A role for TGF-beta signaling in neurodegeneration: evidence from genetically engineered models.
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Remote excitation of neuronal circuits using low-intensity, low-frequency ultrasound.Role of ROS and RNS Sources in Physiological and Pathological ConditionsMicroglial cell dysregulation in brain aging and neurodegenerationPioglitazone improves reversal learning and exerts mixed cerebrovascular effects in a mouse model of Alzheimer's disease with combined amyloid-β and cerebrovascular pathologyTGFβ signaling in the brain increases with aging and signals to astrocytes and innate immune cells in the weeks after stroke.TGF-β induces TIAF1 self-aggregation via type II receptor-independent signaling that leads to generation of amyloid β plaques in Alzheimer's disease.ALK5-dependent TGF-β signaling is a major determinant of late-stage adult neurogenesis.Partial eNOS deficiency causes spontaneous thrombotic cerebral infarction, amyloid angiopathy and cognitive impairment.Experience-dependent gene expression in adult visual cortexChanges of peripheral TGF-β1 depend on monocytes-derived macrophages in Huntington diseaseHigh serum levels of transforming growth factor β1 are associated with increased cortical thickness in cingulate and right frontal areas in healthy subjects.TGF-beta1 reduces the heterogeneity of astrocytic cyclooxygenase-2 and nitric oxide synthase-2 gene expression in a stimulus-independent manner.Early defect of transforming growth factor β1 formation in Huntington's disease.Impaired glutamate recycling and GluN2B-mediated neuronal calcium overload in mice lacking TGF-β1 in the CNS.Cerebrovascular disorders: molecular insights and therapeutic opportunities.Making the brain glow: in vivo bioluminescence imaging to study neurodegeneration.Cell-specific blood-brain barrier regulation in health and disease: a focus on hypoxia.Transforming growth factor-beta1 impairs neuropathic pain through pleiotropic effects.Role of TGFβ signaling in the pathogenesis of Alzheimer's disease.Impaired Wnt signaling in dopamine containing neurons is associated with pathogenesis in a rotenone triggered Drosophila Parkinson's disease model.Chronic over-expression of TGFβ1 alters hippocampal structure and causes learning deficits.
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P2860
A role for TGF-beta signaling in neurodegeneration: evidence from genetically engineered models.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
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2006年论文
@zh-cn
name
A role for TGF-beta signaling ...... genetically engineered models.
@ast
A role for TGF-beta signaling ...... genetically engineered models.
@en
type
label
A role for TGF-beta signaling ...... genetically engineered models.
@ast
A role for TGF-beta signaling ...... genetically engineered models.
@en
prefLabel
A role for TGF-beta signaling ...... genetically engineered models.
@ast
A role for TGF-beta signaling ...... genetically engineered models.
@en
P1476
A role for TGF-beta signaling ...... genetically engineered models.
@en
P2093
Ina Tesseur
Tony Wyss-Coray
P304
P356
10.2174/156720506779025297
P577
2006-12-01T00:00:00Z