Parkinson disease: a new link between monoamine oxidase and mitochondrial electron flow
about
Metabolic control analysis in a cellular model of elevated MAO-B: relevance to Parkinson's diseaseClassification of H₂O₂as a neuromodulator that regulates striatal dopamine release on a subsecond time scaleMAO-B elevation in mouse brain astrocytes results in Parkinson's pathologyInhibition of the cyclooxygenase isoenzymes COX-1 and COX-2 provide neuroprotection in the MPTP-mouse model of Parkinson's diseaseMAOA-a novel decision maker of apoptosis and autophagy in hormone refractory neuroendocrine prostate cancer cells.Mitochondria are the source of hydrogen peroxide for dynamic brain-cell signaling.The AS/AGU rat: a spontaneous model of disruption and degeneration in the nigrostriatal dopaminergic system.Ability to delay neuropathological events associated with astrocytic MAO-B increase in a Parkinsonian mouse model: implications for early intervention on disease progression.Swimming training induces liver mitochondrial adaptations to oxidative stress in rats submitted to repeated exhaustive swimming boutsInflammatory Animal Model for Parkinson's Disease: The Intranigral Injection of LPS Induced the Inflammatory Process along with the Selective Degeneration of Nigrostriatal Dopaminergic Neurons.Pro-apoptotic gene expression mediated by the p38 mitogen-activated protein kinase signal transduction pathwayGlutathione depletion-induced chromosomal DNA fragmentation associated with apoptosis and necrosis.Low monoamine oxidase B in peripheral organs in smokersThiol-redox signaling, dopaminergic cell death, and Parkinson's diseaseAcrolein-mediated injury in nervous system trauma and diseasesNovel suppressors of alpha-synuclein toxicity identified using yeastA FRET-based method to study protein thiol oxidation in histological preparationsEffects of neuroglobin overexpression on mitochondrial function and oxidative stress following hypoxia/reoxygenation in cultured neuronsProteomic analysis of rat brain mitochondria following exposure to dopamine quinone: implications for Parkinson disease.Proteomic identification of dopamine-conjugated proteins from isolated rat brain mitochondria and SH-SY5Y cells.Inhibition of prolyl hydroxylase protects against 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine-induced neurotoxicity: model for the potential involvement of the hypoxia-inducible factor pathway in Parkinson disease.Hypoxia inducible factor-1 as a target for neurodegenerative diseases.Monoamine oxidases in development.Ursolic Acid-enriched herba cynomorii extract induces mitochondrial uncoupling and glutathione redox cycling through mitochondrial reactive oxygen species generation: protection against menadione cytotoxicity in h9c2 cells.Dopamine and α-synuclein dysfunction in Smad3 null micePretreatment with Bacopa monnieri extract offsets 3-nitropropionic acid induced mitochondrial oxidative stress and dysfunctions in the striatum of prepubertal mouse brain.Cerebrospinal Fluid from Sporadic Amyotrophic Lateral Sclerosis Patients Induces Mitochondrial and Lysosomal Dysfunction.Heme-oxygenase-1 promotes polychlorinated biphenyl mixture aroclor 1254-induced oxidative stress and dopaminergic cell injury.COM crystals activate the p38 mitogen-activated protein kinase signal transduction pathway in renal epithelial cells.Protective effect of harmalol and harmaline on MPTP neurotoxicity in the mouse and dopamine-induced damage of brain mitochondria and PC12 cells.Monoamine oxidase a expression is vital for embryonic brain development by modulating developmental apoptosisDetermination of reactive oxygen species associated with the degeneration of dopaminergic neurons during dopamine metabolism.Nitroindazole compounds inhibit the oxidative activation of 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP) neurotoxin to neurotoxic pyridinium cations by human monoamine oxidase (MAO).Mitochondria deficient in complex I activity are depolarized by hydrogen peroxide in nerve terminals: relevance to Parkinson's disease.H(2)O(2) is a novel, endogenous modulator of synaptic dopamine release.Protective effect of harmaline and harmalol against dopamine- and 6-hydroxydopamine-induced oxidative damage of brain mitochondria and synaptosomes, and viability loss of PC12 cells.Hydrogen peroxide removal and glutathione mixed disulfide formation during metabolic inhibition in mesencephalic cultures.Oxalate selectively activates p38 mitogen-activated protein kinase and c-Jun N-terminal kinase signal transduction pathways in renal epithelial cells.Modulation of somatodendritic dopamine release by endogenous H(2)O(2): susceptibility in substantia nigra but resistance in VTA.Oxidation of DOPAC by nitric oxide: effect of superoxide dismutase.
P2860
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P2860
Parkinson disease: a new link between monoamine oxidase and mitochondrial electron flow
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年論文
@yue
1997年論文
@zh-hant
1997年論文
@zh-hk
1997年論文
@zh-mo
1997年論文
@zh-tw
1997年论文
@wuu
1997年论文
@zh
1997年论文
@zh-cn
name
Parkinson disease: a new link between monoamine oxidase and mitochondrial electron flow
@ast
Parkinson disease: a new link between monoamine oxidase and mitochondrial electron flow
@en
type
label
Parkinson disease: a new link between monoamine oxidase and mitochondrial electron flow
@ast
Parkinson disease: a new link between monoamine oxidase and mitochondrial electron flow
@en
prefLabel
Parkinson disease: a new link between monoamine oxidase and mitochondrial electron flow
@ast
Parkinson disease: a new link between monoamine oxidase and mitochondrial electron flow
@en
P2860
P50
P356
P1476
Parkinson disease: a new link between monoamine oxidase and mitochondrial electron flow
@en
P2860
P304
P356
10.1073/PNAS.94.10.4890
P4011
609a981f499edd9c9172aff1f87f3ef1d5b912f5
P407
P577
1997-05-01T00:00:00Z