Botulinum toxin type A normalizes alterations in urothelial ATP and NO release induced by chronic spinal cord injury. - Wikidata - awgldk - TriplyDB

Botulinum toxin type A normalizes alterations in urothelial ATP and NO release induced by chronic spinal cord injury.

Botulinum toxin type A normalizes alterations in urothelial ATP and NO release induced by chronic spinal cord injury.

http://www.wikidata.org/entity/Q36737852

about

Intravesical liposome and antisense treatment for detrusor overactivity and interstitial cystitis/painful bladder syndrome Best of the 2008 AUA Annual Meeting: Highlights from the 2008 Annual Meeting of the American Urological Association, May 17-22, 2008, Orlando, FL Treatment of painful bladder syndrome/interstitial cystitis with botulinum toxin A: why isn't it effective in all patients?Novel Treatment of Chronic Bladder Pain Syndrome and Other Pelvic Pain Disorders by OnabotulinumtoxinA Injection In pursuit of P2X3 antagonists: novel therapeutics for chronic pain and afferent sensitization.Urinary bladder, cystitis and nerve/urothelial interactions.Urothelial signaling.Effect of botulinum toxin A on urothelial-release of ATP and expression of SNARE targets within the urothelium.Expression and distribution of ectonucleotidases in mouse urinary bladder.Local receptors as novel regulators for peripheral clock expression.The Role of the Mucosa in Normal and Abnormal Bladder Function.Overactive and underactive bladder dysfunction is reflected by alterations in urothelial ATP and NO release Role of Purinergic Signaling in Voiding Dysfunction.Cystitis, co-morbid disorders and associated epithelial dysfunction Preclinical assessment of potential interactions between botulinum toxin and neuromodulation for bladder micturition reflex.Does Reduction of Number of Intradetrusor Injection Sites of aboBoNTA (Dysport®) Impact Efficacy and Safety in a Rat Model of Neurogenic Detrusor Overactivity?Common theme for drugs effective in overactive bladder treatment: inhibition of afferent signaling from the bladder Extracellular UDP enhances P2X-mediated bladder smooth muscle contractility via P2Y(6) activation of the phospholipase C/inositol trisphosphate pathway.New developments in the management of overactive bladder: focus on mirabegron and onabotulinumtoxinA.Age-related changes in afferent pathways and urothelial function in the male mouse bladder.Purinergic signalling in the urinary tract in health and disease.Spinal cord injury sequelae alter drug pharmacokinetics: an overview.Researching bladder afferents-determining the effects of β(3) -adrenergic receptor agonists and botulinum toxin type-A.Onabotulinum toxin A for the treatment of neurogenic detrusor overactivity due to spinal cord injury or multiple sclerosis.Targets for botulinum toxin in the lower urinary tract.Potential therapeutic effect of intravesical botulinum toxin type A on bladder pain syndrome/interstitial cystitis.Therapeutic targets for overactive bladder other than smooth muscle.Current and potential urological applications of botulinum toxin A.New Frontiers of Basic Science Research in Neurogenic Lower Urinary Tract Dysfunction.Plasticity of TRPV1-Expressing Sensory Neurons Mediating Autonomic Dysreflexia Following Spinal Cord Injury.Regulation of ACh release from guinea pig bladder urothelial cells: potential role in bladder filling sensations.OnabotulinumtoxinA significantly attenuates bladder afferent nerve firing and inhibits ATP release from the urothelium.Intradetrusor versus suburothelial onabotulinumtoxinA injections for neurogenic detrusor overactivity: a pilot study.Injections of Botulinum Toxin A into the detrusor to treat neurogenic detrusor overactivity secondary to spinal cord injury.The role of the mucosa in modulation of evoked responses in the spinal cord injured rat bladder.Central inhibitory effect of intravesically applied botulinum toxin A in chronic spinal cord injury.Intravesical instillation of botulinum toxin A: an in vivo murine study and pilot clinical trial.Mucosal modulation of contractility in bladder strips from normal and overactive rat models and the effect of botulinum toxin A on overactive bladder strips.Effect of detrusor botulinum toxin a injection on urothelial dysfunction in patients with chronic spinal cord injury: a clinical and immunohistochemistry study before and after treatment.

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Botulinum toxin type A normalizes alterations in urothelial ATP and NO release induced by chronic spinal cord injury.

http://www.wikidata.org/entity/Q36737852

description

2007 nî lūn-bûn

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Botulinum toxin type A normali ...... by chronic spinal cord injury.

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Botulinum toxin type A normali ...... by chronic spinal cord injury.

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Botulinum toxin type A normali ...... by chronic spinal cord injury.

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Botulinum toxin type A normali ...... by chronic spinal cord injury.

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Botulinum toxin type A normali ...... by chronic spinal cord injury.

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Botulinum toxin type A normali ...... by chronic spinal cord injury.

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J.NEUINT.2007.11.006

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Neurochemistry International

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Botulinum toxin type A normali ...... by chronic spinal cord injury.

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Alvaro Munoz

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Christopher P Smith

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David A Gangitano

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George T Somogyi

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Joseph Francis

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K Roger Aoki

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Nilson A Salas

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Timothy B Boone

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P2860

Effect of intravesical nitric oxide therapy on cyclophosphamide-induced cystitis

Altered urinary bladder function in mice lacking the vanilloid receptor TRPV1

P2X2 knockout mice and P2X2/P2X3 double knockout mice reveal a role for the P2X2 receptor subunit in mediating multiple sensory effects of ATP

P2X3 knock-out mice reveal a major sensory role for urothelially released ATP

Botulinum neurotoxins serotypes A and E cleave SNAP-25 at distinct COOH-terminal peptide bonds

Vanilloid receptor expression suggests a sensory role for urinary bladder epithelial cells.

The dual personality of NO.

Non-neuronal acetylcholine and urinary bladder urothelium.

Frequency dependence of muscarinic facilitation of transmitter release in urinary bladder strips from neurally intact or chronic spinal cord transected rats.

Integrative control of the lower urinary tract: preclinical perspective

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1068-1075

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10.1016/J.NEUINT.2007.11.006

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6

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18187233

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2440726

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