Why neurodegenerative diseases are progressive: uncontrolled inflammation drives disease progression. - Wikidata - awgldk - TriplyDB

Why neurodegenerative diseases are progressive: uncontrolled inflammation drives disease progression.

Why neurodegenerative diseases are progressive: uncontrolled inflammation drives disease progression.

http://www.wikidata.org/entity/Q36692860

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Physical Activity, Air Pollution and the Brain CCL8/MCP-2 is a target for mir-146a in HIV-1-infected human microglial cells Systemic administration of an antagonist of the ATP-sensitive receptor P2X7 improves recovery after spinal cord injury A Compendium of Preparation and Application of Stem Cells in Parkinson's Disease: Current Status and Future Prospects Herpes Simplex Virus Type 1 and Other Pathogens are Key Causative Factors in Sporadic Alzheimer's Disease Insights into Neuroinflammation in Parkinson's Disease: From Biomarkers to Anti-Inflammatory Based Therapies Traumatic brain injury, neuroinflammation, and post-traumatic headaches Role of prostaglandins in neuroinflammatory and neurodegenerative diseases Uncovering novel actors in astrocyte-neuron crosstalk in Parkinson's disease: the Wnt/β-catenin signaling cascade as the common final pathway for neuroprotection and self-repair Neuroprotective and symptomatic effects of targeting group III mGlu receptors in neurodegenerative disease Inhibition of microglia activation as a phenotypic assay in early drug discovery Dietary (poly)phenolics in human health: structures, bioavailability, and evidence of protective effects against chronic diseases Potential benefits of therapeutic use of β2-adrenergic receptor agonists in neuroprotection and Parkinsonμs disease AAV-dominant negative tumor necrosis factor (DN-TNF) gene transfer to the striatum does not rescue medium spiny neurons in the YAC128 mouse model of Huntington's disease Emerging Roles for the Immune System in Traumatic Brain Injury Morgellons disease: a filamentous borrelial dermatitis Protective Microglia and Their Regulation in Parkinson's Disease Imaging neuroinflammation in multiple sclerosis using TSPO-PET Glial modulators as potential treatments of psychostimulant abuse Paroxetine ameliorates lipopolysaccharide-induced microglia activation via differential regulation of MAPK signaling The protective effect of melatonin on neural stem cell against LPS-induced inflammation Interactions of iron, dopamine and neuromelanin pathways in brain aging and Parkinson's disease Effects of cordycepin on the microglia-overactivation-induced impairments of growth and development of hippocampal cultured neurons TGF-beta1 blockade of microglial chemotaxis toward Abeta aggregates involves SMAD signaling and down-regulation of CCL5 Acetaminophen inhibits neuronal inflammation and protects neurons from oxidative stress Inhibition of IkappaB kinase-beta protects dopamine neurons against lipopolysaccharide-induced neurotoxicity A Wnt1 regulated Frizzled-1/β-Catenin signaling pathway as a candidate regulatory circuit controlling mesencephalic dopaminergic neuron-astrocyte crosstalk: Therapeutical relevance for neuron survival and neuroprotection Ameliorative Effects of Antioxidants on the Hippocampal Accumulation of Pathologic Tau in a Rat Model of Blast-Induced Traumatic Brain Injury Anti-inflammatory treatment induced regenerative oligodendrogenesis in parkinsonian mice.Anxiety Disorders are Associated with Reduced Heart Rate Variability: A Meta-Analysis.Macrophage antigen complex-1 mediates reactive microgliosis and progressive dopaminergic neurodegeneration in the MPTP model of Parkinson's disease.Gene expression patterns associated with neurological disease in human HIV infection Qingkailing Suppresses the Activation of BV2 Microglial Cells by Inhibiting Hypoxia/Reoxygenation-Induced Inflammatory Responses.Pharmacological Modulation of Functional Phenotypes of Microglia in Neurodegenerative Diseases Oxidation of the endogenous cannabinoid arachidonoyl ethanolamide by the cytochrome P450 monooxygenases: physiological and pharmacological implications.DJ-1 deficient mice demonstrate similar vulnerability to pathogenic Ala53Thr human alpha-syn toxicity.Targeting Wnt signaling at the neuroimmune interface for dopaminergic neuroprotection/repair in Parkinson's disease.Substance P enhances microglial density in the substantia nigra through neurokinin-1 receptor/NADPH oxidase-mediated chemotaxis in mice Adenosine A2A receptor antagonism reverses inflammation-induced impairment of microglial process extension in a model of Parkinson's disease Cerebrospinal fluid metabolomics reveals altered waste clearance and accelerated aging in HIV patients with neurocognitive impairment.

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P2860

Why neurodegenerative diseases are progressive: uncontrolled inflammation drives disease progression.

http://www.wikidata.org/entity/Q36692860

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2008 nî lūn-bûn

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Why neurodegenerative diseases ...... on drives disease progression.

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Trends in Immunology

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Hui-Ming Gao

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Mice deficient in TNF receptors are protected against dopaminergic neurotoxicity: implications for Parkinson's disease

Systemic LPS causes chronic neuroinflammation and progressive neurodegeneration

Physiological contribution of the astrocytic environment of neurons to intersynaptic crosstalk

Therapeutic vaccine for acute and chronic motor neuron diseases: implications for amyotrophic lateral sclerosis.

Microglia and neuroinflammation: a pathological perspective

Evidence of active nerve cell degeneration in the substantia nigra of humans years after 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine exposure

Nonsteroidal antiinflammatory drug use and the risk for Parkinson's disease

NADPH oxidase: an update

Adhesion-promoting receptors on human macrophages recognize Escherichia coli by binding to lipopolysaccharide

Microglia: active sensor and versatile effector cells in the normal and pathologic brain

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Jau-Shyong Hong

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