Resveratrol (trans-3,5,4'-trihydroxystilbene) ameliorates experimental allergic encephalomyelitis, primarily via induction of apoptosis in T cells involving activation of aryl hydrocarbon receptor and estrogen receptor.
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Resveratrol and clinical trials: the crossroad from in vitro studies to human evidencePotential of immunomodulatory agents as adjunct host-directed therapies for multidrug-resistant tuberculosisThe Impact of Neuroimmune Alterations in Autism Spectrum DisorderResveratrol and calcium signaling: molecular mechanisms and clinical relevanceSirtuin deacylases: a molecular link between metabolism and immunityActivation of aryl hydrocarbon receptor (AhR) leads to reciprocal epigenetic regulation of FoxP3 and IL-17 expression and amelioration of experimental colitisThe role of AhR in autoimmune regulation and its potential as a therapeutic target against CD4 T cell mediated inflammatory disorderEvidence for ligand-mediated selective modulation of aryl hydrocarbon receptor activity.Prevention and treatment of diabetes with resveratrol in a non-obese mouse model of type 1 diabetes.Resveratrol (trans-3,5,4'-trihydroxystilbene) induces silent mating type information regulation-1 and down-regulates nuclear transcription factor-kappaB activation to abrogate dextran sulfate sodium-induced colitisRole of microRNAs in resveratrol-mediated mitigation of colitis-associated tumorigenesis in Apc(Min/+) mice.Antibody blockade of CLEC12A delays EAE onset and attenuates disease severity by impairing myeloid cell CNS infiltration and restoring positive immunity.Resveratrol suppresses colitis and colon cancer associated with colitis.Use of natural AhR ligands as potential therapeutic modalities against inflammatory disorders.3,3'-diindolylmethane ameliorates experimental autoimmune encephalomyelitis by promoting cell cycle arrest and apoptosis in activated T cells through microRNA signaling pathways.A novel microRNA-132-sirtuin-1 axis underlies aberrant B-cell cytokine regulation in patients with relapsing-remitting multiple sclerosis [corrected].Immune modulation by chondroitin sulfate and its degraded disaccharide product in the development of an experimental model of multiple sclerosisResveratrol prevents endothelial cells injury in high-dose interleukin-2 therapy against melanomaDose-dependency of resveratrol in providing health benefits.Prenatal exposure to TCDD triggers significant modulation of microRNA expression profile in the thymus that affects consequent gene expressionBryostatin-1, a naturally occurring antineoplastic agent, acts as a Toll-like receptor 4 (TLR-4) ligand and induces unique cytokines and chemokines in dendritic cells.Gene expression in the spinal cord in female lewis rats with experimental autoimmune encephalomyelitis induced with myelin basic proteinThe influence of nutritional factors on the prognosis of multiple sclerosis.Resveratrol (3,5,4'-trihydroxystilbene) protects pregnant mother and fetus from the immunotoxic effects of 2,3,7,8-tetrachlorodibenzo-p-dioxin.Baicalein selectively induces apoptosis in activated lymphocytes and ameliorates concanavalin a-induced hepatitis in mice.Type 1 regulatory T cells (Tr1) in autoimmunityOral resveratrol reduces neuronal damage in a model of multiple sclerosis.Resveratrol neuroprotection in a chronic mouse model of multiple sclerosis.Multiple anti-inflammatory pathways triggered by resveratrol lead to amelioration of staphylococcal enterotoxin B-induced lung injury.Role of resveratrol-induced CD11b(+) Gr-1(+) myeloid derived suppressor cells (MDSCs) in the reduction of CXCR3(+) T cells and amelioration of chronic colitis in IL-10(-/-) mice.Resveratrol (trans-3,5,4'-trihydroxystilbene) suppresses EL4 tumor growth by induction of apoptosis involving reciprocal regulation of SIRT1 and NF-κBDietary Indoles Suppress Delayed-Type Hypersensitivity by Inducing a Switch from Proinflammatory Th17 Cells to Anti-Inflammatory Regulatory T Cells through Regulation of MicroRNA.The role of IL-2 in the activation and expansion of regulatory T-cells and the development of experimental autoimmune encephalomyelitisCD44 Reciprocally regulates the differentiation of encephalitogenic Th1/Th17 and Th2/regulatory T cells through epigenetic modulation involving DNA methylation of cytokine gene promoters, thereby controlling the development of experimental autoimmunApigenin, a Natural Flavonoid, Attenuates EAE Severity Through the Modulation of Dendritic Cell and Other Immune Cell Functions.SIRT1 promotes RGC survival and delays loss of function following optic nerve crush.The importance of NAD in multiple sclerosisResveratrol exacerbates both autoimmune and viral models of multiple sclerosisUse of anti-cancer drugs, mitocans, to enhance the immune responses against tumors.Indoles mitigate the development of experimental autoimmune encephalomyelitis by induction of reciprocal differentiation of regulatory T cells and Th17 cells.
P2860
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P2860
Resveratrol (trans-3,5,4'-trihydroxystilbene) ameliorates experimental allergic encephalomyelitis, primarily via induction of apoptosis in T cells involving activation of aryl hydrocarbon receptor and estrogen receptor.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Resveratrol (trans-3,5,4'-trih ...... eceptor and estrogen receptor.
@ast
Resveratrol (trans-3,5,4'-trih ...... eceptor and estrogen receptor.
@en
type
label
Resveratrol (trans-3,5,4'-trih ...... eceptor and estrogen receptor.
@ast
Resveratrol (trans-3,5,4'-trih ...... eceptor and estrogen receptor.
@en
prefLabel
Resveratrol (trans-3,5,4'-trih ...... eceptor and estrogen receptor.
@ast
Resveratrol (trans-3,5,4'-trih ...... eceptor and estrogen receptor.
@en
P2093
P2860
P356
P1476
Resveratrol (trans-3,5,4'-trih ...... eceptor and estrogen receptor.
@en
P2093
Lorne J Hofseth
Mitzi Nagarkatti
Narendra P Singh
Prakash Nagarkatti
Venkatesh L Hegde
P2860
P304
P356
10.1124/MOL.107.038984
P577
2007-09-14T00:00:00Z