Repression of Na,K-ATPase beta1-subunit by the transcription factor snail in carcinoma. - Wikidata - awgldk - TriplyDB

Repression of Na,K-ATPase beta1-subunit by the transcription factor snail in carcinoma.

Repression of Na,K-ATPase beta1-subunit by the transcription factor snail in carcinoma.

http://www.wikidata.org/entity/Q36781290

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SNAI1 and SNAI2 are asymmetrically expressed at the 2-cell stage and become segregated to the TE in the mouse blastocyst Glucocorticoids suppress renal cell carcinoma progression by enhancing Na,K-ATPase beta-1 subunit expression Downregulation of ATP1A1 promotes cancer development in renal cell carcinoma.The Effect of Snail1 Gene Silencing by siRNA in Metastatic Breast Cancer Cell Lines.Cyclophilin B interacts with sodium-potassium ATPase and is required for pump activity in proximal tubule cells of the kidney Novel role for Na,K-ATPase in phosphatidylinositol 3-kinase signaling and suppression of cell motility.Association with {beta}-COP regulates the trafficking of the newly synthesized Na,K-ATPase Transcriptional factors associated with epithelial-mesenchymal transition in choroidal neovascularization.Cancer as a channelopathy: ion channels and pumps in tumor development and progression Wilms' tumor protein induces an epithelial-mesenchymal hybrid differentiation state in clear cell renal cell carcinoma Roles of the transcription factors snail and slug during mammary morphogenesis and breast carcinoma progression.The sodium pump alpha1 sub-unit: a disease progression-related target for metastatic melanoma treatment Na,K-ATPase β1-subunit is a target of sonic hedgehog signaling and enhances medulloblastoma tumorigenicity.Na,K-ATPase is a target of cigarette smoke and reduced expression predicts poor patient outcome of smokers with lung cancer Transcriptional regulators of Na,K-ATPase subunits.The sodium pump alpha1 subunit as a potential target to combat apoptosis-resistant glioblastomas.Na,K-ATPase Isozymes in Colorectal Cancer and Liver Metastases.Na,K-ATPase β-subunit cis homo-oligomerization is necessary for epithelial lumen formation in mammalian cells.A link between FXYD3 (Mat-8)-mediated Na,K-ATPase regulation and differentiation of Caco-2 intestinal epithelial cells.Tumor suppression in basal keratinocytes via dual non-cell-autonomous functions of a Na,K-ATPase beta subunit.Expression of Na,K-ATPase-beta(1) subunit increases uptake and sensitizes carcinoma cells to oxaliplatin.Unexpected roles of the Na-K-ATPase and other ion transporters in cell junctions and tubulogenesis.Mitochondrial dysfunction and cancer metastasis.Roles of ion transport in control of cell motility.Na+/K+-ATPase and cancer.Targeting Na⁺/K⁺ -translocating adenosine triphosphatase in cancer treatment.FXYD5 (dysadherin) may mediate metastatic progression through regulation of the β-Na+-K+-ATPase subunit in the 4T1 mouse breast cancer model.Na⁺,K⁺-ATPase as the Target Enzyme for Organic and Inorganic Compounds.Pharmacological and immunological targeting of tumor mesenchymalization.Epigenetic silencing of Na,K-ATPase β 1 subunit gene ATP1B1 by methylation in clear cell renal cell carcinoma.alpha-Catenin overrides Src-dependent activation of beta-catenin oncogenic signaling.Evidence for a potential tumor suppressor role for the Na,K-ATPase beta1-subunit.Repression of PTEN phosphatase by Snail1 transcriptional factor during gamma radiation-induced apoptosis.The alpha1 subunit of the sodium pump could represent a novel target to combat non-small cell lung cancers.The role of the beta1 subunit of the Na,K-ATPase and its glycosylation in cell-cell adhesion.Na-K-ATPase regulates tight junction permeability through occludin phosphorylation in pancreatic epithelial cells.Snail1 transcriptional repressor binds to its own promoter and controls its expression.Na,K-ATPase subunits as markers for epithelial-mesenchymal transition in cancer and fibrosis Regulation of Na,K-ATPase β1-subunit in TGF-β2-mediated epithelial-to-mesenchymal transition in human retinal pigmented epithelial cells."The Lower Threshold" phenomenon in tumor cells toward endogenous digitalis-like compounds: Responsible for tumorigenesis?

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P2860

Repression of Na,K-ATPase beta1-subunit by the transcription factor snail in carcinoma.

http://www.wikidata.org/entity/Q36781290

description

2003 nî lūn-bûn

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2003年の論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

@zh-tw

2003年论文

@wuu

2003年论文

@zh

2003年论文

@zh-cn

name

Repression of Na,K-ATPase beta1-subunit by the transcription factor snail in carcinoma.

@en

type

label

Repression of Na,K-ATPase beta1-subunit by the transcription factor snail in carcinoma.

@en

prefLabel

Repression of Na,K-ATPase beta1-subunit by the transcription factor snail in carcinoma.

@en

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MBC.E03-09-0646

P1433

Molecular Biology of the Cell

P1476

Repression of Na,K-ATPase beta1-subunit by the transcription factor snail in carcinoma.

@en

P2093

Ayyappan K Rajasekaran

http://www.w3.org/2001/XMLSchema#string

Cromwell E Espineda

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Jay H Chang

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Jeffery Twiss

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Sigrid A Rajasekaran

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P2860

SLUG, a ces-1-related zinc finger transcription factor gene with antiapoptotic activity, is a downstream target of the E2A-HLF oncoprotein

The SLUG zinc-finger protein represses E-cadherin in breast cancer

A putative fourth Na+,K(+)-ATPase alpha-subunit gene is expressed in testis

Characterization of the fourth alpha isoform of the Na,K-ATPase

The transcription factor snail is a repressor of E-cadherin gene expression in epithelial tumour cells

The snail superfamily of zinc-finger transcription factors

Cadherins: a molecular family important in selective cell-cell adhesion

Cell adhesion: the molecular basis of tissue architecture and morphogenesis

A novel snail-related transcription factor Smuc regulates basic helix-loop-helix transcription factor activities via specific E-box motifs.

The transition to endoreduplication in trophoblast giant cells is regulated by the mSNA zinc finger transcription factor

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1364-1373

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10.1091/MBC.E03-09-0646

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3

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15

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2003-12-29T00:00:00Z

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363145

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