Respiratory and behavioral dysfunction following loss of the GABAA receptor α4 subunit.
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Nalcn Is a "Leak" Sodium Channel That Regulates Excitability of Brainstem Chemosensory Neurons and Breathing.PHYSIOLOGY. Regulation of breathing by CO₂ requires the proton-activated receptor GPR4 in retrotrapezoid nucleus neurons.Dexmedetomidine for rigid bronchoscopy in an infant with tracheal web after ventricular septal defect patch repair.
P2860
Respiratory and behavioral dysfunction following loss of the GABAA receptor α4 subunit.
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name
Respiratory and behavioral dysfunction following loss of the GABAA receptor α4 subunit.
@ast
Respiratory and behavioral dysfunction following loss of the GABAA receptor α4 subunit.
@en
type
label
Respiratory and behavioral dysfunction following loss of the GABAA receptor α4 subunit.
@ast
Respiratory and behavioral dysfunction following loss of the GABAA receptor α4 subunit.
@en
prefLabel
Respiratory and behavioral dysfunction following loss of the GABAA receptor α4 subunit.
@ast
Respiratory and behavioral dysfunction following loss of the GABAA receptor α4 subunit.
@en
P2093
P2860
P356
P1433
P1476
Respiratory and behavioral dysfunction following loss of the GABAA receptor α4 subunit
@en
P2093
Ashley M Stevens
C Jean Loria
Ellen Crummy
Frank J Jacono
Ruth E Siegel
Thomas E Dick
P2860
P304
P356
10.1002/BRB3.122
P577
2013-02-05T00:00:00Z