Peptidergic CGRPα primary sensory neurons encode heat and itch and tonically suppress sensitivity to cold. - Wikidata - awgldk - TriplyDB

Peptidergic CGRPα primary sensory neurons encode heat and itch and tonically suppress sensitivity to cold.

Peptidergic CGRPα primary sensory neurons encode heat and itch and tonically suppress sensitivity to cold.

http://www.wikidata.org/entity/Q36772210

about

Why we scratch an itch: the molecules, cells and circuits of itch The functional and anatomical dissection of somatosensory subpopulations using mouse genetics Inhibition of mechanical allodynia in neuropathic pain by TLR5-mediated A-fiber blockade.New insights into the mechanisms of itch: are pain and itch controlled by distinct mechanisms?The cell biology of acute itch Evolving understanding on the aetiology of thermally provoked itch Calcitonin gene-related peptide: physiology and pathophysiology Activation of mammalian target of rapamycin contributes to pain nociception induced in rats by BmK I, a sodium channel-specific modulator Neural processing of itch.Three functionally distinct classes of C-fibre nociceptors in primates.Neuroepithelial circuit formed by innervation of sensory enteroendocrine cells.Trk receptor signaling and sensory neuron fate are perturbed in human neuropathy caused by Gars mutations.Fatty-acid-binding protein inhibition produces analgesic effects through peripheral and central mechanisms.The Troubled Touch of Autism.A review of topical high-concentration L-menthol as a translational model of cold allodynia and hyperalgesia.Anti-hyperalgesic effects of a novel TRPM8 agonist in neuropathic rats: a comparison with topical menthol.Enhanced behavioral responses to cold stimuli following CGRPα sensory neuron ablation are dependent on TRPM8.αCGRP is essential for algesic exocytotic mobilization of TRPV1 channels in peptidergic nociceptors.Protein kinase Cδ mediates histamine-evoked itch and responses in pruriceptors Deletion of ENTPD3 does not impair nucleotide hydrolysis in primary somatosensory neurons or spinal cord.Spatio-temporal expression and functional involvement of transient receptor potential vanilloid 1 in diabetic mechanical allodynia in rats.ERK2 Alone Drives Inflammatory Pain But Cooperates with ERK1 in Sensory Neuron Survival Molecular and cellular mechanisms that initiate pain and itch.Extrinsic and intrinsic signals converge on the Runx1/CBFβ transcription factor for nonpeptidergic nociceptor maturation.Secreted herpes simplex virus-2 glycoprotein G alters thermal pain sensitivity by modifying NGF effects on TRPV1.Drosophila DH31 Neuropeptide and PDF Receptor Regulate Night-Onset Temperature Preference The role of calcitonin gene-related peptide in peripheral and central pain mechanisms including migraine.Calcium-permeable ion channels in pain signaling.Itch and nerve fibers with special reference to atopic dermatitis: therapeutic implications.Transmitting pain and itch messages: a contemporary view of the spinal cord circuits that generate gate control.Validation of ion channel targets.Cold Temperature Encoding by Cutaneous TRPA1 and TRPM8-Carrying Fibers in the Mouse.CGRP 8-37 enhances lipopolysaccharide-induced acute lung injury and regulating aquaporin 1 and 5 expressions in rats.Paclitaxel alters the evoked release of calcitonin gene-related peptide from rat sensory neurons in culture.The epithelial cell-derived atopic dermatitis cytokine TSLP activates neurons to induce itch.Distinct calcitonin gene-related peptide expression pattern in primary afferents contribute to different neuropathic symptoms following chronic constriction or crush injuries to the rat sciatic nerve.Comprehensive thermal preference phenotyping in mice using a novel automated circular gradient assay.The lipid kinase PIP5K1C regulates pain signaling and sensitization Ciguatoxins Evoke Potent CGRP Release by Activation of Voltage-Gated Sodium Channel Subtypes NaV1.9, NaV1.7 and NaV1.1.Slick (Kcnt2) Sodium-Activated Potassium Channels Limit Peptidergic Nociceptor Excitability and Hyperalgesia.

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Peptidergic CGRPα primary sensory neurons encode heat and itch and tonically suppress sensitivity to cold.

http://www.wikidata.org/entity/Q36772210

description

2013 nî lūn-bûn

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Peptidergic CGRPα primary sens ...... suppress sensitivity to cold.

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Peptidergic CGRPα primary sens ...... suppress sensitivity to cold.

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J.NEURON.2013.01.030

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Peptidergic CGRPα primary sens ...... suppress sensitivity to cold.

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Alaine L Pribisko

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Bonnie Taylor-Blake

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Eric S McCoy

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Jihong Zheng

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Mark J Zylka

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Sarah E Street

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P2860

The capsaicin receptor: a heat-activated ion channel in the pain pathway

Prostatic acid phosphatase is an ectonucleotidase and suppresses pain by generating adenosine

Deletion of vanilloid receptor 1-expressing primary afferent neurons for pain control

Distinct Nav1.7-dependent pain sensations require different sets of sensory and sympathetic neurons

Neuronal circuitry for pain processing in the dorsal horn

Correlations between neuronal morphology and electrophysiological features in the rodent superficial dorsal horn

Organization of intralaminar and translaminar neuronal connectivity in the superficial spinal dorsal horn

Pharmacological blockade of the vanilloid receptor TRPV1 elicits marked hyperthermia in humans

The menthol receptor TRPM8 is the principal detector of environmental cold

TRPM8 is required for cold sensation in mice

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