Too much glucagon, too little insulin: time course of pancreatic islet dysfunction in new-onset type 1 diabetes. - Wikidata - awgldk - TriplyDB

Too much glucagon, too little insulin: time course of pancreatic islet dysfunction in new-onset type 1 diabetes.

Too much glucagon, too little insulin: time course of pancreatic islet dysfunction in new-onset type 1 diabetes.

http://www.wikidata.org/entity/Q36756783

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Using Drosophila to discover mechanisms underlying type 2 diabetes The Role of Dietary Protein and Fat in Glycaemic Control in Type 1 Diabetes: Implications for Intensive Diabetes Management Protein- and diabetes-induced glomerular hyperfiltration: role of glucagon, vasopressin, and urea Evolution of abnormal plasma glucagon responses to mixed-meal feedings in youth with type 1 diabetes during the first 2 years after diagnosis Metabolic implications when employing heavy pre- and post-exercise rapid-acting insulin reductions to prevent hypoglycaemia in type 1 diabetes patients: a randomised clinical trial.Hyperglucagonemia in an animal model of insulin- deficient diabetes: what therapy can improve it?The influence of exposure to maternal diabetes in utero on the rate of decline in β-cell function among youth with diabetes.Minireview: Glucagon in the pathogenesis of hypoglycemia and hyperglycemia in diabetes.Clinical evolution of beta cell function in youth with diabetes: the SEARCH for Diabetes in Youth study Achievement of target A1C levels with negligible hypoglycemia and low glucose variability in youth with short-term type 1 diabetes and residual β-cell function.The metabolic progression to type 1 diabetes as indicated by serial oral glucose tolerance testing in the Diabetes Prevention Trial-type 1.Comparison of Metabolic Outcomes in Children Diagnosed with Type 1 Diabetes Through Research Screening (Diabetes Autoimmunity Study in the Young [DAISY]) Versus in the Community.Early loss of the glucagon response to hypoglycemia in adolescents with type 1 diabetes.Fibroblast growth factor 21 mediates specific glucagon actions Paracrine regulation of glucagon secretion: the β/α/δ model.Lack of association between residual insulin production and glucagon response to hypoglycemia in youth with short duration of type 1 diabetes Blocking of beta-2 adrenergic receptors hastens recovery from hypoglycemia-associated social withdrawal.Metreleptin improves blood glucose in patients with insulin receptor mutations.C-peptide in the natural history of type 1 diabetes Beta-cell-mediated signaling predominates over direct alpha-cell signaling in the regulation of glucagon secretion in humans Acute metabolic effects of exenatide in patients with type 1 diabetes with and without residual insulin to oral and intravenous glucose challenges.The metabolic actions of glucagon revisited Residual C-peptide in type 1 diabetes: what do we really know?Glucagon in artificial pancreas systems: Potential benefits and safety profile of future chronic use.Increasing the protein quantity in a meal results in dose-dependent effects on postprandial glucose levels in individuals with Type 1 diabetes mellitus.Secondary consequences of beta cell inexcitability: identification and prevention in a murine model of K(ATP)-induced neonatal diabetes mellitus.Effect of exenatide on gastric emptying and graft survival in islet allograft recipients Morphological assessment of pancreatic islet hormone content following aerobic exercise training in rats with poorly controlled Type 1 diabetes mellitus.Increasing glucagon secretion could antagonize the action of exogenous insulin for glycemic control in streptozocin-induced diabetic rhesus monkeys.Characterization of residual β cell function in long-standing type 1 diabetes.Decreased α-cell mass and early structural alterations of the exocrine pancreas in patients with type 1 diabetes: An analysis based on the nPOD repository.Residual beta-cell function in diabetes children followed and diagnosed in the TEDDY study compared to community controls.Sustained treatment with a stable long-acting oxyntomodulin analogue improves metabolic control and islet morphology in an experimental model of type 1 diabetes.Glucagon Receptor Signaling Regulates Energy Metabolism via Hepatic Farnesoid X Receptor and Fibroblast Growth Factor 21 The influence of glucagon on postprandial hyperglycaemia in children 5 years after onset of type 1 diabetes

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P2860

Too much glucagon, too little insulin: time course of pancreatic islet dysfunction in new-onset type 1 diabetes.

http://www.wikidata.org/entity/Q36756783

description

2008 nî lūn-bûn

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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2008年论文

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2008年论文

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name

Too much glucagon, too little ...... in new-onset type 1 diabetes.

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Too much glucagon, too little ...... in new-onset type 1 diabetes.

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Too much glucagon, too little ...... in new-onset type 1 diabetes.

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Too much glucagon, too little ...... in new-onset type 1 diabetes.

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Too much glucagon, too little ...... in new-onset type 1 diabetes.

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Too much glucagon, too little ...... in new-onset type 1 diabetes.

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Too much glucagon, too little ...... in new-onset type 1 diabetes.

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Kristina I Rother

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Ninet Sinaii

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Rebecca J Brown

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P2860

Alpha-cells of the endocrine pancreas: 35 years of research but the enigma remains.

Beta-cell function and the development of diabetes-related complications in the diabetes control and complications trial.

Insulin secretion in type 1 diabetes.

Impact of glucagon response on postprandial hyperglycemia in men with impaired glucose tolerance and type 2 diabetes mellitus.

Suppressibility of glucagon secretion by glucose in juvenile diabetes.

Meal-Stimulated Glucagon Release Is Associated with Postprandial Blood Glucose Level and Does Not Interfere with Glycemic Control in Children and Adolescents with New-Onset Type 1 Diabetes

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1403-1404

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10.2337/DC08-0575

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type-1 diabetes

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