Loss of endothelial programmed cell death 10 activates glioblastoma cells and promotes tumor growth.

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Introduction to cerebral cavernous malformation: a brief review.Study of angiogenic signaling pathways in hemangioblastoma.EphB4 forward signalling mediates angiogenesis caused by CCM3/PDCD10-ablation.

P2860

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Loss of endothelial programmed cell death 10 activates glioblastoma cells and promotes tumor growth.

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http://www.wikidata.org/entity/Q36705891

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2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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Loss of endothelial programmed ...... lls and promotes tumor growth.

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Loss of endothelial programmed ...... lls and promotes tumor growth.

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Loss of endothelial programmed ...... lls and promotes tumor growth.

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Loss of endothelial programmed ...... lls and promotes tumor growth.

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Loss of endothelial programmed ...... lls and promotes tumor growth.

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Loss of endothelial programmed ...... lls and promotes tumor growth.

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Loss of endothelial programmed ...... lls and promotes tumor growth.

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Anja Prinz

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Kai Zhao

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Kathy Keyvani

http://www.w3.org/2001/XMLSchema#string

Nicole Lambertz

http://www.w3.org/2001/XMLSchema#string

Ting Lei

http://www.w3.org/2001/XMLSchema#string

Ulrich Sure

http://www.w3.org/2001/XMLSchema#string

Yuan Zhu

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Adaptor protein cerebral cavernous malformation 3 (CCM3) mediates phosphorylation of the cytoskeletal proteins ezrin/radixin/moesin by mammalian Ste20-4 to protect cells from oxidative stress

Loss of CCM3 impairs DLL4-Notch signalling: implication in endothelial angiogenesis and in inherited cerebral cavernous malformations

Mutations within the programmed cell death 10 gene cause cerebral cavernous malformations

Rho kinase inhibition rescues the endothelial cell cerebral cavernous malformation phenotype.

CCM3 signaling through sterile 20-like kinases plays an essential role during zebrafish cardiovascular development and cerebral cavernous malformations

Cerebral cavernous malformations proteins inhibit Rho kinase to stabilize vascular integrity.

Stabilization of VEGFR2 signaling by cerebral cavernous malformation 3 is critical for vascular development

Loss of cerebral cavernous malformation 3 (Ccm3) in neuroglia leads to CCM and vascular pathology.

High-resolution characterization of the pancreatic adenocarcinoma genome.

Changes in leukocyte gene expression profiles induced by antineoplastic chemotherapy.

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538-548

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scholarly article

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10.1093/NEUONC/NOV155

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Ilonka Kreitschmann-Andermahr

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2015-08-08T00:00:00Z

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26254477

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endothelium

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4799675

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Loss of endothelial programmed cell death 10 activates glioblastoma cells and promotes tumor growth.

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P2860

P2860

Loss of endothelial programmed cell death 10 activates glioblastoma cells and promotes tumor growth.

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P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P50

P577

P698

P921

P932

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P50

P577

P698

P921

P932