Melanoma-derived conditioned media efficiently induce the differentiation of monocytes to macrophages that display a highly invasive gene signature.
about
An adaptive signaling network in melanoma inflammatory niches confers tolerance to MAPK signaling inhibition.Antibody therapies for melanoma: new and emerging opportunities to activate immunity (Review).BRAF(V600E) melanoma cells secrete factors that activate stromal fibroblasts and enhance tumourigenicity.Effects of BRAF mutations and BRAF inhibition on immune responses to melanomaDistinct role of FoxO1 in M-CSF- and GM-CSF-differentiated macrophages contributes LPS-mediated IL-10: implication in hyperglycemia.Liposome encapsulated zoledronate favours M1-like behaviour in murine macrophages cultured with soluble factors from breast cancer cells.CSF1-ETS2-induced microRNA in myeloid cells promote metastatic tumor growth.BRAF Inhibition Stimulates Melanoma-Associated Macrophages to Drive Tumor GrowthForkhead Box O1 Regulates Macrophage Polarization Following Staphylococcus aureus Infection: Experimental Murine Data and Review of the Literature.SECTM1 produced by tumor cells attracts human monocytes via CD7-mediated activation of the PI3K pathway.Downregulation of sphingosine kinase-1 induces protective tumor immunity by promoting M1 macrophage response in melanoma.Polarization of macrophages in the tumor microenvironment is influenced by EGFR signaling within colon cancer cellsMicroenvironment and tumor progression of melanoma: new therapeutic prospectives.Melanocytic tumors with intraepidermal melanophages: a report of five cases with review of 231 archived cutaneous melanocytic tumors.Nicotinamide phosphoribosyltransferase (NAMPT/PBEF/visfatin) is a tumoural cytokine released from melanoma.Primary Tr1 cells from metastatic melanoma eliminate tumor-promoting macrophages through granzyme B- and perforin-dependent mechanisms.Mechanisms and prevention of UV-induced melanoma.Identification of Caspase-6 as a New Regulator of Alternatively Activated Macrophages.The macrophage: Switches from a passenger to a driver during anticancer therapy.Strategies to Improve the Efficacy of Dendritic Cell-Based Immunotherapy for Melanoma.Mechanisms of resistance to BRAF and MEK inhibitors and clinical update of US Food and Drug Administration-approved targeted therapy in advanced melanoma
P2860
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P2860
Melanoma-derived conditioned media efficiently induce the differentiation of monocytes to macrophages that display a highly invasive gene signature.
description
2012 nî lūn-bûn
@nan
2012年の論文
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2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
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2012年论文
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2012年论文
@zh-cn
name
Melanoma-derived conditioned m ...... ighly invasive gene signature.
@ast
Melanoma-derived conditioned m ...... ighly invasive gene signature.
@en
type
label
Melanoma-derived conditioned m ...... ighly invasive gene signature.
@ast
Melanoma-derived conditioned m ...... ighly invasive gene signature.
@en
prefLabel
Melanoma-derived conditioned m ...... ighly invasive gene signature.
@ast
Melanoma-derived conditioned m ...... ighly invasive gene signature.
@en
P2093
P2860
P1476
Melanoma-derived conditioned m ...... ighly invasive gene signature.
@en
P2093
Alfonso Lopez-Coral
Frank J Rauscher
Rajasekharan Somasundaram
Rikka Azuma
Russel E Kaufman
Xiaowei Xu
Yingbin Ge
P2860
P304
P356
10.1111/J.1755-148X.2012.01005.X
P577
2012-07-01T00:00:00Z