Silencing of STIM1 attenuates hypoxia-induced PASMCs proliferation via inhibition of the SOC/Ca2+/NFAT pathway
about
Mechanisms of Vascular Smooth Muscle Contraction and the Basis for Pharmacologic Treatment of Smooth Muscle DisordersWnt coreceptor Lrp5 is a driver of idiopathic pulmonary fibrosis.Evaluation of the visual analog score (VAS) to assess acute mountain sickness (AMS) in a hypobaric chamberNFATc3 and VIP in Idiopathic Pulmonary Fibrosis and Chronic Obstructive Pulmonary DiseaseVascular smooth muscle cell phenotype is defined by Ca2+-dependent transcription factors.Bronchoscopic Lung Cryobiopsy Increases Diagnostic Confidence in the Multidisciplinary Diagnosis of Idiopathic Pulmonary Fibrosis.The role of nuclear factor of activated T cells in pulmonary arterial hypertension.Calcineurin/NFAT Activation-Dependence of Leptin Synthesis and Vascular Growth in Response to Mechanical StretchThe role of ion channels in the hypoxia-induced aggressiveness of glioblastoma.Histone deacetylase inhibitors promote eNOS expression in vascular smooth muscle cells and suppress hypoxia-induced cell growth.Peroxisome proliferator-activated receptor gamma depletion stimulates Nox4 expression and human pulmonary artery smooth muscle cell proliferation.Role of Transcription Factors in Pulmonary Artery Smooth Muscle Cells: An Important Link to Hypoxic Pulmonary Hypertension.Orai1, 2, 3 and STIM1 promote store-operated calcium entry in pulmonary arterial smooth muscle cells.FGF9 and FGF18 in idiopathic pulmonary fibrosis promote survival and migration and inhibit myofibroblast differentiation of human lung fibroblasts in vitro.Cardiovascular and Hemostatic Disorders: Role of STIM and Orai Proteins in Vascular Disorders.Pushing chILD Forward: The Bright Future of Children's Interstitial Lung Diseases.Hypoxia induces pulmonary fibroblast proliferation through NFAT signaling.STIM2 (Stromal Interaction Molecule 2)-Mediated Increase in Resting Cytosolic Free Ca2+ Concentration Stimulates PASMC Proliferation in Pulmonary Arterial Hypertension.Volume doubling time of lung cancer detected in idiopathic interstitial pneumonia: comparison with that in chronic obstructive pulmonary disease.Micro-RNA Feedback Loops Modulating the Calcineurin/NFAT Signaling Pathway.Revisiting the Role of TRP, Orai, and ASIC Channels in the Pulmonary Arterial Response to Hypoxia.
P2860
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P2860
Silencing of STIM1 attenuates hypoxia-induced PASMCs proliferation via inhibition of the SOC/Ca2+/NFAT pathway
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
Silencing of STIM1 attenuates ...... n of the SOC/Ca2+/NFAT pathway
@ast
Silencing of STIM1 attenuates ...... n of the SOC/Ca2+/NFAT pathway
@en
type
label
Silencing of STIM1 attenuates ...... n of the SOC/Ca2+/NFAT pathway
@ast
Silencing of STIM1 attenuates ...... n of the SOC/Ca2+/NFAT pathway
@en
prefLabel
Silencing of STIM1 attenuates ...... n of the SOC/Ca2+/NFAT pathway
@ast
Silencing of STIM1 attenuates ...... n of the SOC/Ca2+/NFAT pathway
@en
P2093
P2860
P356
P1433
P1476
Silencing of STIM1 attenuates ...... n of the SOC/Ca2+/NFAT pathway
@en
P2093
P2860
P2888
P356
10.1186/1465-9921-14-2
P577
2013-01-05T00:00:00Z
P5875
P6179
1017721711