral valvular heart disease.

Inflammation as a risk factor for atrial fibrillation.

Histological substrate of atrial biopsies in patients with lone atrial fibrillation.

Interleukin-6 and its receptor: a paradigm for cytokines.

Is atrial fibrillation an inflammatory disorder?

Epidemiology of atrial fibrillation: a current perspective.

Innate immune adaptor MyD88 mediates neutrophil recruitment and myocardial injury after ischemia-reperfusion in mice.

P304

178-184

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scholarly article

P356

10.1016/S1674-8301(11)60023-7

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2011-05-01T00:00:00Z

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236105549

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P698

23554687

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P921

Toll-like receptor

overexpression

P932

3597063

http://www.w3.org/2001/XMLSchema#string

TLR2 was overexpressed independent of IL-6 in patients with valvular atrial fibrillation.

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P2860

P2860

TLR2 was overexpressed independent of IL-6 in patients with valvular atrial fibrillation.

description

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P1433

P1476

P2093

P2860

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P356

P433

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P5875

P698

P921

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P1433

P1476

P2093

P2860

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P5875

P698

P921

P932