Norman Cousins Lecture. Glia as the "bad guys": implications for improving clinical pain control and the clinical utility of opioids.
about
Chronic exposure to corticosterone enhances the neuroinflammatory and neurotoxic responses to methamphetamineAn IL-1 receptor antagonist blocks a morphine-induced attenuation of locomotor recovery after spinal cord injuryProinflammatory cytokines oppose opioid-induced acute and chronic analgesiaMechanisms of chronic central neuropathic pain after spinal cord injuryChemokines and the pathophysiology of neuropathic painNeural Plasticity in Common Forms of Chronic HeadachesMetaplasticity and behavior: how training and inflammation affect plastic potential within the spinal cord and recovery after injuryMental Health Comorbidities in Pediatric Chronic Pain: A Narrative Review of Epidemiology, Models, Neurobiological Mechanisms and TreatmentNon-stereoselective reversal of neuropathic pain by naloxone and naltrexone: involvement of toll-like receptor 4 (TLR4)Glial modulators as potential treatments of psychostimulant abuseThe use of low-dose naltrexone (LDN) as a novel anti-inflammatory treatment for chronic painSilencing the Kir4.1 potassium channel subunit in satellite glial cells of the rat trigeminal ganglion results in pain-like behavior in the absence of nerve injuryA biological substrate for somatoform disorders: importance of pathophysiology.Minimal adverse effects profile following implantation of periauricular percutaneous electrical nerve field stimulators: a retrospective cohort study.Crosstalk between spinal astrocytes and neurons in nerve injury-induced neuropathic pain.Enduring reversal of neuropathic pain by a single intrathecal injection of adenosine 2A receptor agonists: a novel therapy for neuropathic pain.Fibromyalgia: presentation and management with a focus on pharmacological treatment.Botulinum toxin type a injections for cervical and shoulder girdle myofascial pain using an enriched protocol designSupraspinal inactivation of mitochondrial superoxide dismutase is a source of peroxynitrite in the development of morphine antinociceptive tolerance.Chemokine signaling and the management of neuropathic pain.Monocytes/Macrophages control resolution of transient inflammatory pain.Gabapentin improves cold-pressor pain responses in methadone-maintained patients.The glial cell modulator and phosphodiesterase inhibitor, AV411 (ibudilast), attenuates prime- and stress-induced methamphetamine relapse.Intrathecal injection of an alpha seven nicotinic acetylcholine receptor agonist attenuates gp120-induced mechanical allodynia and spinal pro-inflammatory cytokine profiles in rats.Low-dose naltrexone for the treatment of fibromyalgia: findings of a small, randomized, double-blind, placebo-controlled, counterbalanced, crossover trial assessing daily pain levels.The association between spinal cord trauma-sensitive miRNAs and pain sensitivity, and their regulation by morphine.Evidence for brain glial activation in chronic pain patients.The CB2 cannabinoid receptor-selective agonist O-3223 reduces pain and inflammation without apparent cannabinoid behavioral effects.Spinal toll like receptor 3 is involved in chronic pancreatitis-induced mechanical allodynia of ratIbudilast (AV-411). A new class therapeutic candidate for neuropathic pain and opioid withdrawal syndromes.Salivary markers of inflammation in response to acute stress.Exploring the neuroimmunopharmacology of opioids: an integrative review of mechanisms of central immune signaling and their implications for opioid analgesia.Naloxone-precipitated morphine withdrawal behavior and brain IL-1β expression: comparison of different mouse strainsPrior exposure to glucocorticoids potentiates lipopolysaccharide induced mechanical allodynia and spinal neuroinflammationDevelopment of viral vectors for gene therapy for chronic painInflammatory pain is enhanced in delta opioid receptor-knockout miceRepeated morphine treatment-mediated hyperalgesia, allodynia and spinal glial activation are blocked by co-administration of a selective cannabinoid receptor type-2 agonistProtein hormones and immunityGlia as a Link between Neuroinflammation and Neuropathic Pain.The influence of tumor necrosis factor-alpha -308 G/A and IL-6 -174 G/C on pain and analgesia response in lung cancer patients receiving supportive care.
P2860
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P2860
Norman Cousins Lecture. Glia as the "bad guys": implications for improving clinical pain control and the clinical utility of opioids.
description
2006 nî lūn-bûn
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2006年の論文
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2006年論文
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2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
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2006年論文
@zh-tw
2006年论文
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2006年论文
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2006年论文
@zh-cn
name
Norman Cousins Lecture. Glia a ...... e clinical utility of opioids.
@ast
Norman Cousins Lecture. Glia a ...... e clinical utility of opioids.
@en
type
label
Norman Cousins Lecture. Glia a ...... e clinical utility of opioids.
@ast
Norman Cousins Lecture. Glia a ...... e clinical utility of opioids.
@en
prefLabel
Norman Cousins Lecture. Glia a ...... e clinical utility of opioids.
@ast
Norman Cousins Lecture. Glia a ...... e clinical utility of opioids.
@en
P2093
P2860
P50
P1476
Norman Cousins Lecture. Glia a ...... e clinical utility of opioids.
@en
P2093
Annemarie Ledeboer
Erin D Milligan
Steven F Maier
P2860
P304
P356
10.1016/J.BBI.2006.10.011
P577
2006-12-18T00:00:00Z