Initial delay in the immune response to Francisella tularensis is followed by hypercytokinemia characteristic of severe sepsis and correlating with upregulation and release of damage-associated molecular patterns.
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Comparative review of Francisella tularensis and Francisella novicidaFine tuning inflammation at the front door: macrophage complement receptor 3-mediates phagocytosis and immune suppression for Francisella tularensisTLR-dependent control of Francisella tularensis infection and host inflammatory responsesNovel catanionic surfactant vesicle vaccines protect against Francisella tularensis LVS and confer significant partial protection against F. tularensis Schu S4 strainMembers of the Francisella tularensis phagosomal transporter subfamily of major facilitator superfamily transporters are critical for pathogenesisRapid dissemination of Francisella tularensis and the effect of route of infection.Tularemia induces different biochemical responses in BALB/c mice and common volesProtective role of Mincle in bacterial pneumonia by regulation of neutrophil mediated phagocytosis and extracellular trap formationGlycosylation of a Capsule-Like Complex (CLC) by Francisella novicida Is Required for Virulence and Partial Protective Immunity in Mice.Attenuated response of aged mice to respiratory Francisella novicida is characterized by reduced cell death and absence of subsequent hypercytokinemia.FTT0831c/FTL_0325 contributes to Francisella tularensis cell division, maintenance of cell shape, and structural integrity.A galU mutant of Francisella tularensis is attenuated for virulence in a murine pulmonary model of tularemiaHost-pathogen interactions and immune evasion strategies in Francisella tularensis pathogenicityGalectin-3 functions as an alarmin: pathogenic role for sepsis development in murine respiratory tularemia.Features of sepsis caused by pulmonary infection with Francisella tularensis Type A strainA Francisella tularensis locus required for spermine responsiveness is necessary for virulence.Targeting of a Fixed Bacterial Immunogen to Fc Receptors Reverses the Anti-Inflammatory Properties of the Gram-Negative Bacterium, Francisella tularensis, during the Early Stages of InfectionTGF-β Blood Levels Distinguish Between Influenza A (H1N1)pdm09 Virus Sepsis and Sepsis due to Other Forms of Community-Acquired PneumoniaDevelopment of tolerogenic dendritic cells and regulatory T cells favors exponential bacterial growth and survival during early respiratory tularemia.A human macrophage-hepatocyte co-culture model for comparative studies of infection and replication of Francisella tularensis LVS strain and subspecies holarctica and mediasiatica.An Immature Myeloid/Myeloid-Suppressor Cell Response Associated with Necrotizing Inflammation Mediates Lethal Pulmonary Tularemia.Perforin- and granzyme-mediated cytotoxic effector functions are essential for protection against Francisella tularensis following vaccination by the defined F. tularensis subsp. novicida ΔfopC vaccine strain.Aged mice display an altered pulmonary host response to Francisella tularensis live vaccine strain (LVS) infections.Proteomic analysis of bronchoalveolar lavage fluid proteins from mice infected with Francisella tularensis ssp. novicida.Inflammasome-Independent NLRP3 Restriction of a Protective Early Neutrophil Response to Pulmonary Tularemia.Critical Role of Glycogen Synthase Kinase-3β in Regulating the Avian Heterophil Response to Salmonella enterica Serovar EnteritidisKdo hydrolase is required for Francisella tularensis virulence and evasion of TLR2-mediated innate immunity.B1a cells enhance susceptibility to infection with virulent Francisella tularensis via modulation of NK/NKT cell responses.Temporal transcriptional response during infection of type II alveolar epithelial cells with Francisella tularensis live vaccine strain (LVS) supports a general host suppression and bacterial uptake by macropinocytosis.Macrophage Galactose-Type Lectin-1 Deficiency Is Associated with Increased Neutrophilia and Hyperinflammation in Gram-Negative PneumoniaHost immune response and acute disease in a zebrafish model of Francisella pathogenesisType I interferon licenses enhanced innate recognition and transcriptional responses to Franciscella tularensis live vaccine strain.Lethal pulmonary infection with Francisella novicida causes depletion of alphabeta T cells from lungs.MyD88-dependent signaling prolongs survival and reduces bacterial burden during pulmonary infection with virulent Francisella tularensis.Francisella tularensis - Immune Cell Activator, Suppressor, or Stealthy Evader: The Evolving View from the Petri Dish.Lethal pulmonary infection with Francisella novicida is associated with severe sepsisVaccination with an attenuated strain of Francisella novicida prevents T-cell depletion and protects mice infected with the wild-type strain from severe sepsisCardiac Electrical and Structural Changes During Bacterial Infection: An Instructive Model to Study Cardiac Dysfunction in Sepsis.Programmed cell death and the pathogenesis of tissue injury induced by type A Francisella tularensis.TolC-dependent modulation of host cell death by the Francisella tularensis live vaccine strain.
P2860
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P2860
Initial delay in the immune response to Francisella tularensis is followed by hypercytokinemia characteristic of severe sepsis and correlating with upregulation and release of damage-associated molecular patterns.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Initial delay in the immune re ...... associated molecular patterns.
@en
type
label
Initial delay in the immune re ...... associated molecular patterns.
@en
prefLabel
Initial delay in the immune re ...... associated molecular patterns.
@en
P2093
P2860
P356
P1476
Initial delay in the immune re ...... associated molecular patterns.
@en
P2093
Chris A Mares
Elizabeth G Morris
Judy M Teale
Sandra S Ojeda
P2860
P304
P356
10.1128/IAI.00215-08
P407
P577
2008-04-14T00:00:00Z