FliZ Is a posttranslational activator of FlhD4C2-dependent flagellar gene expression.
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Lessons in Fundamental Mechanisms and Diverse Adaptations from the 2015 Bacterial Locomotion and Signal Transduction MeetingNon-genetic diversity shapes infectious capacity and host resistanceEAL domain protein YdiV acts as an anti-FlhD4C2 factor responsible for nutritional control of the flagellar regulon in Salmonella enterica Serovar TyphimuriumThe role of coupled positive feedback in the expression of the SPI1 type three secretion system in Salmonella.Induction of Salmonella pathogenicity island 1 under different growth conditions can affect Salmonella-host cell interactions in vitroRole of cross talk in regulating the dynamic expression of the flagellar Salmonella pathogenicity island 1 and type 1 fimbrial genesThe flagellar regulator fliT represses Salmonella pathogenicity island 1 through flhDC and fliZ.A nutrient-tunable bistable switch controls motility in Salmonella enterica serovar Typhimurium.FliZ induces a kinetic switch in flagellar gene expression.FliZ is a global regulatory protein affecting the expression of flagellar and virulence genes in individual Xenorhabdus nematophila bacterial cells.Refining the binding of the Escherichia coli flagellar master regulator, FlhD4C2, on a base-specific levelC-ring requirement in flagellar type III secretion is bypassed by FlhDC upregulation.Regulation of phenotypic heterogeneity permits Salmonella evasion of the host caspase-1 inflammatory response.Perturbation of FliL interferes with Proteus mirabilis swarmer cell gene expression and differentiation.Characterization of Novel Factors Involved in Swimming and Swarming Motility in Salmonella enterica Serovar TyphimuriumRNA-seq Brings New Insights to the Intra-Macrophage Transcriptome of Salmonella TyphimuriumThe Impact of 18 Ancestral and Horizontally-Acquired Regulatory Proteins upon the Transcriptome and sRNA Landscape of Salmonella enterica serovar Typhimurium.Spontaneous mutations in the flhD operon generate motility heterogeneity in Escherichia coli biofilm.RflM functions as a transcriptional repressor in the autogenous control of the Salmonella Flagellar master operon flhDC.Role of FimW, FimY, and FimZ in regulating the expression of type i fimbriae in Salmonella enterica serovar Typhimurium.High-resolution, long-term characterization of bacterial motility using optical tweezers.Escherichia coli swimming is robust against variations in flagellar number.FliZ acts as a repressor of the ydiV gene, which encodes an anti-FlhD4C2 factor of the flagellar regulon in Salmonella enterica serovar typhimurium.Integrating global regulatory input into the Salmonella pathogenicity island 1 type III secretion system.SprB is the molecular link between Salmonella pathogenicity island 1 (SPI1) and SPI4.Mathematical model of flagella gene expression dynamics in Salmonella enterica serovar typhimurium.The global repressor FliZ antagonizes gene expression by σS-containing RNA polymerase due to overlapping DNA binding specificity.RsmC of Erwinia carotovora subsp. carotovora negatively controls motility, extracellular protein production, and virulence by binding FlhD and modulating transcriptional activity of the master regulator, FlhDC.FliZ regulates expression of the Salmonella pathogenicity island 1 invasion locus by controlling HilD protein activity in Salmonella enterica serovar typhimurium.Endogenously generated 2-aminoacrylate inhibits motility in Salmonella enterica.Mutually repressing repressor functions and multi-layered cellular heterogeneity regulate the bistable Salmonella fliC census.Activation of motility by sensing short-chain fatty acids via two steps in a flagellar gene regulatory cascade in enterohemorrhagic Escherichia coliglnA truncation in Salmonella enterica results in a small colony variant phenotype, attenuated host cell entry, and reduced expression of flagellin and SPI-1 associated effector genes.Dynamic Measures of Flagellar Gene Expression.Continuous control of flagellar gene expression by the σ28-FlgM regulatory circuit in Salmonella enterica.Involvement of Two-Component Signaling on Bacterial Motility and Biofilm Development.Dynamics and Control of Flagella Assembly in Salmonella typhimurium.
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P2860
FliZ Is a posttranslational activator of FlhD4C2-dependent flagellar gene expression.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
FliZ Is a posttranslational activator of FlhD4C2-dependent flagellar gene expression.
@en
type
label
FliZ Is a posttranslational activator of FlhD4C2-dependent flagellar gene expression.
@en
prefLabel
FliZ Is a posttranslational activator of FlhD4C2-dependent flagellar gene expression.
@en
P2093
P2860
P356
P1476
FliZ Is a posttranslational activator of FlhD4C2-dependent flagellar gene expression.
@en
P2093
Christopher V Rao
Jonathon D Brown
Phillip D Aldridge
Supreet Saini
P2860
P304
P356
10.1128/JB.01996-07
P407
P577
2008-05-09T00:00:00Z