Diesel exhaust increases EGFR and phosphorylated C-terminal Tyr 1173 in the bronchial epithelium. - Wikidata - awgldk - TriplyDB

Diesel exhaust increases EGFR and phosphorylated C-terminal Tyr 1173 in the bronchial epithelium.

Diesel exhaust increases EGFR and phosphorylated C-terminal Tyr 1173 in the bronchial epithelium.

http://www.wikidata.org/entity/Q36688527

about

Airway epithelial epidermal growth factor receptor mediates hogbarn dust-induced cytokine release but not Ca2+ response Impact of air pollution on allergic diseases Activation of EGFR by small compounds through coupling the generation of hydrogen peroxide to stable dimerization of Cu/Zn SOD1.Nanomaterials vs Ambient Ultrafine Particles: an Opportunity to Exchange Toxicology Knowledge Recent advances in particulate matter and nanoparticle toxicology: a review of the in vivo and in vitro studies Regulatory T cells protect fine particulate matter-induced inflammatory responses in human umbilical vein endothelial cells Inflammation-related effects of diesel engine exhaust particles: studies on lung cells in vitro Antioxidant airway responses following experimental exposure to wood smoke in man Assessment of the capacity of vehicle cabin air inlet filters to reduce diesel exhaust-induced symptoms in human volunteers.Diesel exhaust particulate (DEP) and nanoparticle exposures: what do DEP human clinical studies tell us about potential human health hazards of nanoparticles?Impaired vascular function after exposure to diesel exhaust generated at urban transient running conditions.Human papillomavirus 16 oncoprotein regulates the translocation of β-catenin via the activation of epidermal growth factor receptor Mechanistic impact of outdoor air pollution on asthma and allergic diseases Urban, traffic- related particles and lung tumors in urethane treated mice A benzimidazole derivative exhibiting antitumor activity blocks EGFR and HER2 activity and upregulates DR5 in breast cancer cells Atmospheric ultrafine particles promote vascular calcification via the NF-κB signaling pathway Engineering tissue alternatives to animals: applying tissue engineering to basic research and safety testing.Health effects of concentrated ambient air particulate matter (CAPs) and its components.Autocrine ligands of the epithelial growth factor receptor mediate inflammatory responses to diesel exhaust particles Roles of oxidative stress in signaling and inflammation induced by particulate matter.Triggering Mechanisms and Inflammatory Effects of Combustion Exhaust Particles with Implication for Carcinogenesis.Mechanisms and treatments for severe, steroid-resistant allergic airway disease and asthma.Ultrafine particles from diesel vehicle emissions at different driving cycles induce differential vascular pro-inflammatory responses: implication of chemical components and NF-kappaB signaling.Airway inflammatory response to diesel exhaust generated at urban cycle running conditions.Emissions from commercial-grade charbroiling meat operations induce oxidative stress and inflammatory responses in human bronchial epithelial cells Mass spectrometry profiling of oxylipins, endocannabinoids, and N-acylethanolamines in human lung lavage fluids reveals responsiveness of prostaglandin E2 and associated lipid metabolites to biodiesel exhaust exposure.Effects of pomegranate juice on hyperoxaluria-induced oxidative stress in the rat kidneys.Diesel exhaust particulates affect cell signaling, mucin profiles, and apoptosis in trachea explants of Balb/C mice.Nuclear factor κB1/RelA mediates the inflammation and/or survival of human airway exposed to sulfur mustard.

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P2860

Diesel exhaust increases EGFR and phosphorylated C-terminal Tyr 1173 in the bronchial epithelium.

http://www.wikidata.org/entity/Q36688527

description

2008 nî lūn-bûn

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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2008年论文

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2008年论文

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name

Diesel exhaust increases EGFR ...... 3 in the bronchial epithelium.

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Diesel exhaust increases EGFR ...... 3 in the bronchial epithelium.

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type

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Diesel exhaust increases EGFR ...... 3 in the bronchial epithelium.

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Diesel exhaust increases EGFR ...... 3 in the bronchial epithelium.

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prefLabel

Diesel exhaust increases EGFR ...... 3 in the bronchial epithelium.

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Diesel exhaust increases EGFR ...... 3 in the bronchial epithelium.

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P1433

Particle and Fibre Toxicology

P1476

Diesel exhaust increases EGFR ...... 3 in the bronchial epithelium.

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Donna E Davies

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Frank J Kelly

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Jamshid Pourazar

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Susan J Wilson

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Thomas Sandström

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P2860

Shc phosphotyrosine-binding domain dominantly interacts with epidermal growth factor receptors and mediates Ras activation in intact cells

Quinones and aromatic chemical compounds in particulate matter induce mitochondrial dysfunction: implications for ultrafine particle toxicity

Air pollution exposure assessment for epidemiologic studies of pregnant women and children: lessons learned from the Centers for Children's Environmental Health and Disease Prevention Research.

Activation of stress-activated protein kinases/c-Jun N-terminal protein kinases (SAPKs/JNKs) by a novel mitogen-activated protein kinase kinase

1,2-Naphthoquinone activates vanilloid receptor 1 through increased protein tyrosine phosphorylation, leading to contraction of guinea pig trachea

Grb2 interaction with MEK-kinase 1 is involved in regulation of Jun-kinase activities in response to epidermal growth factor

Activation of the EGF receptor signaling pathway in human airway epithelial cells exposed to metals

Activation of phospholipase C-gamma by phosphatidylinositol 3,4,5-trisphosphate

The role of individual SH2 domains in mediating association of phospholipase C-gamma1 with the activated EGF receptor

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Stephen T. Holgate

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phosphorylation

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