Intermittent nicotine exposure upregulates nAChRs in VTA dopamine neurons and sensitises locomotor responding to the drug. - Wikidata - awgldk - TriplyDB

Intermittent nicotine exposure upregulates nAChRs in VTA dopamine neurons and sensitises locomotor responding to the drug.

Intermittent nicotine exposure upregulates nAChRs in VTA dopamine neurons and sensitises locomotor responding to the drug.

http://www.wikidata.org/entity/Q36703862

about

The concept of allosteric interaction and its consequences for the chemistry of the brain Inside-out neuropharmacology of nicotinic drugs Mephedrone and nicotine: oxidative stress and behavioral interactions in animal models Levamisole enhances the rewarding and locomotor-activating effects of cocaine in rats.Nicotine and Resting-State Functional Connectivity: Effects of Intermittent Doses.α4α6β2* nicotinic acetylcholine receptor activation on ventral tegmental area dopamine neurons is sufficient to stimulate a depolarizing conductance and enhance surface AMPA receptor function Nicotine produces chronic behavioral sensitization with changes in accumbal neurotransmission and increased sensitivity to re-exposure.Exposure to nicotine enhances its subsequent self-administration: contribution of nicotine-associated contextual stimuli α6β2*-subtype nicotinic acetylcholine receptors are more sensitive than α4β2*-subtype receptors to regulation by chronic nicotine administration.Obsessive-compulsive symptoms and negative affect during tobacco withdrawal in a non-clinical sample of African American smokers.Nicotinic receptor blockade decreases fos immunoreactivity within orexin/hypocretin-expressing neurons of nicotine-exposed rats.

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P2860

Intermittent nicotine exposure upregulates nAChRs in VTA dopamine neurons and sensitises locomotor responding to the drug.

http://www.wikidata.org/entity/Q36703862

description

2013 nî lūn-bûn

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2013年の論文

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Intermittent nicotine exposure ...... omotor responding to the drug.

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Intermittent nicotine exposure ...... omotor responding to the drug.

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Intermittent nicotine exposure ...... omotor responding to the drug.

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Intermittent nicotine exposure ...... omotor responding to the drug.

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Intermittent nicotine exposure ...... omotor responding to the drug.

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Anitha P Govind

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Daniel S McGehee

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Danyan Mao

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Henry Chi

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James J Cortright

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Lorinda K Baker

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Michael Iacoviello

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Paul Vezina

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Stacy Herrera

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William N Green

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P2860

The neural basis of drug craving: an incentive-sensitization theory of addiction

Drug abuse: hedonic homeostatic dysregulation

Alpha-conotoxin MII-sensitive nicotinic acetylcholine receptors in the nucleus accumbens shell regulate progressive ratio responding maintained by nicotine.

Single cocaine exposure in vivo induces long-term potentiation in dopamine neurons.

Synaptic plasticity and nicotine addiction.

In vitro identification and electrophysiological characterization of dopamine neurons in the ventral tegmental area

Up-regulation of nicotinic receptors by nicotine varies with receptor subtype.

Functional nicotinic acetylcholine receptors containing α6 subunits are on GABAergic neuronal boutons adherent to ventral tegmental area dopamine neurons.

Elevated levels of GluR1 in the midbrain: a trigger for sensitization to drugs of abuse?

Nicotine potentiation of excitatory inputs to ventral tegmental area dopamine neurons.

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1004-1011

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10.1111/EJN.12114

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6

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37

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