Microglia activation as a biomarker for traumatic brain injury - Wikidata - awgldk - TriplyDB

Microglia activation as a biomarker for traumatic brain injury

Microglia activation as a biomarker for traumatic brain injury

http://www.wikidata.org/entity/Q36715224

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Cathepsin B is a New Drug Target for Traumatic Brain Injury Therapeutics: Evidence for E64d as a Promising Lead Drug Candidate Traumatic Brain Injury pathophysiology and treatments: early, intermediate, and late phases post-injury Linking traumatic brain injury to chronic traumatic encephalopathy: identification of potential mechanisms leading to neurofibrillary tangle development Mesenchymal stem cell-based treatments for stroke, neural trauma, and heat stroke Inflammatory reaction after traumatic brain injury: therapeutic potential of targeting cell-cell communication by chemokines The role of the surface on microglia function: implications for central nervous system tissue engineering Nuclear Factor Erythroid 2-Related Factor 2 (Nrf2) Mediates Neuroprotection in Traumatic Brain Injury at Least in Part by Inactivating Microglia Vascular and inflammatory factors in the pathophysiology of blast-induced brain injury.Effects of blast overpressure on neurons and glial cells in rat organotypic hippocampal slice cultures.Acetazolamide Mitigates Astrocyte Cellular Edema Following Mild Traumatic Brain Injury Suppressed cytokine expression immediatey following traumatic brain injury in neonatal rats indicates an expeditious endogenous anti-inflammatory response Role of microglia in a mouse model of paediatric traumatic brain injury Selective estrogen receptor modulators regulate reactive microglia after penetrating brain injury.Increased Amyloid Precursor Protein and Tau Expression Manifests as Key Secondary Cell Death in Chronic Traumatic Brain Injury.Military personnel with chronic symptoms following blast traumatic brain injury have differential expression of neuronal recovery and epidermal growth factor receptor genes.Traumatic brain injury causes selective, CD74-dependent peripheral lymphocyte activation that exacerbates neurodegeneration.Combination therapy of human umbilical cord blood cells and granulocyte colony stimulating factor reduces histopathological and motor impairments in an experimental model of chronic traumatic brain injury.Contributions of the immune system to the pathophysiology of traumatic brain injury - evidence by intravital microscopy.Possible roles of COX-1 in learning and memory impairment induced by traumatic brain injury in mice.Neuroinflammatory responses to traumatic brain injury: etiology, clinical consequences, and therapeutic opportunities.RNA-sequencing reveals oligodendrocyte and neuronal transcripts in microglia relevant to central nervous system disease.Non-Neuronal Cells Are Required to Mediate the Effects of Neuroinflammation: Results from a Neuron-Enriched Culture System.Activation of microglial P2Y12 receptor is required for outward potassium currents in response to neuronal injury.Call Off the Dog(ma): M1/M2 Polarization Is Concurrent following Traumatic Brain Injury.Intravenous Bone Marrow Stem Cell Grafts Preferentially Migrate to Spleen and Abrogate Chronic Inflammation in Stroke.Time-dependent effects of CX3CR1 in a mouse model of mild traumatic brain injury Neurotrauma: The Crosstalk between Neurotrophins and Inflammation in the Acutely Injured Brain.Administration of DHA Reduces Endoplasmic Reticulum Stress-Associated Inflammation and Alters Microglial or Macrophage Activation in Traumatic Brain Injury.Age exacerbates the CCR2/5-mediated neuroinflammatory response to traumatic brain injury.Brain Region-Specific Histopathological Effects of Varying Trajectories of Controlled Cortical Impact Injury Model of Traumatic Brain Injury.Astrocyte Hypertrophy Contributes to Aberrant Neurogenesis after Traumatic Brain Injury Older Age Results in Differential Gene Expression after Mild Traumatic Brain Injury and Is Linked to Imaging Differences at Acute Follow-up.Etifoxine improves sensorimotor deficits and reduces glial activation, neuronal degeneration, and neuroinflammation in a rat model of traumatic brain injury.Vascular damage: a persisting pathology common to Alzheimer's disease and traumatic brain injury.Mesenchymal Stem Cells of Dental Origin-Their Potential for Antiinflammatory and Regenerative Actions in Brain and Gut Damage.The contribution of astrocytes and microglia to traumatic brain injury Neuroinflammation after intracerebral hemorrhage.G-CSF as an adjunctive therapy with umbilical cord blood cell transplantation for traumatic brain injury.Microglia Responses in Acute and Chronic Neurological Diseases: What Microglia-Specific Transcriptomic Studies Taught (and did Not Teach) Us.Minocycline plus N-acteylcysteine induces remyelination, synergistically protects oligodendrocytes, and modifies neuroinflammation in a rat model of mild traumatic brain injury.

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P2860

Microglia activation as a biomarker for traumatic brain injury

http://www.wikidata.org/entity/Q36715224

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2013 nî lūn-bûn

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2013年論文

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name

Microglia activation as a biomarker for traumatic brain injury

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Microglia activation as a biomarker for traumatic brain injury

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Microglia activation as a biomarker for traumatic brain injury

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Microglia activation as a biomarker for traumatic brain injury

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Microglia activation as a biomarker for traumatic brain injury

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Microglia activation as a biomarker for traumatic brain injury

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Frontiers in Neurology

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Microglia activation as a biomarker for traumatic brain injury

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Brian Giunta

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Cesar V Borlongan

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Diana G Hernandez-Ontiveros

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Jun Tan

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Naoki Tajiri

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Sandra Acosta

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P2860

A call for transparent reporting to optimize the predictive value of preclinical research

The microglial "activation" continuum: from innate to adaptive responses

Engulfing astrocytes protect neurons from contact-induced apoptosis following injury

Microglia sculpt postnatal neural circuits in an activity and complement-dependent manner

Control of microglial neurotoxicity by the fractalkine receptor

Fractalkine modulates TNF-alpha secretion and neurotoxicity induced by microglial activation

Changes in autophagy after traumatic brain injury

Temporal changes in cell marker expression and cellular infiltration in a controlled cortical impact model in adult male C57BL/6 mice

Microglia: active sensor and versatile effector cells in the normal and pathologic brain

Monocyte and macrophage heterogeneity

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traumatic brain injury

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