Role of the peroxynitrite-poly(ADP-ribose) polymerase pathway in human disease.
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Fatty acid amide hydrolase is a key regulator of endocannabinoid-induced myocardial tissue injuryOxidative-Nitrosative Stress and Myocardial Dysfunctions in Sepsis: Evidence from the Literature and Postmortem ObservationsInterplay of oxidative, nitrosative/nitrative stress, inflammation, cell death and autophagy in diabetic cardiomyopathyCurrent understanding of immunity to Trypanosoma cruzi infection and pathogenesis of Chagas diseaseNuclear DNA damage signalling to mitochondria in ageingIn vitro and in vivo induction and activation of nNOS by LPS in oligodendrocytesPoly(ADP-ribose) (PAR) binding to apoptosis-inducing factor is critical for PAR polymerase-1-dependent cell death (parthanatos)Chronic PARP-1 inhibition reduces carotid vessel remodeling and oxidative damage of the dorsal hippocampus in spontaneously hypertensive rats.PARP-inhibitor treatment prevents hypertension induced cardiac remodeling by favorable modulation of heat shock proteins, Akt-1/GSK-3β and several PKC isoforms.Pathophysiologically relevant levels of hydrogen peroxide induce glutamate-independent neurodegeneration that involves activation of transient receptor potential melastatin 7 channelsA nuclease that mediates cell death induced by DNA damage and poly(ADP-ribose) polymerase-1Effect of parthanatos on ropivacaine-induced damage in SH-SY5Y cells.Iron stimulates plasma-activated medium-induced A549 cell injury.Dihydro-CDDO-trifluoroethyl amide (dh404), a novel Nrf2 activator, suppresses oxidative stress in cardiomyocytes.Effect of total flavonoids of Spatholobus suberectus Dunn on PCV2 induced oxidative stress in RAW264.7 cells.Xanthine oxidase inhibitor allopurinol attenuates the development of diabetic cardiomyopathy.Evolutionary history of the poly(ADP-ribose) polymerase gene family in eukaryotes.Novel neuroprotective strategies in ischemic retinal lesions.Mechanisms of neurovascular dysfunction in acute ischemic brain.New therapeutic and biomarker discovery for peripheral diabetic neuropathy: PARP inhibitor, nitrotyrosine, and tumor necrosis factor-{alpha}Poly(ADP-ribose) polymerase-dependent energy depletion occurs through inhibition of glycolysis.Modulation of poly(ADP-ribose) polymerase-1 (PARP-1)-mediated oxidative cell injury by ring finger protein 146 (RNF146) in cardiac myocytesEndothelial dysfunction is a potential contributor to multiple organ failure and mortality in aged mice subjected to septic shock: preclinical studies in a murine model of cecal ligation and puncturePeroxynitrite-driven mechanisms in diabetes and insulin resistance - the latest advancesHyperglycemia as a Risk Factor of Ischemic StrokePoly(ADP-ribose) polymerase-1 (PARP-1) gene deficiency alleviates diabetic kidney disease.Pathophysiological roles of peroxynitrite in circulatory shockAag-initiated base excision repair promotes ischemia reperfusion injury in liver, brain, and kidney.Mechanisms of disease: the oxidative stress theory of diabetic neuropathyReduced estradiol-induced vasodilation and poly-(ADP-ribose) polymerase (PARP) activity in the aortas of rats with experimental polycystic ovary syndrome (PCOS).Insulin renders diabetic rats resistant to acute ischemic stroke by arresting nitric oxide reaction with superoxide to form peroxynitrite.Signaling mechanism of poly(ADP-ribose) polymerase-1 (PARP-1) in inflammatory diseases.Mechanistic insight into DNA damage and repair in ischemic stroke: exploiting the base excision repair pathway as a model of neuroprotection.The key role of transient receptor potential melastatin-2 channels in amyloid-β-induced neurovascular dysfunction.PARP inhibition alleviates diabetes-induced systemic oxidative stress and neural tissue 4-hydroxynonenal adduct accumulation: correlation with peripheral nerve function.Effects of a potent peroxynitrite decomposition catalyst in murine models of endotoxemia and sepsis.Poly (ADP-ribose) polymerase-1 initiated neuronal cell death pathway--do androgens matter?Increased poly(ADP-ribosyl)ation in skeletal muscle tissue of pediatric patients with severe burn injury: prevention by propranolol treatment.PARP inhibition attenuates acute kidney allograft rejection by suppressing cell death pathways and activating PI-3K-Akt cascade.Burn and smoke injury activates poly(ADP-ribose)polymerase in circulating leukocytes.
P2860
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P2860
Role of the peroxynitrite-poly(ADP-ribose) polymerase pathway in human disease.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Role of the peroxynitrite-poly(ADP-ribose) polymerase pathway in human disease.
@ast
Role of the peroxynitrite-poly(ADP-ribose) polymerase pathway in human disease.
@en
type
label
Role of the peroxynitrite-poly(ADP-ribose) polymerase pathway in human disease.
@ast
Role of the peroxynitrite-poly(ADP-ribose) polymerase pathway in human disease.
@en
prefLabel
Role of the peroxynitrite-poly(ADP-ribose) polymerase pathway in human disease.
@ast
Role of the peroxynitrite-poly(ADP-ribose) polymerase pathway in human disease.
@en
P2860
P1476
Role of the peroxynitrite-poly(ADP-ribose) polymerase pathway in human disease.
@en
P2093
Csaba Szabo
P2860
P356
10.2353/AJPATH.2008.080019
P407
P50
P577
2008-06-05T00:00:00Z